Boyman Liron相关文献与解析，生知库 | 链接生命科学的每一步

Constitutive deletion of the obscurin-Ig58/59 domains induces atrial remodeling and Ca2+-based arrhythmogenesis.

obscurin-Ig58/59 结构域的组成性缺失可诱发心房重构和 Ca2+ 介导的心律失常

doi:10.1172/jci.insight.184202

Grogan Alyssa, Brong Annie, Joca Humberto C, Boyman Liron, Kaplan Aaron D, Ward Christopher W, Greiser Maura, Kontrogianni-Konstantopoulos Aikaterini

其它

发表日期：2025

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Disruption of P2Y2 Signaling Promotes Breast Tumor Cell Dissemination by Reducing ATP-Dependent Calcium Elevation and Actin Localization to Cell Junctions.

P2Y2 信号传导的破坏通过减少 ATP 依赖性钙离子升高和肌动蛋白在细胞连接处的定位来促进乳腺肿瘤细胞扩散

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms26094286

Mull Makenzy L, Pratt Stephen J P, Thompson Keyata N, Annis David A, Lee Rachel M, Ju Julia A, Gilchrist Darin E, Stemberger Megan B, Boyman Liron, Lederer William J, Vitolo Michele I, Martin Stuart S

Ryanodine receptor stabilization therapy suppresses Ca(2+)- based arrhythmias in a novel model of metabolic HFpEF

在一种新型代谢性射血分数保留型心力衰竭（HFpEF）模型中，雷诺定受体稳定疗法可抑制钙离子（Ca²⁺）介导的心律失常。

期刊:Journal of Molecular and Cellular Cardiology

影响因子:4.7

doi:10.1016/j.yjmcc.2024.07.006

Kaplan, Aaron D; Boyman, Liron; Ward, Christopher W; Lederer, W Jonathan; Greiser, Maura

Ryanodine Receptor Stabilization Therapy Suppresses Ca (2+) -Based Arrhythmias in a Novel Model of Metabolic HFpEF

雷诺定受体稳定疗法可抑制代谢性射血分数保留型心力衰竭（HFpEF）新模型中的钙离子介导的心律失常。

期刊:

影响因子:

doi:10.1101/2023.06.21.544411

Kaplan, Aaron D; Boyman, Liron; Ward, Christopher W; Lederer, W Jonathan; Greiser, Maura

Na (+) /K (+) ATPase-Ca (v) 1.2 nanodomain differentially regulates intracellular [Na (+) ], [Ca (2+) ] and local adrenergic signaling in cardiac myocytes

Na(+)/K(+)ATPase-Ca(v)1.2纳米结构域差异性地调节心肌细胞内的[Na(+)]、[Ca(2+)]和局部肾上腺素能信号传导

期刊:

影响因子:

doi:10.1101/2023.08.31.553598

Karbowski, Mariusz; Boyman, Liron; Garber, Libet; Joca, Humberto C; Verhoeven, Nicolas; Coleman, Andrew K; Ward, Christopher W; Lederer, W Jonathan; Greiser, Maura

Understanding the Dynamics of the Transient and Permanent Opening Events of the Mitochondrial Permeability Transition Pore with a Novel Stochastic Model

利用新型随机模型理解线粒体通透性转换孔瞬态和永久开放事件的动态过程

期刊:Membranes

影响因子:3.6

doi:10.3390/membranes12050494

Yalamanchili, Keertana; Afzal, Nasrin; Boyman, Liron; Mannella, Carmen A; Lederer, W Jonathan; Jafri, M Saleet

线粒体

发表日期：2022

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Calcium Signaling Silencing in Atrial Fibrillation: Implications for Atrial Sodium Homeostasis

心房颤动中钙信号沉默：对心房钠稳态的影响

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms221910513

Kaplan, Aaron D; Joca, Humberto C; Boyman, Liron; Greiser, Maura

信号转导

发表日期：2021

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Calcium influx through the mitochondrial calcium uniporter holocomplex, MCU(cx)

钙离子通过线粒体钙单向转运蛋白全复合物MCU(cx)流入

期刊:Journal of Molecular and Cellular Cardiology

影响因子:4.7

doi:10.1016/j.yjmcc.2020.10.015

Boyman, Liron; Greiser, Maura; Lederer, W Jonathan

How the mitochondrial calcium uniporter complex (MCU(cx)) works

线粒体钙单向转运蛋白复合物 (MCU(cx)) 的工作原理

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.2015886117

Boyman, Liron; Lederer, W Jonathan

Outer mitochondrial membrane E3Â Ub ligase MARCH5 controls de novo peroxisome biogenesis.

线粒体外膜 E3Â 泛素连接酶 MARCH5 控制过氧化物酶体的从头生物合成

Constitutive deletion of the obscurin-Ig58/59 domains induces atrial remodeling and Ca2+-based arrhythmogenesis.

obscurin-Ig58/59 结构域的组成性缺失可诱发心房重构和 Ca2+ 介导的心律失常

Disruption of P2Y2 Signaling Promotes Breast Tumor Cell Dissemination by Reducing ATP-Dependent Calcium Elevation and Actin Localization to Cell Junctions.

P2Y2 信号传导的破坏通过减少 ATP 依赖性钙离子升高和肌动蛋白在细胞连接处的定位来促进乳腺肿瘤细胞扩散

Ryanodine receptor stabilization therapy suppresses Ca(2+)- based arrhythmias in a novel model of metabolic HFpEF

在一种新型代谢性射血分数保留型心力衰竭（HFpEF）模型中，雷诺定受体稳定疗法可抑制钙离子（Ca²⁺）介导的心律失常。

Ryanodine Receptor Stabilization Therapy Suppresses Ca (2+) -Based Arrhythmias in a Novel Model of Metabolic HFpEF

雷诺定受体稳定疗法可抑制代谢性射血分数保留型心力衰竭（HFpEF）新模型中的钙离子介导的心律失常。

Na (+) /K (+) ATPase-Ca (v) 1.2 nanodomain differentially regulates intracellular [Na (+) ], [Ca (2+) ] and local adrenergic signaling in cardiac myocytes

Na(+)/K(+)ATPase-Ca(v)1.2纳米结构域差异性地调节心肌细胞内的[Na(+)]、[Ca(2+)]和局部肾上腺素能信号传导

Understanding the Dynamics of the Transient and Permanent Opening Events of the Mitochondrial Permeability Transition Pore with a Novel Stochastic Model

利用新型随机模型理解线粒体通透性转换孔瞬态和永久开放事件的动态过程

Calcium Signaling Silencing in Atrial Fibrillation: Implications for Atrial Sodium Homeostasis

心房颤动中钙信号沉默：对心房钠稳态的影响

Calcium influx through the mitochondrial calcium uniporter holocomplex, MCU(cx)

钙离子通过线粒体钙单向转运蛋白全复合物MCU(cx)流入

How the mitochondrial calcium uniporter complex (MCU(cx)) works

线粒体钙单向转运蛋白复合物 (MCU(cx)) 的工作原理