Cai Weikang相关文献与解析，生知库 | 链接生命科学的每一步

Knockout of the intellectual disability-linked gene Hs6st2 in mice decreases heparan sulfate 6-O-sulfation, impairs dendritic spines of hippocampal neurons, and affects memory

敲除小鼠中与智力障碍相关的基因Hs6st2会降低硫酸乙酰肝素6-O-硫酸化水平，损害海马神经元的树突棘，并影响记忆力。

Moon, Sohyun; Lee, Hiu Ham; Archer-Hartmann, Stephanie; Nagai, Naoko; Mubasher, Zainab; Parappurath, Mahima; Ahmed, Laiba; Ramos, Raddy L; Kimata, Koji; Azadi, Parastoo; Cai, Weikang; Zhao, Jerry Yingtao

神经科学

发表日期：2024

文献求助收藏

Case report: A rare case of simultaneous necrotizing fasciitis of the breast and forearm

病例报告：一例罕见的乳房和前臂同时发生坏死性筋膜炎的病例

期刊:Frontiers in Medicine

影响因子:3

doi:10.3389/fmed.2024.1413593

Xu, Ruofei; Fang, Tingting; Cai, Weikang

发表日期：2024

文献求助收藏

Loss of insulin signaling in astrocytes exacerbates Alzheimer-like phenotypes in a 5xFAD mouse model

星形胶质细胞中胰岛素信号的缺失会加剧5xFAD小鼠模型中的阿尔茨海默病样表型。

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.2220684120

Chen, Wenqiang; Huang, Qian; Lazdon, Ekaterina Katie; Gomes, Antonio; Wong, Marisa; Stephens, Emily; Royal, Tabitha Grace; Frenkel, Dan; Cai, Weikang; Kahn, C Ronald

Neuropeptide Cholecystokinin: A Potential Molecular Link Between Obesity, Gut, and Emotion

神经肽胆囊收缩素：肥胖、肠道和情绪之间潜在的分子联系

期刊:Biological Psychiatry: Global Open Science

影响因子:3.7

doi:10.1016/j.bpsgos.2023.08.010

Huang, Qian; Cai, Weikang

Insulin action in the brain: cell types, circuits, and diseases

胰岛素在大脑中的作用：细胞类型、回路和疾病

期刊:Trends in Neurosciences

影响因子:15.1

doi:10.1016/j.tins.2022.03.001

Chen, Wenqiang; Cai, Weikang; Hoover, Benjamin; Kahn, C Ronald

Peripheral Insulin Regulates a Broad Network of Gene Expression in Hypothalamus, Hippocampus, and Nucleus Accumbens

外周胰岛素调节下丘脑、海马和伏隔核中广泛的基因表达网络

期刊:Diabetes

影响因子:7.5

doi:10.2337/db20-1119

Cai, Weikang; Zhang, Xuemei; Batista, Thiago M; García-Martín, Rubén; Softic, Samir; Wang, Guoxiao; Ramirez, Alfred K; Konishi, Masahiro; O'Neill, Brian T; Kim, Jong Hun; Kim, Jason K; Kahn, C Ronald

Insulin signaling in the hippocampus and amygdala regulates metabolism and neurobehavior

海马体和杏仁核中的胰岛素信号传导调节新陈代谢和神经行为

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.1817391116

Soto, Marion; Cai, Weikang; Konishi, Masahiro; Kahn, C Ronald

Adipocyte Dynamics and Reversible Metabolic Syndrome in Mice with an Inducible Adipocyte-Specific Deletion of the Insulin Receptor

小鼠脂肪细胞特异性胰岛素受体诱导缺失后，脂肪细胞动力学和可逆性代谢综合征

期刊:Cell Metabolism

影响因子:30.9

doi:10.1016/j.cmet.2016.12.008

Sakaguchi, Masaji; Fujisaka, Shiho; Cai, Weikang; Winnay, Jonathon N; Konishi, Masahiro; O'Neill, Brian T; Li, Mengyao; García-Martín, Rubén; Takahashi, Hirokazu; Hu, Jiang; Kulkarni, Rohit N; Kahn, C Ronald

Astrocyte involvement in metabolic regulation and disease

星形胶质细胞参与代谢调节和疾病

Lrtm1: A Novel Sensor of Insulin Signaling and Regulator of Metabolism and Activity.

Lrtm1：一种新型的胰岛素信号传感器和代谢及活动调节器

Knockout of the intellectual disability-linked gene Hs6st2 in mice decreases heparan sulfate 6-O-sulfation, impairs dendritic spines of hippocampal neurons, and affects memory

敲除小鼠中与智力障碍相关的基因Hs6st2会降低硫酸乙酰肝素6-O-硫酸化水平，损害海马神经元的树突棘，并影响记忆力。

Case report: A rare case of simultaneous necrotizing fasciitis of the breast and forearm

病例报告：一例罕见的乳房和前臂同时发生坏死性筋膜炎的病例

Loss of insulin signaling in astrocytes exacerbates Alzheimer-like phenotypes in a 5xFAD mouse model

星形胶质细胞中胰岛素信号的缺失会加剧5xFAD小鼠模型中的阿尔茨海默病样表型。

Neuropeptide Cholecystokinin: A Potential Molecular Link Between Obesity, Gut, and Emotion

神经肽胆囊收缩素：肥胖、肠道和情绪之间潜在的分子联系

Insulin action in the brain: cell types, circuits, and diseases

胰岛素在大脑中的作用：细胞类型、回路和疾病

Peripheral Insulin Regulates a Broad Network of Gene Expression in Hypothalamus, Hippocampus, and Nucleus Accumbens

外周胰岛素调节下丘脑、海马和伏隔核中广泛的基因表达网络

Insulin signaling in the hippocampus and amygdala regulates metabolism and neurobehavior

海马体和杏仁核中的胰岛素信号传导调节新陈代谢和神经行为

Adipocyte Dynamics and Reversible Metabolic Syndrome in Mice with an Inducible Adipocyte-Specific Deletion of the Insulin Receptor

小鼠脂肪细胞特异性胰岛素受体诱导缺失后，脂肪细胞动力学和可逆性代谢综合征