日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

IGH::FENDRR and specific KRAS mutations define a novel B-ALL molecular subtype with poor chemotherapy response

IGH::FENDRR 和特定的 KRAS 突变定义了一种新型的 B 细胞急性淋巴细胞白血病 (B-ALL) 分子亚型,该亚型对化疗反应较差。

Bendig, Sonja; Hartmann, Alina M; Wessels, Wiebke; Beder, Thomas; Kim, Rathana; Passet, Marie; Gao, Qingsong; Wolgast, Nadine; Horns, Johanna M; Alves Santos, Leonardo; Iben, Katharina; Steffen, Fabio D; Cantoni, Loredana; Kehden, Britta; Chitadze, Guranda; Künstner, Axel; Busch, Hauke; Bornhauser, Beat; Bourquin, Jean-Pierre; Leguay, Thibaut Tl; Boissel, Nicolas; Gökbuget, Nicola; Iacobucci, Ilaria; Mullighan, Charles G; Clappier, Emmanuelle; Baldus, Claudia D; Brüggemann, Monika; Bastian, Lorenz

Intra-subtype heterogeneity shapes treatment response in KMT2A-rearranged ALL across all age groups

亚型内异质性影响所有年龄组中KMT2A重排ALL的治疗反应

Hartmann, Alina M; Bastian, Lorenz; Barz, Malwine J; Haas, Johannes; Amelunxen, Eric; Ehm, Patrick; Lenk, Lennart; Kotrova, Michaela; Beder, Thomas; Steffen, Fabio D; Rauwolf, Kerstin; Wolgast, Nadine; Bendig, Sonja; Bozzetti, Cecilia; Alten, Julia; Mondal, Mayukh; Rademacher, Annika; Heymann, Julia; Walter, Wencke; Haferlach, Claudia; Ströh, Aeint-Steffen; Bergmann, Anke K; Burmeister, Thomas; Gökbuget, Nicola; Bornhauser, Beat; Bourquin, Jean-Pierre; Brüggemann, Monika; Schrappe, Martin; Cario, Gunnar; Baldus, Claudia D

Functional precision approach in patients with very high risk acute lymphoblastic leukaemia in India: a single-centre cohort study

印度高危急性淋巴细胞白血病患者的功能精准治疗:一项单中心队列研究

Sidhu, Jasmeet; Chakraborty, Arijit; Das, Parag; Steffen, Fabio D; Kundu, Subhajit; Basu, Sangramjit; Tarafdar, Bhaswati; Dey, Tanima; Kar, Abhirupa; Biswas, Mousumi; Das, Ankita; Sivadasan, Naveen; Dasgupta, Pritha; Ghara, Niharendu; Bornhauser, Beat; Bourquin, Jean-Pierre; Krishnan, Shekhar; Saha, Vaskar

Co-editing of NKG2A and FAS increases long-term cytotoxic capacity and persistence of CAR NK cells

NKG2A 和 FAS 的共编辑可增强 CAR NK 细胞的长期细胞毒性和持久性。

Juliane Mietz,Lukas Egli,Meike Misiune,Arianna Picozzi,Nicolò Coianiz,Danielle Thompson,Marianna Ponzo,Alisson Charmey,Beat Bornhauser,Chiara F Magnani,Christian Münz,Obinna Chijioke

A clinical-stage oncology compound selectively targets drug-resistant cancers

一种处于临床阶段的肿瘤化合物可选择性地靶向耐药性癌症。

Long, Kaitlin; Bhattacharjee, Debanjan; Newman-Stonebraker, Samuel H; Suhr, Simon; Mercado, Brandon Q; Scheib, Emily; Tighe, Anthony; Romero, Luciano; Thompson, Sarah L; Sausville, Erin L; John, Kristen M; Julian, Linda; Mishra, Sanat; Klingbeil, Olaf; Gupta, Punya; Bhatt, Uditi; Gao, Allen C; Ricardo, Sara; Vakoc, Christopher R; Bornhauser, Beat C; Corsello, Steven M; Taylor, Stephen S; Chen, Jue; Holland, Patrick L; Sheltzer, Jason M

Transcriptional remodeling shapes therapeutic vulnerability to necroptosis in acute lymphoblastic leukemia.

转录重塑影响急性淋巴细胞白血病细胞对坏死性凋亡的治疗敏感性。

Saorin Anna, Dehler Anna, Galvan Bartimée, Steffen Fabio, Ray Marine, Lu Dong, Yu Xin, Kim James, Drakul Aneta, Kisele Samanta, Wang Jin, Bourquin Jean-Pierre, Bornhauser Beat C

Role of stem-like cells in chemotherapy resistance and relapse in pediatric T-cell acute lymphoblastic leukemia.

干细胞样细胞在儿童T细胞急性淋巴细胞白血病化疗耐药和复发中的作用

Costea Julia, Rauwolf Kerstin K, Zafferani Pietro, Rausch Tobias, Mathioudaki Anna, Zaugg Judith, Schrappe Martin, Eckert Cornelia, Escherich Gabriele, Bourquin Jean P, Bornhauser Beat, Kulozik Andreas E, Korbel Jan O

Transcriptional and epigenetic rewiring by the NUP98::KDM5A fusion oncoprotein directly activates CDK12.

NUP98::KDM5A 融合癌蛋白通过转录和表观遗传重编程直接激活 CDK12

Troester Selina, Eder Thomas, Wukowits Nadja, Piontek Martin, Fernández-Pernas Pablo, Schmoellerl Johannes, Haladik Ben, Manhart Gabriele, Allram Melanie, Maurer-Granofszky Margarita, Scheidegger Nastassja, Nebral Karin, Superti-Furga Giulio, Meisel Roland, Bornhauser Beat, Valent Peter, Dworzak Michael N, Zuber Johannes, Boztug Kaan, Grebien Florian

A three-dimensional ex vivo model recapitulates in vivo features and drug resistance phenotypes in childhood acute lymphoblastic leukemia

三维体外模型重现了儿童急性淋巴细胞白血病的体内特征和耐药表型

Magdalini Kanari ,Iria Jimenez Garcia ,Fabio D Steffen ,Lisa A Krattiger ,Charles Bataclan ,Wangjie Liu ,Benjamin R Simona ,Bart Deplancke ,Olaia Naveiras ,Martin Ehrbar ,Beat Bornhauser ,Jean-Pierre Bourquin

SOD2 is a regulator of proteasomal degradation promoting an adaptive cellular starvation response.

SOD2 是蛋白酶体降解的调节因子,可促进细胞适应性饥饿反应

Ibrahim Nurul Khalida, Schreek Sabine, Cinar Buesra, Stasche Anna Sophie, Lee Su Hyun, Zeug Andre, Dolgner Tim, Niessen Julia, Ponimaskin Evgeni, Shcherbata Halyna, Fehlhaber Beate, Bourquin Jean-Pierre, Bornhauser Beat, Stanulla Martin, Pich Andreas, Gutierrez Alejandro, Hinze Laura