Bornstein Paul相关文献与解析，生知库 | 链接生命科学的每一步

Timothy S Luongo，Jared M Eller，Mu-Jie Lu，Marc Niere，Fabio Raith，Caroline Perry，Marc R Bornstein，Paul Oliphint，Lin Wang，Melanie R McReynolds，Marie E Migaud，Joshua D Rabinowitz，F Brad Johnson，Kai Johnsson，Mathias Ziegler，Xiaolu A Cambronne，Joseph A Baur

线粒体

发表日期：2020

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Thrombospondin-4 regulates vascular inflammation and atherogenesis

血小板反应蛋白-4调节血管炎症和动脉粥样硬化发生

期刊:Circulation Research

影响因子:16.2

doi:10.1161/CIRCRESAHA.110.232371

Frolova, Ella G; Pluskota, Elzbieta; Krukovets, Irene; Burke, Tim; Drumm, Carla; Smith, Jonathan D; Blech, Lauren; Febbraio, Maria; Bornstein, Paul; Plow, Edward F; Stenina, Olga I

Expression in SPARC-null mice of collagen type I lacking the globular domain of the α1(I) N-propeptide results in abdominal hernias and loss of dermal collagen

在SPARC基因敲除小鼠中表达缺乏α1(I) N-前肽球状结构域的I型胶原蛋白会导致腹疝和真皮胶原蛋白丢失。

期刊:Matrix Biology

影响因子:4.8

doi:10.1016/j.matbio.2010.08.002

Card, Lauren; Henderson, Nikki; Zhang, Yuhua; Bornstein, Paul; Bradshaw, Amy D

ARC

发表日期：2010

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Mice that lack matrix metalloproteinase-9 display delayed wound healing associated with delayed reepithelization and disordered collagen fibrillogenesis

缺乏基质金属蛋白酶-9的小鼠表现出伤口愈合延迟，伴有上皮再生延迟和胶原纤维形成紊乱。

期刊:Matrix Biology

影响因子:4.8

doi:10.1016/j.matbio.2009.01.001

Kyriakides, Themis R; Wulsin, Drausin; Skokos, Eleni A; Fleckman, Philip; Pirrone, Annalisa; Shipley, J Michael; Senior, Robert M; Bornstein, Paul

发表日期：2009

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Thrombospondins function as regulators of angiogenesis

血小板反应蛋白在血管生成中发挥调节作用。

期刊:Journal of Cell Communication and Signaling

影响因子:3.9

doi:10.1007/s12079-009-0060-8

Matricellular proteins: an overview

细胞外基质蛋白：概述

期刊:Journal of Cell Communication and Signaling

影响因子:3.9

doi:10.1007/s12079-009-0069-z

Macrophage fusion, giant cell formation, and the foreign body response require matrix metalloproteinase 9

巨噬细胞融合、巨细胞形成和异物反应都需要基质金属蛋白酶9。

期刊:Journal of Leukocyte Biology

影响因子:3.1

doi:10.1189/jlb.1008588

MacLauchlan, Susan; Skokos, Eleni A; Meznarich, Norman; Zhu, Dana H; Raoof, Sana; Shipley, J Michael; Senior, Robert M; Bornstein, Paul; Kyriakides, Themis R

Disruption of the thrombospondin-2 gene alters the lamellar morphology but does not permit vascularization of the adult mouse lumbar disc

血小板反应蛋白-2基因的缺失会改变成年小鼠腰椎间盘的层状结构，但不会促进其血管化。

期刊:Arthritis Research & Therapy

影响因子:4.6

doi:10.1186/ar2483

Gruber, Helen E; Bornstein, Paul; Sage, E Helene; Ingram, Jane A; Zinchenko, Natalia; Norton, H James; Hanley, Edward N Jr

血小板

发表日期：2008

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Thrombospondin-2 modulates extracellular matrix remodeling during physiological angiogenesis

血小板反应蛋白-2在生理性血管生成过程中调节细胞外基质重塑。

期刊:American Journal of Pathology

影响因子:3.6

doi:10.2353/ajpath.2008.080128

Krady, Marie M; Zeng, Jianmin; Yu, Jun; MacLauchlan, Susan; Skokos, Eleni A; Tian, Weiming; Bornstein, Paul; Sessa, William C; Kyriakides, Themis R

Thrombospondin-1 inhibits VEGF levels in the ovary directly by binding and internalization via the low density lipoprotein receptor-related protein-1 (LRP-1).

血小板反应蛋白-1通过低密度脂蛋白受体相关蛋白-1 (LRP-1) 结合和内化，直接抑制卵巢中的 VEGF 水平

期刊:Journal of Cellular Physiology

影响因子:4

doi:10.1002/jcp.20904

Greenaway James, Lawler Jack, Moorehead Roger, Bornstein Paul, Lamarre Jonathan, Petrik Jim

其它

发表日期：2007

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SLC25A51 is a mammalian mitochondrial NAD+ transporter

SLC25A51 是一种哺乳动物线粒体 NAD+ 转运蛋白

Thrombospondin-4 regulates vascular inflammation and atherogenesis

血小板反应蛋白-4调节血管炎症和动脉粥样硬化发生

Expression in SPARC-null mice of collagen type I lacking the globular domain of the α1(I) N-propeptide results in abdominal hernias and loss of dermal collagen

在SPARC基因敲除小鼠中表达缺乏α1(I) N-前肽球状结构域的I型胶原蛋白会导致腹疝和真皮胶原蛋白丢失。

Mice that lack matrix metalloproteinase-9 display delayed wound healing associated with delayed reepithelization and disordered collagen fibrillogenesis

缺乏基质金属蛋白酶-9的小鼠表现出伤口愈合延迟，伴有上皮再生延迟和胶原纤维形成紊乱。

Thrombospondins function as regulators of angiogenesis

血小板反应蛋白在血管生成中发挥调节作用。

Matricellular proteins: an overview

细胞外基质蛋白：概述

Macrophage fusion, giant cell formation, and the foreign body response require matrix metalloproteinase 9

巨噬细胞融合、巨细胞形成和异物反应都需要基质金属蛋白酶9。

Disruption of the thrombospondin-2 gene alters the lamellar morphology but does not permit vascularization of the adult mouse lumbar disc

血小板反应蛋白-2基因的缺失会改变成年小鼠腰椎间盘的层状结构，但不会促进其血管化。

Thrombospondin-2 modulates extracellular matrix remodeling during physiological angiogenesis

血小板反应蛋白-2在生理性血管生成过程中调节细胞外基质重塑。

Thrombospondin-1 inhibits VEGF levels in the ovary directly by binding and internalization via the low density lipoprotein receptor-related protein-1 (LRP-1).

血小板反应蛋白-1通过低密度脂蛋白受体相关蛋白-1 (LRP-1) 结合和内化，直接抑制卵巢中的 VEGF 水平