Single point mutations in the helicase domain of the NS3 protein enhance dengue virus replicative capacity in human monocyte-derived dendritic cells and circumvent the type I interferon response

NS3蛋白解旋酶结构域的单点突变可增强登革病毒在人单核细胞来源的树突状细胞中的复制能力，并规避I型干扰素反应。