Cabiscol相关文献与解析，生知库 | 链接生命科学的每一步

Pazos-Gil Maria, Medina-Carbonero Marta, Sanz-AlcÃ¡zar Arabela, Portillo-Carrasquer Marta, Oliveira-Jorge Luiza, HernÃ¡ndez Gonzalo, SÃ¡nchez Mayka, Delaspre Fabien, Cabiscol Elisa, Ros Joaquim, Tamarit Jordi

发表日期：2026

文献求助收藏

Corrigendum to "Deciphering the ferroptosis pathways in dorsal root ganglia of Friedreich ataxia models. The role of LKB1/AMPK, KEAP1, and GSK3β in the impairment of the NRF2 response" [Redox Biol. 76 (2024) 103339]

对“解读弗里德赖希共济失调模型背根神经节中的铁死亡通路。LKB1/AMPK、KEAP1 和 GSK3β 在 NRF2 反应受损中的作用”[Redox Biol. 76 (2024) 103339]的更正

期刊:Redox Biology

影响因子:11.9

doi:10.1016/j.redox.2025.103583

Sanz-Alcázar, Arabela; Portillo-Carrasquer, Marta; Delaspre, Fabien; Pazos-Gil, Maria; Tamarit, Jordi; Ros, Joaquim; Cabiscol, Elisa

Endogenous mitochondrial hydrogen peroxide regulates neurogenesis during cortical development.

内源性线粒体过氧化氢在皮层发育过程中调节神经发生。

期刊:Redox Biology

影响因子:11.9

doi:10.1016/j.redox.2025.103940

Mengual Regina, Bobo-JimÃ©nez VerÃ³nica, RodrÃguez Cristina, Lapresa Rebeca, GarcÃa-RodrÃguez DarÃo, JimÃ©nez-Blasco Daniel, Cabiscol Elisa, Ros Joaquim, Delgado-Esteban MarÃa, BolaÃ±os Juan P, Ãngeles Almeida

Correction: Mitochondrial impairment, decreased Sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich ataxia models

更正：弗里德赖希共济失调模型中背根神经节线粒体功能障碍、Sirtuin活性降低和蛋白质乙酰化

期刊:Cellular and Molecular Life Sciences

影响因子:6.2

doi:10.1007/s00018-025-05948-7

Sanz-Alcázar, Arabela; Britti, Elena; Delaspre, Fabien; Medina-Carbonero, Marta; Pazos-Gil, Maria; Tamarit, Jordi; Ros, Joaquim; Cabiscol, Elisa

Deciphering the ferroptosis pathways in dorsal root ganglia of Friedreich ataxia models. The role of LKB1/AMPK, KEAP1, and GSK3β in the impairment of the NRF2 response

解读弗里德赖希共济失调模型背根神经节中的铁死亡途径。LKB1/AMPK、KEAP1 和 GSK3β 在 NRF2 反应受损中的作用

期刊:Redox Biology

影响因子:10.7

doi:10.1016/j.redox.2024.103339

Arabela Sanz-Alcázar, Marta Portillo-Carrasquer, Fabien Delaspre, Maria Pazos-Gil, Jordi Tamarit, Joaquim Ros, Elisa Cabiscol

Mitochondrial impairment, decreased sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich Ataxia models

弗里德赖希共济失调模型中的线粒体损伤、sirtuin 活性降低和背根神经节中的蛋白质乙酰化

期刊:Cellular and Molecular Life Sciences

影响因子:6.2

doi:10.1007/s00018-023-05064-4

Arabela Sanz-Alcázar, Elena Britti, Fabien Delaspre, Marta Medina-Carbonero, Maria Pazos-Gil, Jordi Tamarit, Joaquim Ros, Elisa Cabiscol

Oxidative stress-induced FAK activation contributes to uterine serous carcinoma aggressiveness

氧化应激诱导的 FAK 激活导致子宫浆液性癌的侵袭性

期刊:Molecular Oncology

影响因子:5

doi:10.1002/1878-0261.13346

Isabel C Lopez-Mejia, Jordi Pijuan, Raúl Navaridas, Maria Santacana, Sònia Gatius, Ana Velasco, Gerard Castellà, Anaïs Panosa, Elisa Cabiscol, Miquel Pinyol, Laura Coll, Núria Bonifaci, Laura Plata Peña, August Vidal, Alberto Villanueva, Eloi Gari, David Llobet-Navàs, Lluis Fajas, Xavier Matias-Guiu

