A myosin hypertrophic cardiomyopathy mutation disrupts the super-relaxed state and boosts contractility by enhanced actin attachment
肌球蛋白肥厚型心肌病突变会破坏超松弛状态，并通过增强肌动蛋白附着来提高收缩力。
期刊:Proceedings of the National Academy of Sciences of the United States of America
影响因子:9.1
doi:10.1073/pnas.2521561122
Cail, Robert C; Barua, Bipasha; Báez-Cruz, Faviolla A; Winkelmann, Donald A; Goldman, Yale E; Ostap, E Michael
