日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Akt kinase C-terminal modifications control activation loop dephosphorylation and enhance insulin response

Akt激酶C端修饰控制激活环去磷酸化并增强胰岛素反应

Chan, Tung O; Zhang, Jin; Tiegs, Brian C; Blumhof, Brian; Yan, Linda; Keny, Nikhil; Penny, Morgan; Li, Xue; Pascal, John M; Armen, Roger S; Rodeck, Ulrich; Penn, Raymond B

miR-146a targets Fos expression in human cardiac cells

miR-146a靶向人类心脏细胞中的Fos表达

Palomer, Xavier; Capdevila-Busquets, Eva; Botteri, Gaia; Davidson, Mercy M; Rodríguez, Cristina; Martínez-González, José; Vidal, Francisco; Barroso, Emma; Chan, Tung O; Feldman, Arthur M; Vázquez-Carrera, Manuel

Induced overexpression of Na(+)/Ca(2+) exchanger does not aggravate myocardial dysfunction induced by transverse aortic constriction.

诱导 Na(+)/Ca(2+) 交换器过度表达不会加重横向主动脉缩窄引起的心肌功能障碍

Wang Jufang, Gao Erhe, Chan Tung O, Zhang Xue-Qian, Song Jianliang, Shang Xiying, Koch Walter J, Feldman Arthur M, Cheung Joseph Y

A truncated fragment of Src protein kinase generated by calpain-mediated cleavage is a mediator of neuronal death in excitotoxicity

由钙蛋白酶介导的切割产生的Src蛋白激酶截短片段是兴奋性毒性中神经元死亡的介质。

Hossain, M Iqbal; Roulston, Carli L; Kamaruddin, M Aizuddin; Chu, Percy W Y; Ng, Dominic C H; Dusting, Gregory J; Bjorge, Jeffrey D; Williamson, Nicholas A; Fujita, Donald J; Cheung, Steve N; Chan, Tung O; Hill, Andrew F; Cheng, Heung-Chin

Residues 248-252 and 300-304 of the cardiac Na+/Ca2+ exchanger are involved in its regulation by phospholemman

心脏Na+/Ca2+交换器的248-252和300-304位氨基酸残基参与磷脂酰肌醇对其的调节。

Zhang, Xue-Qian; Wang, JuFang; Song, Jianliang; Ji, Angi M; Chan, Tung O; Cheung, Joseph Y

Phospholemman and beta-adrenergic stimulation in the heart

心脏中的磷脂和β-肾上腺素能刺激

Wang, JuFang; Gao, Erhe; Song, Jianliang; Zhang, Xue-Qian; Li, Jifen; Koch, Walter J; Tucker, Amy L; Philipson, Kenneth D; Chan, Tung O; Feldman, Arthur M; Cheung, Joseph Y

Induced overexpression of Na+/Ca2+ exchanger transgene: altered myocyte contractility, [Ca2+]i transients, SR Ca2+ contents, and action potential duration.

诱导 Na+/Ca2+ 交换转基因过表达:改变心肌细胞收缩力、[Ca2+]i 瞬变、SR Ca2+ 含量和动作电位持续时间

Wang JuFang, Chan Tung O, Zhang Xue-Qian, Gao Erhe, Song Jianliang, Koch Walter J, Feldman Arthur M, Cheung Joseph Y

Cardiac-restricted overexpression of the A(2A)-adenosine receptor in FVB mice transiently increases contractile performance and rescues the heart failure phenotype in mice overexpressing the A(1)-adenosine receptor.

在 FVB 小鼠中,心脏特异性过表达 A(2A)-腺苷受体可暂时提高收缩性能,并挽救过表达 A(1)-腺苷受体的小鼠的心力衰竭表型

Chan Tung O, Funakoshi Hajime, Song Jianliang, Zhang Xue-Qian, Wang JuFang, Chung Paul H, DeGeorge Brent R Jr, Li Xue, Zhang Jin, Herrmann David E, Diamond Maura, Hamad Eman, Houser Steven R, Koch Walter J, Cheung Joseph Y, Feldman Arthur M