Overexpression of VLA-4 in glial-restricted precursors enhances their endothelial docking and induces diapedesis in a mouse stroke model.

在小鼠中风模型中，神经胶质细胞限制性前体细胞中 VLA-4 的过度表达增强了其与内皮细胞的结合，并诱导了白细胞渗出