Cacalano Nicholas A相关文献与解析，生知库

Specific bone region localization of osteolytic versus osteoblastic lesions in a patient-derived xenograft model of bone metastatic prostate cancer

在前列腺癌骨转移患者来源的异种移植模型中，溶骨性病变与成骨性病变在特定骨区域的定位

doi:10.1016/j.ajur.2016.09.001

Hirata, Takeshi; Park, Seung Chol; Muldong, Michelle T; Wu, Christina N; Yamaguchi, Tomonori; Strasner, Amy; Raheem, Omer; Kumon, Hiromi; Sah, Robert L; Cacalano, Nicholas A; Jamieson, Catriona H M; Kane, Christopher J; Masuda, Koichi; Kulidjian, Anna A; Jamieson, Christina A M

PCSD1, a new patient-derived model of bone metastatic prostate cancer, is castrate-resistant in the bone-niche.

PCSD1 是一种新的源自患者的骨转移性前列腺癌模型，在骨微环境中具有去势抵抗性

期刊:Journal of Translational Medicine

影响因子:7.5

doi:10.1186/s12967-014-0275-1

Godebu Elana, Muldong Michelle, Strasner Amy, Wu Christina N, Park Seung Chol, Woo Jason R, Ma Wenxue, Liss Michael A, Hirata Takeshi, Raheem Omer, Cacalano Nicholas A, Kulidjian Anna A, Jamieson Christina A M

Strategies to rescue mesenchymal stem cells (MSCs) and dental pulp stem cells (DPSCs) from NK cell mediated cytotoxicity

拯救间充质干细胞（MSCs）和牙髓干细胞（DPSCs）免受NK细胞介导的细胞毒性的策略

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0009874

Jewett, Anahid; Arasteh, Aida; Tseng, Han-Ching; Behel, Armin; Arasteh, Hobie; Yang, Wendy; Cacalano, Nicholas A; Paranjpe, Avina

N-acetyl cysteine mediates protection from 2-hydroxyethyl methacrylate induced apoptosis via nuclear factor kappa B-dependent and independent pathways: potential involvement of JNK

N-乙酰半胱氨酸通过核因子κB依赖性和非依赖性途径介导对2-羟乙基甲基丙烯酸酯诱导的细胞凋亡的保护作用：JNK可能参与其中

期刊:Toxicological Sciences

影响因子:4.1

doi:10.1093/toxsci/kfp010

Paranjpe, Avina; Cacalano, Nicholas A; Hume, Wyatt R; Jewett, Anahid

Potential contribution of naÃ¯ve immune effectors to oral tumor resistance: role in synergistic induction of VEGF, IL-6, and IL-8 secretion.

幼稚免疫效应细胞对口服肿瘤耐药性的潜在贡献：在协同诱导 VEGF、IL-6 和 IL-8 分泌中的作用

期刊:Cancer Immunology Immunotherapy

影响因子:5.1

doi:10.1007/s00262-007-0375-3

Teruel Antonia, Romero Marcela, Cacalano Nicholas A, Head Christian, Jewett Anahid

Epidermal growth factor receptor vIII expression in U87 glioblastoma cells alters their proteasome composition, function, and response to irradiation

U87胶质母细胞瘤细胞中表皮生长因子受体vIII的表达会改变其蛋白酶体的组成、功能以及对辐射的反应。

期刊:Molecular Cancer Research

影响因子:4.7

doi:10.1158/1541-7786.MCR-07-0313

Kim, Kwanghee; Brush, James M; Watson, Philip A; Cacalano, Nicholas A; Iwamoto, Keisuke S; McBride, William H

N-acetylcysteine protects dental pulp stromal cells from HEMA-induced apoptosis by inducing differentiation of the cells.

N-乙酰半胱氨酸通过诱导细胞分化来保护牙髓基质细胞免受HEMA诱导的细胞凋亡

期刊:Free Radical Biology and Medicine

影响因子:8.2

doi:10.1016/j.freeradbiomed.2007.07.011

Paranjpe Avina, Cacalano Nicholas A, Hume Wyatt R, Jewett Anahid

Inhibition of nuclear factor kappa B (NFkappaB) activity in oral tumor cells prevents depletion of NK cells and increases their functional activation

抑制口腔肿瘤细胞中的核因子κB (NF-κB) 活性可防止NK细胞耗竭并增强其功能活化。

期刊:Cancer Immunology Immunotherapy

影响因子:5.1

doi:10.1007/s00262-005-0093-7

Jewett, Anahid; Cacalano, Nicholas A; Teruel, Antonia; Romero, Marcela; Rashedi, Marjan; Wang, Meiying; Nakamura, Hiromi

Regulation of Natural Killer Cell Function by STAT3

STAT3对自然杀伤细胞功能的调控

Specific bone region localization of osteolytic versus osteoblastic lesions in a patient-derived xenograft model of bone metastatic prostate cancer

在前列腺癌骨转移患者来源的异种移植模型中，溶骨性病变与成骨性病变在特定骨区域的定位

PCSD1, a new patient-derived model of bone metastatic prostate cancer, is castrate-resistant in the bone-niche.

PCSD1 是一种新的源自患者的骨转移性前列腺癌模型，在骨微环境中具有去势抵抗性

Strategies to rescue mesenchymal stem cells (MSCs) and dental pulp stem cells (DPSCs) from NK cell mediated cytotoxicity

拯救间充质干细胞（MSCs）和牙髓干细胞（DPSCs）免受NK细胞介导的细胞毒性的策略

N-acetyl cysteine mediates protection from 2-hydroxyethyl methacrylate induced apoptosis via nuclear factor kappa B-dependent and independent pathways: potential involvement of JNK

N-乙酰半胱氨酸通过核因子κB依赖性和非依赖性途径介导对2-羟乙基甲基丙烯酸酯诱导的细胞凋亡的保护作用：JNK可能参与其中

Potential contribution of naÃ¯ve immune effectors to oral tumor resistance: role in synergistic induction of VEGF, IL-6, and IL-8 secretion.

幼稚免疫效应细胞对口服肿瘤耐药性的潜在贡献：在协同诱导 VEGF、IL-6 和 IL-8 分泌中的作用

Epidermal growth factor receptor vIII expression in U87 glioblastoma cells alters their proteasome composition, function, and response to irradiation

U87胶质母细胞瘤细胞中表皮生长因子受体vIII的表达会改变其蛋白酶体的组成、功能以及对辐射的反应。

N-acetylcysteine protects dental pulp stromal cells from HEMA-induced apoptosis by inducing differentiation of the cells.

N-乙酰半胱氨酸通过诱导细胞分化来保护牙髓基质细胞免受HEMA诱导的细胞凋亡

Inhibition of nuclear factor kappa B (NFkappaB) activity in oral tumor cells prevents depletion of NK cells and increases their functional activation

抑制口腔肿瘤细胞中的核因子κB (NF-κB) 活性可防止NK细胞耗竭并增强其功能活化。