Exogenous H2S contributes to recovery of ischemic post-conditioning-induced cardioprotection by decrease of ROS level via down-regulation of NF-κB and JAK2-STAT3 pathways in the aging cardiomyocytes
外源性H2S通过下调衰老心肌细胞中的NF-κB和JAK2-STAT3通路降低ROS水平,从而促进缺血后适应诱导的心脏保护作用的恢复。
期刊:Cell and Bioscience
影响因子:6.2
doi:10.1186/s13578-016-0090-x
Li, Lina; Li, Meixiu; Li, Youyou; Sun, Weiming; Wang, Yuehong; Bai, Shuzhi; Li, Hongxia; Wu, Bo; Yang, Guangdong; Wang, Rui; Wu, Lingyun; Li, Hongzhu; Xu, Changing
JAK/STAT
信号转导
NF-κB
JAK2
STAT3
衰老
细胞生物学