Cannons Jennifer L相关文献与解析，生知库

Golec, Dominic P; Gazzinelli-Guimaraes, Pedro H; Chauss, Daniel; Yu, Kang; Nagashima, Hiroyuki; Cruz, Anthony C; Hill, Tom; Ganesan, Sundar; Cannons, Jennifer L; Perry, Jillian K; Nivelo, Luis; Joshi, Ilin; Pereira, Nicolas; Silva Oliveira, Fabrício Marcus; Zheng, Yufan; Jean Pierre, Makheni; Druey, Kirk M; Lack, Justin B; Dang, Eric V; Nutman, Thomas B; Villarino, Alejandro V; O'Shea, John J; Afzali, Behdad; Schwartzberg, Pamela L

T细胞

信号转导

T细胞

A PI3KÎ´-Foxo1-FasL signaling amplification loop rewires CD4(+) T helper cell signaling, differentiation and epigenetic remodeling.

PI3KÎ´-Foxo1-FasL 信号放大环路重塑 CD4(+) T 辅助细胞信号传导、分化和表观遗传重塑

期刊:

影响因子:

doi:10.1101/2024.10.28.620691

Golec Dominic P, Gazzinelli-Guimaraes Pedro, Chauss Daniel, Nagashima Hiroyuki, Yu Kang, Hill Tom, Nivelo Luis, Cannons Jennifer L, Perry Jillian, Joshi Ilin, Pereira Nicolas, Oliveira FabrÃcio Marcus Silva, Cruz Anthony C, Druey Kirk M, Lack Justin B, Nutman Thomas B, Villarino Alejandro V, O'Shea John J, Afzali Behdad, Schwartzberg Pamela L

Mg(2+) regulation of kinase signaling and immune function

Mg(2+)对激酶信号传导和免疫功能的调控

期刊:Journal of Experimental Medicine

影响因子:10.6

doi:10.1084/jem.20181970

Kanellopoulou, Chryssa; George, Alex B; Masutani, Evan; Cannons, Jennifer L; Ravell, Juan C; Yamamoto, Tori N; Smelkinson, Margery G; Jiang, Ping Du; Matsuda-Lennikov, Mami; Reilley, Julie; Handon, Robin; Lee, Ping-Hsien; Miller, J Richard; Restifo, Nicholas P; Zheng, Lixin; Schwartzberg, Pamela L; Young, Matthew; Lenardo, Michael J

Hyperactivated PI3Kδ promotes self and commensal reactivity at the expense of optimal humoral immunity

过度激活的PI3Kδ会促进自身和共生菌的反应性，从而损害最佳的体液免疫功能。

期刊:Nature Immunology

影响因子:27.6

doi:10.1038/s41590-018-0182-3

Preite, Silvia; Cannons, Jennifer L; Radtke, Andrea J; Vujkovic-Cvijin, Ivan; Gomez-Rodriguez, Julio; Volpi, Stefano; Huang, Bonnie; Cheng, Jun; Collins, Nicholas; Reilley, Julie; Handon, Robin; Dobbs, Kerry; Huq, Lutfi; Raman, Indu; Zhu, Chengsong; Li, Quan-Zhen; Li, Ming O; Pittaluga, Stefania; Uzel, Gulbu; Notarangelo, Luigi D; Belkaid, Yasmine; Germain, Ronald N; Schwartzberg, Pamela L

X-Linked Lymphoproliferative Disease Type 1: A Clinical and Molecular Perspective

X连锁淋巴增生性疾病1型：临床和分子视角

期刊:Frontiers in Immunology

影响因子:5.9

doi:10.3389/fimmu.2018.00666

Panchal, Neelam; Booth, Claire; Cannons, Jennifer L; Schwartzberg, Pamela L

发表日期：2018

文献求助收藏

PI3K Orchestrates T Follicular Helper Cell Differentiation in a Context Dependent Manner: Implications for Autoimmunity

PI3K 以依赖于环境的方式调控滤泡辅助性 T 细胞分化：对自身免疫的启示

期刊:Frontiers in Immunology

影响因子:5.9

doi:10.3389/fimmu.2018.03079

Preite, Silvia; Huang, Bonnie; Cannons, Jennifer L; McGavern, Dorian B; Schwartzberg, Pamela L

SAP and Lessons Learned from a Primary Immunodeficiency

SAP及从原发性免疫缺陷中汲取的经验教训

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.1701007

Cannons, Jennifer L; Schwartzberg, Pamela L

免疫/内分泌

发表日期：2017

文献求助收藏

Type I IFN induces binding of STAT1 to Bcl6: divergent roles of STAT family transcription factors in the T follicular helper cell genetic program.

