Carlo Follo相关文献与解析，生知库 | 链接生命科学的每一步

High Expression of the Lysosomal Protease Cathepsin D Confers Better Prognosis in Neuroblastoma Patients by Contrasting EGF-Induced Neuroblastoma Cell Growth

溶酶体蛋白酶组织蛋白酶 D 的高表达与 EGF 诱导的神经母细胞瘤细胞生长相比，可为神经母细胞瘤患者提供更好的预后

期刊:International Journal of Molecular Sciences

影响因子:0.7

doi:10.3390/ijms23094782

Eleonora Secomandi, Amreen Salwa, Chiara Vidoni, Alessandra Ferraresi, Carlo Follo, Ciro Isidoro

Amino acid response by Halofuginone in Cancer cells triggers autophagy through proteasome degradation of mTOR

癌细胞中氟吉诺酮的氨基酸反应通过蛋白酶体降解 mTOR 触发自噬

期刊:Cell Communication and Signaling

影响因子:8.2

doi:10.1186/s12964-019-0354-2

Carlo Follo, Chiara Vidoni, Federica Morani, Alessandra Ferraresi, Christian Seca, Ciro Isidoro

Autophagy facilitates the release of immunogenic signals following chemotherapy in 3D models of mesothelioma

自噬促进间皮瘤 3D 模型中化疗后免疫原性信号的释放

期刊:Molecular Carcinogenesis

影响因子:3

doi:10.1002/mc.23050

Carlo Follo, Yao Cheng, William G Richards, Raphael Bueno, V Courtney Broaddus

Autophagy in 3D In Vitro and Ex Vivo Cancer Models

3D 体外和离体癌症模型中的自噬

期刊:Methods in Molecular Biology

影响因子:

doi:10.1007/978-1-4939-8873-0_31

Carlo Follo, Dario Barbone, William G Richards, Raphael Bueno, V Courtney Broaddus

Inhibition of autophagy initiation potentiates chemosensitivity in mesothelioma

抑制自噬启动可增强间皮瘤的化学敏感性

期刊:Molecular Carcinogenesis

影响因子:3

doi:10.1002/mc.22757

Carlo Follo, Yao Cheng, William G Richards, Raphael Bueno, Virginia Courtney Broaddus

The protein restriction mimetic Resveratrol is an autophagy inducer stronger than amino acid starvation in ovarian cancer cells

蛋白质限制模拟物白藜芦醇是卵巢癌细胞中比氨基酸饥饿更强的自噬诱导剂

期刊:Molecular Carcinogenesis

影响因子:3

doi:10.1002/mc.22711

Alessandra Ferraresi, Rossella Titone, Carlo Follo, Andrea Castiglioni, Giovanna Chiorino, Danny N Dhanasekaran, Ciro Isidoro

Autophagy initiation correlates with the autophagic flux in 3D models of mesothelioma and with patient outcome

自噬启动与间皮瘤 3D 模型中的自噬通量以及患者预后相关

期刊:Autophagy

影响因子:14.6

doi:10.1080/15548627.2016.1173799

Carlo Follo, Dario Barbone, William G Richards, Raphael Bueno, V Courtney Broaddus

Analysis of Gene Expression in 3D Spheroids Highlights a Survival Role for ASS1 in Mesothelioma

3D 球体基因表达分析突出了 ASS1 在间皮瘤中的生存作用

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0150044

Dario Barbone, Loes Van Dam, Carlo Follo, Puthen V Jithesh, Shu-Dong Zhang, William G Richards, Raphael Bueno, Dean A Fennell, V Courtney Broaddus

3D/类器官

发表日期：2016

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PTEN dephosphorylates AKT to prevent the expression of GLUT1 on plasmamembrane and to limit glucose consumption in cancer cells

PTEN 使 AKT 去磷酸化，从而阻止 GLUT1 在质膜上的表达，并限制癌细胞中的葡萄糖消耗

期刊:Oncotarget

影响因子:

doi:10.18632/oncotarget.13113

Suratchanee Phadngam, Andrea Castiglioni, Alessandra Ferraresi, Federica Morani, Carlo Follo, Ciro Isidoro

Isolation, Characterization, and Autophagy Function of BECN1-Splicing Isoforms in Cancer Cells

BECN1 剪接异构体的分离、表征和癌细胞中的自噬功能

High Expression of the Lysosomal Protease Cathepsin D Confers Better Prognosis in Neuroblastoma Patients by Contrasting EGF-Induced Neuroblastoma Cell Growth

溶酶体蛋白酶组织蛋白酶 D 的高表达与 EGF 诱导的神经母细胞瘤细胞生长相比，可为神经母细胞瘤患者提供更好的预后

Amino acid response by Halofuginone in Cancer cells triggers autophagy through proteasome degradation of mTOR

癌细胞中氟吉诺酮的氨基酸反应通过蛋白酶体降解 mTOR 触发自噬

Autophagy facilitates the release of immunogenic signals following chemotherapy in 3D models of mesothelioma

自噬促进间皮瘤 3D 模型中化疗后免疫原性信号的释放

Autophagy in 3D In Vitro and Ex Vivo Cancer Models

3D 体外和离体癌症模型中的自噬

Inhibition of autophagy initiation potentiates chemosensitivity in mesothelioma

抑制自噬启动可增强间皮瘤的化学敏感性

The protein restriction mimetic Resveratrol is an autophagy inducer stronger than amino acid starvation in ovarian cancer cells

蛋白质限制模拟物白藜芦醇是卵巢癌细胞中比氨基酸饥饿更强的自噬诱导剂

Autophagy initiation correlates with the autophagic flux in 3D models of mesothelioma and with patient outcome

自噬启动与间皮瘤 3D 模型中的自噬通量以及患者预后相关

Analysis of Gene Expression in 3D Spheroids Highlights a Survival Role for ASS1 in Mesothelioma

3D 球体基因表达分析突出了 ASS1 在间皮瘤中的生存作用

PTEN dephosphorylates AKT to prevent the expression of GLUT1 on plasmamembrane and to limit glucose consumption in cancer cells

PTEN 使 AKT 去磷酸化，从而阻止 GLUT1 在质膜上的表达，并限制癌细胞中的葡萄糖消耗