日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Overexpression of the STOX1B isoform of STOX1 triggers preeclampsia-like symptoms through HNF4α-dependent alterations of coagulation cascades in mice

STOX1B亚型的过度表达可通过HNF4α依赖性的凝血级联反应改变,在小鼠体内引发类似先兆子痫的症状。

Collinot, Hélène; Strubé, Louise; Benouda, Ikram; Castille, Johan; Floriot, Sandrine; Daniel-Carlier, Nathalie; Passet, Bruno; Lager, Franck; Lagoutte, Isabelle; Renault, Gilles; Saintpierre, Benjamin; Favier, Maryline; Djeridane, Djihane; Méhats, Céline; Miralles, Francisco; Vilotte, Jean-Luc; Vaiman, Daniel

Congenital microcoria deletion in mouse links Sox21 dysregulation to disease and suggests a role for TGFB2 in glaucoma and myopia.

小鼠先天性小角膜缺失将 Sox21 失调与疾病联系起来,并表明 TGFB2 在青光眼和近视中发挥作用

Erjavec Elisa, Angée Clémentine, Hadjadj Djihad, Passet Bruno, David Pierre, Kostic Corinne, Dodé Emmanuel, Zanlonghi Xavier, Cagnard Nicolas, Nedelec Brigitte, Crippa Sylvain V, Bole-Feysot Christine, Zarhrate Mohammed, Creuzet Sophie, Castille Johan, Vilotte Jean-Luc, Calvas Patrick, Plaisancié Julie, Chassaing Nicolas, Kaplan Josseline, Rozet Jean-Michel, Fares Taie L

Wnt, glucocorticoid and cellular prion protein cooperate to drive a mesenchymal phenotype with poor prognosis in colon cancer

Wnt信号通路、糖皮质激素和细胞朊蛋白协同作用,驱动结肠癌细胞向间质表型转化,导致预后不良。

Mouillet-Richard, Sophie; Gougelet, Angélique; Passet, Bruno; Brochard, Camille; Le Corre, Delphine; Pitasi, Caterina Luana; Joubel, Camille; Sroussi, Marine; Gallois, Claire; Lavergne, Julien; Castille, Johan; Vilotte, Marthe; Daniel-Carlier, Nathalie; Pilati, Camilla; de Reyniès, Aurélien; Djouadi, Fatima; Colnot, Sabine; André, Thierry; Taieb, Julien; Vilotte, Jean-Luc; Romagnolo, Béatrice; Laurent-Puig, Pierre

Variation in the prion protein gene (PRNP) open reading frame sequence in French cervids

法国鹿科动物朊病毒蛋白基因(PRNP)开放阅读框序列的变异

Laubier, Johann; Van De Wiele, Anne; Barboiron, Aurélie; Laloë, Denis; Saint-Andrieux, Christine; Castille, Johan; Meloni, Emma; Ernst, Sonja; Pellerin, Maryline; Floriot, Sandrine; Daniel-Carlier, Nathalie; Passet, Bruno; Merlet, Joël; Verheyden, Hélène; Béringue, Vincent; Andréoletti, Olivier; Houston, Fiona; Vilotte, Jean-Luc; Bourret, Vincent; Moazami-Goudarzi, Katayoun

Potential genetic robustness of Prnp and Sprn double knockout mouse embryos towards ShRNA-lentiviral inoculation

Prnp和Sprn双敲除小鼠胚胎对shRNA慢病毒接种的潜在遗传稳健性

Rau, Andrea; Passet, Bruno; Castille, Johan; Daniel-Carlier, Nathalie; Asset, Alexandre; Lecardonnel, Jérome; Moroldo, Marco; Jaffrézic, Florence; Laloë, Denis; Moazami-Goudarzi, Katayoun; Vilotte, Jean-Luc

Cellular prion protein dysfunction in a prototypical inherited metabolic myopathy

典型遗传性代谢性肌病中的细胞朊蛋白功能障碍

Boufroura, Fatima-Zohra; Tomkiewicz-Raulet, Céline; Poindessous, Virginie; Castille, Johan; Vilotte, Jean-Luc; Bastin, Jean; Mouillet-Richard, Sophie; Djouadi, Fatima

CEP250 is Required for Maintaining Centrosome Cohesion in the Germline and Fertility in Male Mice

CEP250是维持雄性小鼠生殖细胞系中心体黏附和生育能力所必需的

Floriot, Sandrine; Bellutti, Laura; Castille, Johan; Moison, Pauline; Messiaen, Sébastien; Passet, Bruno; Boulanger, Laurent; Boukadiri, Abdelhak; Tourpin, Sophie; Beauvallet, Christian; Vilotte, Marthe; Riviere, Julie; Péchoux, Christine; Bertaud, Maud; Vilotte, Jean-Luc; Livera, Gabriel

Host prion protein expression levels impact prion tropism for the spleen

宿主朊病毒蛋白表达水平影响朊病毒对脾脏的嗜性

Béringue, Vincent; Tixador, Philippe; Andréoletti, Olivier; Reine, Fabienne; Castille, Johan; Laï, Thanh-Lan; Le Dur, Annick; Laisné, Aude; Herzog, Laetitia; Passet, Bruno; Rezaei, Human; Vilotte, Jean-Luc; Laude, Hubert

Improving the Predictive Value of Prion Inactivation Validation Methods to Minimize the Risks of Iatrogenic Transmission With Medical Instruments

提高朊病毒灭活验证方法的预测价值,以最大限度地降低医疗器械医源性传播的风险

Moudjou, Mohammed; Castille, Johan; Passet, Bruno; Herzog, Laetitia; Reine, Fabienne; Vilotte, Jean-Luc; Rezaei, Human; Béringue, Vincent; Igel-Egalon, Angélique

Mutated but Not Deleted Ovine PrP(C) N-Terminal Polybasic Region Strongly Interferes with Prion Propagation in Transgenic Mice

绵羊PrP(C) N端多碱性区域发生突变但未缺失,强烈干扰转基因小鼠体内朊病毒的传播。

Khalifé, Manal; Reine, Fabienne; Paquet-Fifield, Sophie; Castille, Johan; Herzog, Laetitia; Vilotte, Marthe; Moudjou, Mohammed; Moazami-Goudarzi, Katayoun; Makhzami, Samira; Passet, Bruno; Andréoletti, Olivier; Vilette, Didier; Laude, Hubert; Béringue, Vincent; Vilotte, Jean-Luc