Burciaga Samuel D相关文献与解析，生知库

DeGolier, Kole R; Pham-Danis, Catherine; Burciaga, Samuel D; Walsh, Zachary H; Brzezinski, Christine; Novak, Amanda J; Leach, Lillie; Cimons, Jennifer; Li, Wei; Zhongyu, Zhu; Dimitrov, Dimiter; Scott-Browne, James P; Kohler, M Eric; Fry, Terry J

CD22

CD2

发表日期：2026

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R213G-Mediated Redistribution of EC-SOD Protects Against Sugen-Hypoxia Pulmonary Hypertension in Mice

R213G介导的EC-SOD重分布可保护小鼠免受Sugen-缺氧性肺动脉高压的影响

期刊:Pulmonary Circulation

影响因子:2.5

doi:10.1002/pul2.70307

Colon Hidalgo, Daniel; Lewis, Caitlin V; Nguyen, Thi-Tina N; Posey, Janelle N; Burciaga, Samuel D; Dee, Nathan; Sul, Christina; Harral, Julie; Irwin, David; Delaney, Cassidy; Nozik, Eva S

T-bet overexpression enhances CAR T cell effector functions and antigen sensitivity

T-bet 过表达增强 CAR T 细胞效应功能和抗原敏感性

期刊:Journal for ImmunoTherapy of Cancer

影响因子:10.6

doi:10.1136/jitc-2024-010962

Cimons, Jennifer M; DeGolier, Kole R; Burciaga, Samuel D; Yarnell, Michael C; Novak, Amanda J; Rivera-Reyes, Amalia M; Kohler, M Eric; Fry, Terry J

Vascular EC-SOD limits the accumulation, proinflammatory profibrotic reprogramming, and hyaluronan binding of interstitial macrophages in hypoxia

血管内皮细胞超氧化物歧化酶 (EC-SOD) 可限制缺氧条件下间质巨噬细胞的聚集、促炎促纤维化重编程以及透明质酸结合。

期刊:American Journal of Physiology-Lung Cellular and Molecular Physiology

影响因子:3.5

doi:10.1152/ajplung.00399.2024

Lewis, Caitlin V; Nguyen, Thi-Tina N; Porfilio, Timothy E; Burciaga, Samuel D; Posey, Janelle N; Jordan, Mariah; Colon Hidalgo, Daniel; Stenmark, Kurt R; Mickael, Claudia; Sul, Christina; Oberley-Deegan, Rebecca E; Delaney, Cassidy; Nozik, Eva S

Lung EC-SOD Overexpression Prevents Hypoxia-Induced Platelet Activation and Lung Platelet Accumulation

肺内皮细胞超氧化物歧化酶（EC-SOD）过表达可预防缺氧诱导的血小板活化和肺血小板聚集

期刊:Antioxidants

影响因子:6.6

doi:10.3390/antiox13080975

Colon Hidalgo, Daniel; Jordan, Mariah; Posey, Janelle N; Burciaga, Samuel D; Nguyen, Thi-Tina N; Sul, Christina; Lewis, Caitlin V; Delaney, Cassidy; Nozik, Eva S

Redistribution of SOD3 expression due to R213G polymorphism affects pulmonary interstitial macrophage reprogramming in response to hypoxia.

由于 R213G 多态性导致 SOD3 表达重新分布，影响肺间质巨噬细胞在缺氧反应中的重编程

期刊:Physiological Genomics

影响因子:2.5

doi:10.1152/physiolgenomics.00078.2024

Lewis Caitlin V, Garcia Anastacia M, Burciaga Samuel D, Posey Janelle N, Jordan Mariah, Nguyen Thi-Tina N, Stenmark Kurt R, Mickael Claudia, Sul Christina, Delaney Cassidy, Nozik Eva S

细胞生物学

发表日期：2024

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Rational redesign of antigen binding domain improves in vivo efficacy of the CD22-CAR

对抗原结合域进行合理重新设计可提高CD22-CAR的体内疗效。

R213G-Mediated Redistribution of EC-SOD Protects Against Sugen-Hypoxia Pulmonary Hypertension in Mice

R213G介导的EC-SOD重分布可保护小鼠免受Sugen-缺氧性肺动脉高压的影响

T-bet overexpression enhances CAR T cell effector functions and antigen sensitivity

T-bet 过表达增强 CAR T 细胞效应功能和抗原敏感性

Vascular EC-SOD limits the accumulation, proinflammatory profibrotic reprogramming, and hyaluronan binding of interstitial macrophages in hypoxia

血管内皮细胞超氧化物歧化酶 (EC-SOD) 可限制缺氧条件下间质巨噬细胞的聚集、促炎促纤维化重编程以及透明质酸结合。

Lung EC-SOD Overexpression Prevents Hypoxia-Induced Platelet Activation and Lung Platelet Accumulation

肺内皮细胞超氧化物歧化酶（EC-SOD）过表达可预防缺氧诱导的血小板活化和肺血小板聚集

Redistribution of SOD3 expression due to R213G polymorphism affects pulmonary interstitial macrophage reprogramming in response to hypoxia.

由于 R213G 多态性导致 SOD3 表达重新分布，影响肺间质巨噬细胞在缺氧反应中的重编程