Mice harboring the FXN I151F pathological point mutation present decreased frataxin levels, a Friedreich ataxia-like phenotype, and mitochondrial alterations

携带 FXN I151F 病理点突变的小鼠表现出 frataxin 水平降低、弗里德赖希共济失调样表型和线粒体改变

期刊:Cellular and Molecular Life Sciences

影响因子:6.2

doi:10.1007/s00018-021-04100-5

Marta Medina-Carbonero, Arabela Sanz-Alcázar, Elena Britti, Fabien Delaspre, Elisa Cabiscol, Joaquim Ros, Jordi Tamarit

信号转导

发表日期：2022

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Mitochondrial Localization of the Yeast Forkhead Factor Hcm1

酵母叉头因子 Hcm1 的线粒体定位

期刊:International Journal of Molecular Sciences

影响因子:0.7

doi:10.3390/ijms21249574

María José Rodríguez Colman, Joaquim Ros, Elisa Cabiscol

Loss of glutathione redox homeostasis impairs proteostasis by inhibiting autophagy-dependent protein degradation.

谷胱甘肽氧化还原稳态的丧失会抑制自噬依赖性蛋白质降解，从而损害蛋白质稳态

期刊:Cell Death and Differentiation

影响因子:15.4

doi:10.1038/s41418-018-0270-9

Guerrero-GÃ³mez David, Mora-Lorca JosÃ© Antonio, SÃ¡enz-Narciso Beatriz, Naranjo-Galindo Francisco JosÃ©, MuÃ±oz-Lobato Fernando, Parrado-FernÃ¡ndez Cristina, Goikolea Julen, Cedazo-Minguez Ãngel, Link Christopher D, Neri Christian, Sequedo MarÃa Dolores, VÃ¡zquez-Manrique Rafael P, FernÃ¡ndez-SuÃ¡rez Elena, Goder Veit, PanÃ© Roser, Cabiscol Elisa, Askjaer Peter, Cabello Juan, Miranda-Vizuete Antonio

Pathological frataxin deficiency in mice causes tissue-specific alterations in iron homeostasis.

小鼠病理性弗拉塔辛缺乏会导致组织特异性的铁稳态改变。

Corrigendum to "Deciphering the ferroptosis pathways in dorsal root ganglia of Friedreich ataxia models. The role of LKB1/AMPK, KEAP1, and GSK3β in the impairment of the NRF2 response" [Redox Biol. 76 (2024) 103339]

对“解读弗里德赖希共济失调模型背根神经节中的铁死亡通路。LKB1/AMPK、KEAP1 和 GSK3β 在 NRF2 反应受损中的作用”[Redox Biol. 76 (2024) 103339]的更正

Endogenous mitochondrial hydrogen peroxide regulates neurogenesis during cortical development.

内源性线粒体过氧化氢在皮层发育过程中调节神经发生。

Correction: Mitochondrial impairment, decreased Sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich ataxia models

更正：弗里德赖希共济失调模型中背根神经节线粒体功能障碍、Sirtuin活性降低和蛋白质乙酰化

Deciphering the ferroptosis pathways in dorsal root ganglia of Friedreich ataxia models. The role of LKB1/AMPK, KEAP1, and GSK3β in the impairment of the NRF2 response

解读弗里德赖希共济失调模型背根神经节中的铁死亡途径。LKB1/AMPK、KEAP1 和 GSK3β 在 NRF2 反应受损中的作用

Mitochondrial impairment, decreased sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich Ataxia models

弗里德赖希共济失调模型中的线粒体损伤、sirtuin 活性降低和背根神经节中的蛋白质乙酰化

Oxidative stress-induced FAK activation contributes to uterine serous carcinoma aggressiveness

氧化应激诱导的 FAK 激活导致子宫浆液性癌的侵袭性

Mice harboring the FXN I151F pathological point mutation present decreased frataxin levels, a Friedreich ataxia-like phenotype, and mitochondrial alterations

携带 FXN I151F 病理点突变的小鼠表现出 frataxin 水平降低、弗里德赖希共济失调样表型和线粒体改变

Mitochondrial Localization of the Yeast Forkhead Factor Hcm1

酵母叉头因子 Hcm1 的线粒体定位

Loss of glutathione redox homeostasis impairs proteostasis by inhibiting autophagy-dependent protein degradation.

谷胱甘肽氧化还原稳态的丧失会抑制自噬依赖性蛋白质降解，从而损害蛋白质稳态