Briva相关文献与解析，生知库 | 链接生命科学的每一步

Martín Angulo, Agustina Vacca, Romina Rodríguez, María Noel Marin, Ana Laura Suárez, Gissel Jorge, Oscar Nosiglia, Victoria Cambón, Anaclara Ríos, Matías Iglesias, Mariana Seija, Carlos Escande, Javier Hurtado, Arturo Briva

发表日期：2022

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Mechanical Risks of Ventilator Sharing in the COVID-19 Era: A Simulation-Based Study

新冠疫情时代呼吸机共享的机械风险：一项基于模拟的研究

期刊:

影响因子:

doi:10.1016/j.arbres.2020.06.006

Angulo, Martín; Beltramelli, Rodrigo; Amarelle, Luciano; Alzugaray, Pedro; Briva, Arturo; Santos, Cristina

Effects of hypercapnia in acute respiratory distress syndrome

高碳酸血症对急性呼吸窘迫综合征的影响

期刊:Annals of Translational Medicine

影响因子:

doi:10.21037/atm.2018.01.09

Nin, Nicolás; Angulo, Martín; Briva, Arturo

发表日期：2018

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Spectral phasor analysis of LAURDAN fluorescence in live A549 lung cells to study the hydration and time evolution of intracellular lamellar body-like structures

对活体 A549 肺细胞中的 LAURDAN 荧光进行光谱相量分析，以研究细胞内层状体状结构的水化和时间演变

期刊:

影响因子:3.3

doi:10.1016/j.bbamem.2016.07.017

Leonel Malacrida, Soledad Astrada, Arturo Briva, Mariela Bollati-Fogolín, Enrico Gratton, Luis A Bagatolli

Lung protection: an intervention for tidal volume reduction in a teaching intensive care unit

肺保护：教学重症监护病房中潮气量减少的干预措施

期刊:Revista Brasileira de Terapia Intensiva

影响因子:

doi:10.5935/0103-507X.20160067

Briva, Arturo; Gaiero, Cristina

发表日期：2016

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Hypoxia leads to Na,K-ATPase downregulation via Ca(2+) release-activated Ca(2+) channels and AMPK activation

缺氧通过 Ca(2+) 释放激活的 Ca(2+) 通道和 AMPK 激活导致 Na,K-ATPase 下调

期刊:Molecular and Cellular Biology

影响因子:3.2

doi:10.1128/MCB.05114-11

Galina A Gusarova, Humberto E Trejo, Laura A Dada, Arturo Briva, Lynn C Welch, Robert B Hamanaka, Gökhan M Mutlu, Navdeep S Chandel, Murali Prakriya, Jacob I Sznajder

[Permissive and non-permissive hypercapnia: mechanisms of action and consequences of high carbon dioxide levels]

[允许性高碳酸血症和非允许性高碳酸血症：高二氧化碳水平的作用机制和后果]

期刊:Archivos De Bronconeumologia

影响因子:9.2

doi:10.1016/j.arbres.2010.01.009

Briva, Arturo; Lecuona, Emilia; Sznajder, Jacob I

发表日期：2010

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Insulin regulates alveolar epithelial function by inducing Na+/K+-ATPase translocation to the plasma membrane in a process mediated by the action of Akt

胰岛素通过诱导Na+/K+-ATPase转位至质膜来调节肺泡上皮功能，这一过程由Akt介导。

期刊:Journal of Cell Science

影响因子:3.6

doi:10.1242/jcs.066464

Comellas, Alejandro P; Kelly, Aileen M; Trejo, Humberto E; Briva, Arturo; Lee, Joyce; Sznajder, Jacob I; Dada, Laura A

免疫/内分泌

发表日期：2010

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Extracellular signal-regulated kinase (ERK) participates in the hypercapnia-induced Na,K-ATPase downregulation

细胞外信号调节激酶（ERK）参与高碳酸血症诱导的Na,K-ATP酶下调

期刊:FEBS Letters

影响因子:3

doi:10.1016/j.febslet.2010.08.002

Welch, Lynn C; Lecuona, Emilia; Briva, Arturo; Trejo, Humberto E; Dada, Laura A; Sznajder, Jacob I

Endothelin-1 impairs alveolar epithelial function via endothelial ETB receptor

内皮素-1通过内皮细胞ETB受体损害肺泡上皮功能。

期刊:American Journal of Respiratory and Critical Care Medicine

影响因子:19.4

doi:10.1164/rccm.200804-540OC

Comellas, Alejandro P; Briva, Arturo; Dada, Laura A; Butti, Maria L; Trejo, Humberto E; Yshii, Cecilia; Azzam, Zaher S; Litvan, Juan; Chen, Jiwang; Lecuona, Emilia; Pesce, Liuska M; Yanagisawa, Masashi; Sznajder, Jacob I

Peripheral and respiratory muscle impairment during murine acute lung injury

小鼠急性肺损伤期间的外周和呼吸肌损伤

Mechanical Risks of Ventilator Sharing in the COVID-19 Era: A Simulation-Based Study

新冠疫情时代呼吸机共享的机械风险：一项基于模拟的研究

Effects of hypercapnia in acute respiratory distress syndrome

高碳酸血症对急性呼吸窘迫综合征的影响

Spectral phasor analysis of LAURDAN fluorescence in live A549 lung cells to study the hydration and time evolution of intracellular lamellar body-like structures

对活体 A549 肺细胞中的 LAURDAN 荧光进行光谱相量分析，以研究细胞内层状体状结构的水化和时间演变

Lung protection: an intervention for tidal volume reduction in a teaching intensive care unit

肺保护：教学重症监护病房中潮气量减少的干预措施

Hypoxia leads to Na,K-ATPase downregulation via Ca(2+) release-activated Ca(2+) channels and AMPK activation

缺氧通过 Ca(2+) 释放激活的 Ca(2+) 通道和 AMPK 激活导致 Na,K-ATPase 下调

[Permissive and non-permissive hypercapnia: mechanisms of action and consequences of high carbon dioxide levels]

[允许性高碳酸血症和非允许性高碳酸血症：高二氧化碳水平的作用机制和后果]

Insulin regulates alveolar epithelial function by inducing Na+/K+-ATPase translocation to the plasma membrane in a process mediated by the action of Akt

胰岛素通过诱导Na+/K+-ATPase转位至质膜来调节肺泡上皮功能，这一过程由Akt介导。

Extracellular signal-regulated kinase (ERK) participates in the hypercapnia-induced Na,K-ATPase downregulation

细胞外信号调节激酶（ERK）参与高碳酸血症诱导的Na,K-ATP酶下调

Endothelin-1 impairs alveolar epithelial function via endothelial ETB receptor

内皮素-1通过内皮细胞ETB受体损害肺泡上皮功能。