Cevey相关文献与解析，生知库 | 链接生命科学的每一步

Díaz, Ariana; D'Attilio, Luciano; Penas, Federico; Bongiovanni, Bettina; Massa, Estefanía; Cevey, Agata; Santucci, Natalia; Bottasso, Oscar; Goren, Nora; Bay, María Luisa

发表日期：2023

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Fenofibrate Increases the Population of Non-Classical Monocytes in Asymptomatic Chagas Disease Patients and Modulates Inflammatory Cytokines in PBMC

非诺贝特可增加无症状恰加斯病患者的非经典单核细胞数量，并调节 PBMC 中的炎症细胞因子

期刊:Frontiers in Cellular and Infection Microbiology

影响因子:4.6

doi:10.3389/fcimb.2021.785166

Azul V Pieralisi, Ágata C Cevey, Federico N Penas, Nilda Prado, Ana Mori, Mónica Gili, Gerardo A Mirkin, Juan Gagliardi, Nora B Goren

Benznidazole Anti-Inflammatory Effects in Murine Cardiomyocytes and Macrophages Are Mediated by Class I PI3Kδ

苯并硝唑在小鼠心肌细胞和巨噬细胞中的抗炎作用由 I 类 PI3Kδ 介导

期刊:Frontiers in Immunology

影响因子:5.7

doi:10.3389/fimmu.2021.782891

Ágata C Cevey, Paula D Mascolo, Federico N Penas, Azul V Pieralisi, Aldana S Sequeyra, Gerardo A Mirkin, Nora B Goren

Pyridinecarboxylic Acid Derivative Stimulates Pro-Angiogenic Mediators by PI3K/AKT/mTOR and Inhibits Reactive Nitrogen and Oxygen Species and NF-κB Activation Through a PPARγ-Dependent Pathway in T. cruzi-Infected Macrophages

吡啶羧酸衍生物通过 PI3K/AKT/mTOR 刺激促血管生成介质，并通过 PPARγ 依赖性途径抑制克氏锥虫感染巨噬细胞中的活性氮和氧物质以及 NF-κB 活化

期刊:Frontiers in Immunology

影响因子:5.7

doi:10.3389/fimmu.2019.02955

Federico Nicolás Penas, Davide Carta, Ágata Carolina Cevey, María Jimena Rada, Azul Victoria Pieralisi, María Grazia Ferlin, María Elena Sales, Gerardo A Mirkin, Nora Beatriz Goren

IL-10/STAT3/SOCS3 Axis Is Involved in the Anti-inflammatory Effect of Benznidazole

IL-10/STAT3/SOCS3轴参与苯并硝唑的抗炎作用

期刊:Frontiers in Immunology

影响因子:5.7

doi:10.3389/fimmu.2019.01267

Ágata C Cevey, Federico N Penas, Catalina D Alba Soto, Gerardo A Mirkin, Nora B Goren

Budget Impact Analysis Of Subcutaneous Belimumab In Patients With Systemic Lupus Erythematosus In Spain

西班牙系统性红斑狼疮患者皮下注射贝利木单抗的预算影响分析

期刊:

影响因子:

doi:10.2147/CEOR.S218464

Cevey, Maria; Calvo-Alén, Jaime; Crespo, Carlos; Robles-Marhuenda, Angel; Smolen, Lee; Cortés-Hernández, Josefina

Treatment with a New Peroxisome Proliferator-Activated Receptor Gamma Agonist, Pyridinecarboxylic Acid Derivative, Increases Angiogenesis and Reduces Inflammatory Mediators in the Heart of Trypanosoma cruzi-Infected Mice

使用新型过氧化物酶体增殖激活受体γ激动剂吡啶羧酸衍生物进行治疗，可增加克氏锥虫感染小鼠心脏的血管生成并减少炎症介质

期刊:Frontiers in Immunology

影响因子:5.7

doi:10.3389/fimmu.2017.01738

Federico Nicolás Penas, Davide Carta, Ganna Dmytrenko, Gerado A Mirkin, Carlos Pablo Modenutti, Ágata Carolina Cevey, Maria Jimena Rada, Maria Grazia Ferlin, María Elena Sales, Nora Beatriz Goren

