Boal Andrew M相关文献与解析，生知库 | 链接生命科学的每一步

Intraocular Sustained Release of EPO-R76E Mitigates Glaucoma Pathogenesis by Activating the NRF2/ARE Pathway

眼内持续释放 EPO-R76E 通过激活 NRF2/ARE 通路减轻青光眼的发病机制

doi:10.3390/antiox12030556

Naguib, Sarah; DeJulius, Carlisle R; Backstrom, Jon R; Haider, Ameer A; Ang, John M; Boal, Andrew M; Calkins, David J; Duvall, Craig L; Rex, Tonia S

Retinal ganglion cells adapt to ionic stress in experimental glaucoma

视网膜神经节细胞在实验性青光眼中适应离子应激

期刊:Frontiers in Neuroscience

影响因子:3.2

doi:10.3389/fnins.2023.1142668

Boal, Andrew M; McGrady, Nolan R; Holden, Joseph M; Risner, Michael L; Calkins, David J

Sensitivity to extracellular potassium underlies type-intrinsic differences in retinal ganglion cell excitability

细胞外钾敏感性是视网膜神经节细胞兴奋性类型内在差异的基础。

期刊:Frontiers in Cellular Neuroscience

影响因子:4

doi:10.3389/fncel.2022.966425

Boal, Andrew M; McGrady, Nolan R; Risner, Michael L; Calkins, David J

细胞生物学

发表日期：2022

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TRPV1 Supports Axogenic Enhanced Excitability in Response to Neurodegenerative Stress

TRPV1支持轴突生成增强的兴奋性以应对神经退行性应激

期刊:Frontiers in Cellular Neuroscience

影响因子:4

doi:10.3389/fncel.2020.603419

Risner, Michael L; McGrady, Nolan R; Boal, Andrew M; Pasini, Silvia; Calkins, David J

Potassium-Dependent Coupling of Retinal Astrocyte Light Response to MÃ¼ller Glia.

钾依赖性视网膜星形胶质细胞光反应与穆勒胶质细胞的耦合

Intraocular Sustained Release of EPO-R76E Mitigates Glaucoma Pathogenesis by Activating the NRF2/ARE Pathway

眼内持续释放 EPO-R76E 通过激活 NRF2/ARE 通路减轻青光眼的发病机制

Retinal ganglion cells adapt to ionic stress in experimental glaucoma

视网膜神经节细胞在实验性青光眼中适应离子应激

Sensitivity to extracellular potassium underlies type-intrinsic differences in retinal ganglion cell excitability

细胞外钾敏感性是视网膜神经节细胞兴奋性类型内在差异的基础。

TRPV1 Supports Axogenic Enhanced Excitability in Response to Neurodegenerative Stress

TRPV1支持轴突生成增强的兴奋性以应对神经退行性应激