日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Evidence for Non-Cancer-Specific T Cell Exhaustion in the Tcl1 Mouse Model for Chronic Lymphocytic Leukemia

Tcl1小鼠慢性淋巴细胞白血病模型中非癌症特异性T细胞耗竭的证据

Thomas Parigger ,Franz Josef Gassner ,Christian Scherhäufl ,Aryunni Abu Bakar ,Jan Philip Höpner ,Alexandra Hödlmoser ,Markus Steiner ,Kemal Catakovic ,Roland Geisberger ,Richard Greil ,Nadja Zaborsky

Imprecision and DNA Break Repair Biased towards Incompatible End Joining in Leukemia

白血病中不精确和 DNA 断裂修复偏向不相容的末端连接

Franz Josef Gassner #, Maria Schubert #, Stefan Rebhandl #, Karina Spandl, Nadja Zaborsky, Kemal Catakovic, Stephanie Blaimer, Daniel Hebenstreit, Richard Greil, Roland Geisberger

Fludarabine and rituximab with escalating doses of lenalidomide followed by lenalidomide/rituximab maintenance in previously untreated chronic lymphocytic leukaemia (CLL): the REVLIRIT CLL-5 AGMT phase I/II study

在既往未接受治疗的慢性淋巴细胞白血病 (CLL) 患者中,采用氟达拉滨和利妥昔单抗联合递增剂量的来那度胺治疗,随后进行来那度胺/利妥昔单抗维持治疗:REVLIRIT CLL-5 AGMT I/II 期研究

Egle, Alexander; Steurer, Michael; Melchardt, Thomas; Weiss, Lukas; Gassner, Franz Josef; Zaborsky, Nadja; Geisberger, Roland; Catakovic, Kemal; Hartmann, Tanja Nicole; Pleyer, Lisa; Voskova, Daniela; Thaler, Josef; Lang, Alois; Girschikofsky, Michael; Petzer, Andreas; Greil, Richard

Correction to: Fludarabine and rituximab with escalating doses of lenalidomide followed by lenalidomide/rituximab maintenance in previously untreated chronic lymphocytic leukaemia (CLL): the REVLIRIT CLL-5 AGMT phase I/II study

更正:氟达拉滨和利妥昔单抗联合递增剂量来那度胺治疗,随后进行来那度胺/利妥昔单抗维持治疗,用于治疗既往未接受治疗的慢性淋巴细胞白血病 (CLL):REVLIRIT CLL-5 AGMT I/II 期研究

Egle, Alexander; Steurer, Michael; Melchardt, Thomas; Weiss, Lukas; Gassner, Franz Josef; Zaborsky, Nadja; Geisberger, Roland; Catakovic, Kemal; Hartmann, Tanja Nicole; Pleyer, Lisa; Voskova, Daniela; Thaler, Josef; Lang, Alois; Girschikofsky, Michael; Petzer, Andreas; Greil, Richard

T cell exhaustion: from pathophysiological basics to tumor immunotherapy

细胞耗竭:从病理生理基础到肿瘤免疫治疗

Kemal Catakovic, Eckhard Klieser, Daniel Neureiter, Roland Geisberger

TIGIT expressing CD4+T cells represent a tumor-supportive T cell subset in chronic lymphocytic leukemia

表达 TIGIT 的 CD4+T 细胞是慢性淋巴细胞白血病中的肿瘤支持性 T 细胞亚群

Kemal Catakovic, Franz Josef Gassner, Christoph Ratswohl, Nadja Zaborsky, Stefan Rebhandl, Maria Schubert, Markus Steiner, Julia Christine Gutjahr, Lisa Pleyer, Alexander Egle, Tanja Nicole Hartmann, Richard Greil, Roland Geisberger

Chronic lymphocytic leukaemia induces an exhausted T cell phenotype in the TCL1 transgenic mouse model

慢性淋巴细胞白血病在TCL1转基因小鼠模型中诱导T细胞耗竭表型

Gassner, Franz J; Zaborsky, Nadja; Catakovic, Kemal; Rebhandl, Stefan; Huemer, Michael; Egle, Alexander; Hartmann, Tanja N; Greil, Richard; Geisberger, Roland

Chemotherapy-induced augmentation of T cells expressing inhibitory receptors is reversed by treatment with lenalidomide in chronic lymphocytic leukemia

在慢性淋巴细胞白血病中,化疗诱导的表达抑制性受体的T细胞增多可通过来那度胺治疗逆转。

Gassner, Franz J; Zaborsky, Nadja; Neureiter, Daniel; Huemer, Michael; Melchardt, Thomas; Egle, Alexander; Rebhandl, Stefan; Catakovic, Kemal; Hartmann, Tanja N; Greil, Richard; Geisberger, Roland

Alternative splice variants of AID are not stoichiometrically present at the protein level in chronic lymphocytic leukemia

在慢性淋巴细胞白血病中,AID 的替代剪接变体在蛋白质水平上不按化学计量存在

Stefan Rebhandl, Michael Huemer, Nadja Zaborsky, Franz Josef Gassner, Kemal Catakovic, Thomas Klaus Felder, Richard Greil, Roland Geisberger