信号转导

发表日期：2017

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Treatment with Fenofibrate plus a low dose of Benznidazole attenuates cardiac dysfunction in experimental Chagas disease

非诺贝特联合小剂量苯硝唑治疗可减轻实验性恰加斯病的心脏功能障碍。

期刊:International Journal for Parasitology-Drugs and Drug Resistance

影响因子:3.4

doi:10.1016/j.ijpddr.2017.10.003

Cevey, Ágata C; Mirkin, Gerardo A; Donato, Martín; Rada, María J; Penas, Federico N; Gelpi, Ricardo J; Goren, Nora B

发表日期：2017

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Etiology of community-acquired pneumonia in hospitalized children based on WHO clinical guidelines

根据世界卫生组织临床指南，分析住院儿童社区获得性肺炎的病因

期刊:European Journal of Pediatrics

影响因子:2.6

doi:10.1007/s00431-009-0943-y

Cevey-Macherel, Manon; Galetto-Lacour, Annick; Gervaix, Alain; Siegrist, Claire-Anne; Bille, Jacques; Bescher-Ninet, Béatrice; Kaiser, Laurent; Krahenbuhl, Jean-Daniel; Gehri, Mario

肺炎

发表日期：2009

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Studies on the contribution of PPAR Gamma to tuberculosis physiopathology

PPARγ在结核病病理生理学中的作用研究

Fenofibrate Increases the Population of Non-Classical Monocytes in Asymptomatic Chagas Disease Patients and Modulates Inflammatory Cytokines in PBMC

非诺贝特可增加无症状恰加斯病患者的非经典单核细胞数量，并调节 PBMC 中的炎症细胞因子

Benznidazole Anti-Inflammatory Effects in Murine Cardiomyocytes and Macrophages Are Mediated by Class I PI3Kδ

苯并硝唑在小鼠心肌细胞和巨噬细胞中的抗炎作用由 I 类 PI3Kδ 介导

Pyridinecarboxylic Acid Derivative Stimulates Pro-Angiogenic Mediators by PI3K/AKT/mTOR and Inhibits Reactive Nitrogen and Oxygen Species and NF-κB Activation Through a PPARγ-Dependent Pathway in T. cruzi-Infected Macrophages

吡啶羧酸衍生物通过 PI3K/AKT/mTOR 刺激促血管生成介质，并通过 PPARγ 依赖性途径抑制克氏锥虫感染巨噬细胞中的活性氮和氧物质以及 NF-κB 活化

IL-10/STAT3/SOCS3 Axis Is Involved in the Anti-inflammatory Effect of Benznidazole

IL-10/STAT3/SOCS3轴参与苯并硝唑的抗炎作用

Budget Impact Analysis Of Subcutaneous Belimumab In Patients With Systemic Lupus Erythematosus In Spain

西班牙系统性红斑狼疮患者皮下注射贝利木单抗的预算影响分析

Treatment with a New Peroxisome Proliferator-Activated Receptor Gamma Agonist, Pyridinecarboxylic Acid Derivative, Increases Angiogenesis and Reduces Inflammatory Mediators in the Heart of Trypanosoma cruzi-Infected Mice

使用新型过氧化物酶体增殖激活受体γ激动剂吡啶羧酸衍生物进行治疗，可增加克氏锥虫感染小鼠心脏的血管生成并减少炎症介质

Treatment with Fenofibrate plus a low dose of Benznidazole attenuates cardiac dysfunction in experimental Chagas disease

非诺贝特联合小剂量苯硝唑治疗可减轻实验性恰加斯病的心脏功能障碍。

Etiology of community-acquired pneumonia in hospitalized children based on WHO clinical guidelines

根据世界卫生组织临床指南，分析住院儿童社区获得性肺炎的病因