日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Polθ activity modulates sensitivity to standard therapies in DNMT3A-deficient leukemia.

Polθ 活性调节 DNMT3A 缺陷型白血病对标准疗法的敏感性。

Le Bac Viet, Vekariya Umeshkumar, Toma Monika M, Nieborowska-Skorska Margaret, Caron Marie-Christine, Gozdecka Malgorzata, Haydar Zayd, Walsh Martin, Ghosh Jayashri, Vaughan-Williams Elaine, Podszywalow-Bartnicka Paulina, Kukuyan Anna-Mariya, Ziolkowska Sylwia, Atkins Jessica, Hadzijusufovic Emir, Chandramouly Gurushankar, Nejati Reza, Piwocka Katarzyna, Pomerantz Richard, Vassiliou George S, Huntly Brian J P, Valent Peter, Wasik Mariusz, Bellacosa Alfonso, Masson Jean-Yves, Gupta Gaorav P, Challen Grant A, Skorski Tomasz

Comparison with Ezh2 reveals the PRC2-dependent functions of Jarid2 in hematopoietic stem Cell lineage commitment.

与 Ezh2 的比较揭示了 Jarid2 在造血干细胞谱系定向中的 PRC2 依赖性功能。

Bjeije Hassan, Han Wentao, Lin Shuyang, Issa Nancy, Krishnan Aishwarya, Raj Infencia Xavier, Arand Jason, Li Yanan, Yang Wei, Magee Jeffrey A, Challen Grant A

Stathmin 1 regulates mitophagy and cellular function in hematopoietic stem cells

Stathmin 1 调节造血干细胞中的线粒体自噬和细胞功能

Chiquetto, Luana; Schuetz, Meg; Dong, Qian; Warmka, Mia; Valin, Liana; Jones, Ashley; Hunt, Patrick; Petermeier, Copper; Wang, Jie; Roundy, Nate; Greenberg, Zev J; Yang, Wei; Zhang, Christine R; Challen, Grant A; Luke, Cliff J; Signer, Robert A J; Beatty, Wandy L; Sykes, Stephen; Li, Weikai; Kast, David J; Schuettpelz, Laura G

JAK2 (V617F) Myeloproliferative Neoplasms Support Parallel Evolution of Independent Leukemic Clones

JAK2 (V617F) 骨髓增生性肿瘤支持独立白血病克隆的平行进化

Parsons, Tyler M; Krishnan, Aishwarya; Raj, Infencia Xavier; Young, Andrew L; O'Leary, David R; Arand, Jason; Cox, Maggie; Oh, Stephen T; Challen, Grant A

Parallel Evolution of Leukemic Clones in Myeloproliferative Neoplasms.

骨髓增生性肿瘤中白血病克隆的平行演化。

Parsons Tyler M, Krishnan Aishwarya, Raj Infencia Xavier, Young Andrew L, O'Leary David R, Arand Jason, Cox Maggie, Oh Stephen T, Challen Grant A

JARID2 Inhibition Reprograms Human Hematopoietic Progenitor Cells To Enhance Bone Marrow Transplantation.

JARID2抑制剂可重编程人类造血祖细胞以增强骨髓移植

Han Wentao, Bjeije Hassan, Celik Hamza, Rettig Michael, Issa Nancy, Young Andrew L, Li Yanan, Xavier Raj Infencia, Zhang Christine R, Krishnan Aishwarya, Parsons Tyler M, Burkart Samantha C, Arand Jason, Yang Wei, Magee Jeffrey A, Challen Grant A

Functional Comparison to Ezh2 Reveals PRC2-Independent Functions of Jarid2 in Hematopoietic Stem Cell Lineage Commitment.

与 Ezh2 的功能比较揭示了 Jarid2 在造血干细胞谱系定向中的 PRC2 非依赖性功能

Bjeije Hassan, Han Wentao, Issa Nancy, Krishnan Aishwarya, Raj Infencia Xavier, Arand Jason, Li Yanan, Yang Wei, Magee Jeffrey A, Challen Grant A

Engineering a humanized animal model of polycythemia vera with minimal JAK2V617F mutant allelic burden

构建JAK2V617F突变等位基因负荷最小的人源化真性红细胞增多症动物模型

Parsons, Tyler M; Krishnan, Aishwarya; Dunuwille, Wangisa M B; Young, Andrew L; Arand, Jason; Han, Wentao; Challen, Grant A

BIRC5 upregulation enhances DNMT3A-mutant T-ALL cell survival and pathogenesis

BIRC5 上调增强 DNMT3A 突变型 T 细胞急性淋巴细胞白血病 (T-ALL) 细胞的存活和致病性

Dunuwille, Wangisa; Wilson, William C; Bjeije, Hassan; Issa, Nancy; Han, Wentao; Parsons, Tyler M; Young, Andrew L; Xavier Raj, Infencia; Krishnan, Aishwarya; Gaur, Tarang; Wang, Eunice S; Weng, Andrew P; Stubbs, Matthew C; Celik, Hamza; Cashen, Amanda F; Edwards, John R; Challen, Grant A

DNA polymerase theta-mediated DNA repair is a functional dependency and therapeutic vulnerability in DNMT3A deficient leukemia cells

DNA聚合酶θ介导的DNA修复是DNMT3A缺陷型白血病细胞的功能依赖性和治疗脆弱性所在。

Le, Bac Viet; Vekariya, Umeshkumar; Toma, Monika M; Nieborowska-Skorska, Margaret; Caron, Marie-Christine; Gozdecka, Malgorzata; Haydar, Zayd; Walsh, Martin; Ghosh, Jayashri; Vaughan-Williams, Elaine; Podszywalow-Bartnicka, Paulina; Kukuyan, Anna-Mariya; Ziolkowska, Sylwia; Hadzijusufovic, Emir; Chandramouly, Gurushankar; Piwocka, Katarzyna; Pomerantz, Richard; Vassiliou, George S; Huntly, Brian Jp; Valent, Peter; Bellacosa, Alfonso; Masson, Jean-Yves; Gupta, Gaorav P; Challen, Grant A; Skorski, Tomasz