Borbolis相关文献与解析，生知库 | 链接生命科学的每一步

Urolithin Α modulates inter-organellar communication via calcium-dependent mitophagy to promote healthy ageing

尿石素A通过钙依赖性线粒体自噬调节细胞器间通讯，从而促进健康衰老

doi:10.1080/15548627.2025.2561073

Antonis Roussos,Katerina Kitopoulou,Fivos Borbolis,Christina Ploumi,Despoina D Gianniou,Zhiquan Li,Haijun He,Eleni Tsakiri,Helena Borland,Ioannis K Kostakis,Martina Samiotaki,Ioannis P Trougakos,Vilhelm A Bohr,Konstantinos Palikaras

Calcium-mediated regulation of mitophagy: implications in neurodegenerative diseases

钙介导的线粒体自噬调控：在神经退行性疾病中的意义

期刊:

影响因子:

doi:10.1038/s44324-025-00049-2

Borbolis, Fivos; Ploumi, Christina; Palikaras, Konstantinos

Loss of DNA glycosylases improves health and cognitive function in a C. elegans model of human tauopathy

在秀丽隐杆线虫的人类tau蛋白病模型中，DNA糖基化酶的缺失可改善健康和认知功能。

期刊:Nucleic Acids Research

影响因子:13.1

doi:10.1093/nar/gkae705

Tiwari, Vinod; Buvarp, Elisabeth; Borbolis, Fivos; Puligilla, Chandrakala; Croteau, Deborah L; Palikaras, Konstantinos; Bohr, Vilhelm A

The Crosstalk between Microbiome and Mitochondrial Homeostasis in Neurodegeneration

微生物组与线粒体稳态在神经退行性疾病中的相互作用

期刊:Cells

影响因子:5.2

doi:10.3390/cells12030429

Borbolis, Fivos; Mytilinaiou, Eirini; Palikaras, Konstantinos

Caenorhabditis elegans as a Model System to Study Human Neurodegenerative Disorders

秀丽隐杆线虫作为研究人类神经退行性疾病的模型系统

期刊:Biomolecules

影响因子:4.8

doi:10.3390/biom13030478

Roussos, Antonis; Kitopoulou, Katerina; Borbolis, Fivos; Palikaras, Konstantinos

Selective Destabilization of Transcripts by mRNA Decapping Regulates Oocyte Maturation and Innate Immunity Gene Expression during Ageing in C. elegans

mRNA脱帽选择性地使转录本不稳定，从而调控秀丽隐杆线虫衰老过程中卵母细胞成熟和先天免疫基因表达

期刊:Biology-Basel

影响因子:3.5

doi:10.3390/biology12020171

Borbolis, Fivos; Ranti, Dimitra; Papadopoulou, Maria-Despina; Dimopoulou, Sofia; Malatras, Apostolos; Michalopoulos, Ioannis; Syntichaki, Popi

The compartmentalised nature of neuronal mitophagy: molecular insights and implications

神经元线粒体自噬的区室化特性：分子层面的见解和意义

期刊:Expert Reviews in Molecular Medicine

影响因子:5.5

doi:10.1017/erm.2022.31

Borbolis, Fivos; Palikaras, Konstantinos

Organismal and Cellular Stress Responses upon Disruption of Mitochondrial Lonp1 Protease

线粒体 Lonp1 蛋白酶破坏后的生物体和细胞应激反应

期刊:Cells

影响因子:5.1

doi:10.3390/cells11081363

Eirini Taouktsi, Eleni Kyriakou, Stefanos Smyrniotis, Fivos Borbolis, Labrina Bondi, Socratis Avgeris, Efstathios Trigazis, Stamatis Rigas, Gerassimos E Voutsinas, Popi Syntichaki

mRNA decapping is an evolutionarily conserved modulator of neuroendocrine signaling that controls development and ageing

mRNA 脱帽是控制发育和衰老的神经内分泌信号调节剂，在进化上是保守的

期刊:Elife

影响因子:6.4

doi:10.7554/eLife.53757

Fivos Borbolis, John Rallis, George Kanatouris, Nikolitsa Kokla, Antonis Karamalegkos, Christina Vasileiou, Katerina M Vakaloglou, George Diallinas, Dimitrios J Stravopodis, Christos G Zervas, Popi Syntichaki

信号转导

神经

细胞生物学

Developmental Biology

Novel roles of DNA glycosylases in neurodegenerative diseases and aging

DNA糖基化酶在神经退行性疾病和衰老中的新作用

Urolithin Α modulates inter-organellar communication via calcium-dependent mitophagy to promote healthy ageing

尿石素A通过钙依赖性线粒体自噬调节细胞器间通讯，从而促进健康衰老

Calcium-mediated regulation of mitophagy: implications in neurodegenerative diseases

钙介导的线粒体自噬调控：在神经退行性疾病中的意义

Loss of DNA glycosylases improves health and cognitive function in a C. elegans model of human tauopathy

在秀丽隐杆线虫的人类tau蛋白病模型中，DNA糖基化酶的缺失可改善健康和认知功能。

The Crosstalk between Microbiome and Mitochondrial Homeostasis in Neurodegeneration

微生物组与线粒体稳态在神经退行性疾病中的相互作用

Caenorhabditis elegans as a Model System to Study Human Neurodegenerative Disorders

秀丽隐杆线虫作为研究人类神经退行性疾病的模型系统

Selective Destabilization of Transcripts by mRNA Decapping Regulates Oocyte Maturation and Innate Immunity Gene Expression during Ageing in C. elegans

mRNA脱帽选择性地使转录本不稳定，从而调控秀丽隐杆线虫衰老过程中卵母细胞成熟和先天免疫基因表达

The compartmentalised nature of neuronal mitophagy: molecular insights and implications

神经元线粒体自噬的区室化特性：分子层面的见解和意义

Organismal and Cellular Stress Responses upon Disruption of Mitochondrial Lonp1 Protease

线粒体 Lonp1 蛋白酶破坏后的生物体和细胞应激反应

mRNA decapping is an evolutionarily conserved modulator of neuroendocrine signaling that controls development and ageing

mRNA 脱帽是控制发育和衰老的神经内分泌信号调节剂，在进化上是保守的