Booth Laurence相关文献与解析，生知库 | 链接生命科学的每一步

Booth, Laurence; Booth, Michael R; Roberts, Jane L; Yue, Yang; Kinsey, Emily; Poklepovic, Andrew; Boone, David; Cowhart, L Ashley; Baharaff, Allen; Dent, Paul

SCD

细胞生物学

肿瘤

GZ17-6.02 interacts with bexarotene to kill mycosis fungoides cells

GZ17-6.02 与贝沙罗汀相互作用杀死蕈样肉芽肿细胞

期刊:Oncotarget

影响因子:

doi:10.18632/oncotarget.28557

Michael R Booth, Laurence Booth, Jane L Roberts, Cameron West, Paul Dent

Neratinib kills B-RAF V600E melanoma via ROS-dependent autophagosome formation and death receptor signaling.

奈拉替尼通过 ROS 依赖性自噬体形成和死亡受体信号传导杀死 B-RAF V600E 黑色素瘤

期刊:Pigment Cell & Melanoma Research

影响因子:2.6

doi:10.1111/pcmr.13014

Dent Paul, Booth Laurence, Poklepovic Andrew, Kirkwood John M

Osimertinib-resistant NSCLC cells activate ERBB2 and YAP/TAZ and are killed by neratinib.

奥希替尼耐药的非小细胞肺癌细胞激活ERBB2和YAP/TAZ，并被奈拉替尼杀死

期刊:Biochemical Pharmacology

影响因子:5.6

doi:10.1016/j.bcp.2021.114642

Dent Paul, Booth Laurence, Poklepovic Andrew, Von Hoff Daniel, Martinez Jennifer, Zhou Yong, Hancock John F

The role of cell signaling in the crosstalk between autophagy and apoptosis in the regulation of tumor cell survival in response to sorafenib and neratinib

细胞信号传导在自噬和凋亡之间的相互作用中，调控肿瘤细胞对索拉非尼和奈拉替尼的存活反应中的作用

期刊:Seminars in Cancer Biology

影响因子:15.7

doi:10.1016/j.semcancer.2019.10.013

Booth, Laurence A; Roberts, Jane L; Dent, Paul

Neratinib degrades MST4 via autophagy that reduces membrane stiffness and is essential for the inactivation of PI3K, ERK1/2, and YAP/TAZ signaling

奈拉替尼通过自噬降解MST4，从而降低细胞膜刚性，这对PI3K、ERK1/2和YAP/TAZ信号通路的失活至关重要。

期刊:Journal of Cellular Physiology

影响因子:4

doi:10.1002/jcp.29443

Dent, Paul; Booth, Laurence; Poklepovic, Andrew; Martinez, Jennifer; Hoff, Daniel Von; Hancock, John F

Metabolism of Histone Deacetylase Proteins Opsonizes Tumor Cells to Checkpoint Inhibitory Immunotherapies

组蛋白去乙酰化酶的代谢可调理肿瘤细胞对检查点抑制性免疫疗法的反应

期刊:

影响因子:

doi:10.20900/immunometab20200002

Dent, Paul; Booth, Laurence; Poklepovic, Andrew

Neratinib inhibits Hippo/YAP signaling, reduces mutant K-RAS expression, and kills pancreatic and blood cancer cells.

奈拉替尼抑制 Hippo/YAP 信号传导，降低突变 K-RAS 表达，并杀死胰腺癌和血癌细胞

期刊:Oncogene

影响因子:7.3

doi:10.1038/s41388-019-0849-8

Dent Paul, Booth Laurence, Roberts Jane L, Liu Junchen, Poklepovic Andrew, Lalani Alshad S, Tuveson David, Martinez Jennifer, Hancock John F

Kinase inhibitors: look beyond the label on the bottle

激酶抑制剂：不要只看瓶子上的标签。

期刊:Cancer Drug Resistance

影响因子:5.2

doi:10.20517/cdr.2019.80

Dent, Paul; Poklepovic, Andrew; Booth, Laurence; Hancock, John F

信号转导

发表日期：2019

文献求助收藏

The SCD1 inhibitor aramchol interacts with regorafenib and metformin to kill tumor cells

SCD1抑制剂阿莫西林与瑞戈非尼和二甲双胍相互作用，可杀死肿瘤细胞。

The SCD1 inhibitor aramchol interacts with regorafenib to kill GI tumor cells in vitro and in vivo

SCD1抑制剂阿拉姆醇与瑞戈非尼相互作用，可在体外和体内杀死胃肠道肿瘤细胞。

GZ17-6.02 interacts with bexarotene to kill mycosis fungoides cells

GZ17-6.02 与贝沙罗汀相互作用杀死蕈样肉芽肿细胞

Neratinib kills B-RAF V600E melanoma via ROS-dependent autophagosome formation and death receptor signaling.

奈拉替尼通过 ROS 依赖性自噬体形成和死亡受体信号传导杀死 B-RAF V600E 黑色素瘤

Osimertinib-resistant NSCLC cells activate ERBB2 and YAP/TAZ and are killed by neratinib.

奥希替尼耐药的非小细胞肺癌细胞激活ERBB2和YAP/TAZ，并被奈拉替尼杀死

The role of cell signaling in the crosstalk between autophagy and apoptosis in the regulation of tumor cell survival in response to sorafenib and neratinib

细胞信号传导在自噬和凋亡之间的相互作用中，调控肿瘤细胞对索拉非尼和奈拉替尼的存活反应中的作用

Neratinib degrades MST4 via autophagy that reduces membrane stiffness and is essential for the inactivation of PI3K, ERK1/2, and YAP/TAZ signaling

奈拉替尼通过自噬降解MST4，从而降低细胞膜刚性，这对PI3K、ERK1/2和YAP/TAZ信号通路的失活至关重要。

Metabolism of Histone Deacetylase Proteins Opsonizes Tumor Cells to Checkpoint Inhibitory Immunotherapies

组蛋白去乙酰化酶的代谢可调理肿瘤细胞对检查点抑制性免疫疗法的反应

Neratinib inhibits Hippo/YAP signaling, reduces mutant K-RAS expression, and kills pancreatic and blood cancer cells.

奈拉替尼抑制 Hippo/YAP 信号传导，降低突变 K-RAS 表达，并杀死胰腺癌和血癌细胞

Kinase inhibitors: look beyond the label on the bottle

激酶抑制剂：不要只看瓶子上的标签。