I 型 IFN 诱导 STAT1 与 Bcl6 结合：STAT 家族转录因子在 T 滤泡辅助细胞遗传程序中发挥不同的作用

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.1300675

Nakayamada Shingo, Poholek Amanda C, Lu Kristina T, Takahashi Hayato, Kato Masanari, Iwata Shigeru, Hirahara Kiyoshi, Cannons Jennifer L, Schwartzberg Pamela L, Vahedi Golnaz, Sun Hong-Wei, Kanno Yuka, O'Shea John J

细胞生物学

发表日期：2014

文献求助收藏

Ncking BCR-mediated PI3K activation

Ncking BCR介导的PI3K激活

期刊:EMBO Reports

影响因子:6.2

doi:10.1038/embor.2013.133

Cannons, Jennifer L; Zhao, Fang; Schwartzberg, Pamela L

BCR

发表日期：2013

文献求助收藏

The receptor Ly108 functions as a SAP adaptor-dependent on-off switch for T cell help to B cells and NKT cell development

Ly108受体作为一种SAP衔接蛋白依赖性的开关，调控T细胞对B细胞的辅助作用以及NKT细胞的发育。

期刊:Immunity

影响因子:26.3

doi:10.1016/j.immuni.2012.05.016

Kageyama, Robin; Cannons, Jennifer L; Zhao, Fang; Yusuf, Isharat; Lao, Christopher; Locci, Michela; Schwartzberg, Pamela L; Crotty, Shane

A PI3Kδ-Foxo1-FasL signaling amplification loop rewires CD4+ T cell signaling and differentiation

PI3Kδ-Foxo1-FasL信号放大环路重塑CD4+ T细胞信号传导和分化

A PI3KÎ´-Foxo1-FasL signaling amplification loop rewires CD4(+) T helper cell signaling, differentiation and epigenetic remodeling.

PI3KÎ´-Foxo1-FasL 信号放大环路重塑 CD4(+) T 辅助细胞信号传导、分化和表观遗传重塑

Mg(2+) regulation of kinase signaling and immune function

Mg(2+)对激酶信号传导和免疫功能的调控

Hyperactivated PI3Kδ promotes self and commensal reactivity at the expense of optimal humoral immunity

过度激活的PI3Kδ会促进自身和共生菌的反应性，从而损害最佳的体液免疫功能。

X-Linked Lymphoproliferative Disease Type 1: A Clinical and Molecular Perspective

X连锁淋巴增生性疾病1型：临床和分子视角

PI3K Orchestrates T Follicular Helper Cell Differentiation in a Context Dependent Manner: Implications for Autoimmunity

PI3K 以依赖于环境的方式调控滤泡辅助性 T 细胞分化：对自身免疫的启示

SAP and Lessons Learned from a Primary Immunodeficiency

SAP及从原发性免疫缺陷中汲取的经验教训

Type I IFN induces binding of STAT1 to Bcl6: divergent roles of STAT family transcription factors in the T follicular helper cell genetic program.

I 型 IFN 诱导 STAT1 与 Bcl6 结合：STAT 家族转录因子在 T 滤泡辅助细胞遗传程序中发挥不同的作用

Ncking BCR-mediated PI3K activation

Ncking BCR介导的PI3K激活

The receptor Ly108 functions as a SAP adaptor-dependent on-off switch for T cell help to B cells and NKT cell development

Ly108受体作为一种SAP衔接蛋白依赖性的开关，调控T细胞对B细胞的辅助作用以及NKT细胞的发育。