日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

N-Glycolylneuraminic acid deficiency worsens cardiac and skeletal muscle pathophysiology in α-sarcoglycan-deficient mice.

N-糖基神经氨酸缺乏会加重α-肌聚糖缺乏小鼠的心脏和骨骼肌病理生理

Martin Paul T, Camboni Marybeth, Xu Rui, Golden Bethannie, Chandrasekharan Kumaran, Wang Chiou-Miin, Varki Ajit, Janssen Paul M L

Immunization with the SDPM1 peptide lowers amyloid plaque burden and improves cognitive function in the APPswePSEN1(A246E) transgenic mouse model of Alzheimer's disease.

用 SDPM1 肽进行免疫接种可降低 APPswePSEN1(A246E) 转基因阿尔茨海默病小鼠模型的淀粉样斑块负荷并改善认知功能

Wang Chiou-Miin, Devries Sarah, Camboni Marybeth, Glass Matthew, Martin Paul T

Overexpression of Galgt2 in skeletal muscle prevents injury resulting from eccentric contractions in both mdx and wild-type mice

在mdx小鼠和野生型小鼠中,骨骼肌中Galgt2的过表达均可预防离心收缩造成的损伤。

Martin, Paul T; Xu, Rui; Rodino-Klapac, Louise R; Oglesbay, Elaine; Camboni, Marybeth; Montgomery, Chrystal L; Shontz, Kim; Chicoine, Louis G; Clark, K Reed; Sahenk, Zarife; Mendell, Jerry R; Janssen, Paul M L

Overexpression of Galgt2 reduces dystrophic pathology in the skeletal muscles of alpha sarcoglycan-deficient mice.

Galgt2 的过度表达可减轻 α-肌聚糖缺乏小鼠骨骼肌中的营养不良病理

Xu Rui, DeVries Sarah, Camboni Marybeth, Martin Paul T

Postnatal overexpression of the CT GalNAc transferase inhibits muscular dystrophy in mdx mice without altering muscle growth or neuromuscular development: evidence for a utrophin-independent mechanism

CT GalNAc转移酶的出生后过表达可抑制mdx小鼠的肌营养不良症,且不影响肌肉生长或神经肌肉发育:这是肌营养不良蛋白非依赖性机制的证据。

Xu, Rui; Camboni, Marybeth; Martin, Paul T

Overexpression of the cytotoxic T cell (CT) carbohydrate inhibits muscular dystrophy in the dyW mouse model of congenital muscular dystrophy 1A

细胞毒性T细胞(CT)碳水化合物的过表达可抑制先天性肌营养不良症1A型dyW小鼠模型中的肌营养不良症。

Xu, Rui; Chandrasekharan, Kumaran; Yoon, Jung Hae; Camboni, Marybeth; Martin, Paul T