Chao Julie相关文献与解析，生知库 | 链接生命科学的每一步

Reduced Plasma Kallistatin Is Associated With the Severity of Coronary Artery Disease, and Kallistatin Treatment Attenuates Atherosclerotic Plaque Formation in Mice

血浆中卡利斯他汀水平降低与冠状动脉疾病的严重程度相关，而卡利斯他汀治疗可减轻小鼠动脉粥样硬化斑块的形成。

期刊:Journal of the American Heart Association

影响因子:5.3

doi:10.1161/JAHA.118.009562

Yao, Yuyu; Li, Bing; Liu, Chang; Fu, Cong; Li, Pengfei; Guo, Youming; Ma, Genshan; Liu, Naifeng; Chao, Lee; Chao, Julie

Kallistatin attenuates endothelial senescence by modulating Let-7g-mediated miR-34a-SIRT1-eNOS pathway

卡利斯他汀通过调节Let-7g介导的miR-34a-SIRT1-eNOS通路来减弱内皮细胞衰老。

期刊:Journal of Cellular and Molecular Medicine

影响因子:4.2

doi:10.1111/jcmm.13734

Guo, Youming; Chao, Lee; Chao, Julie

Protective Role of Endogenous Kallistatin in Vascular Injury and Senescence by Inhibiting Oxidative Stress and Inflammation

内源性激肽释放酶抑制剂通过抑制氧化应激和炎症在血管损伤和衰老中发挥保护作用

期刊:Oxidative Medicine and Cellular Longevity

影响因子:

doi:10.1155/2018/4138560

Chao, Julie; Guo, Youming; Chao, Lee

炎症

氧化应激

炎症/感染

Immunology/Inflammation

Kallistatin reduces vascular senescence and aging by regulating microRNA-34a-SIRT1 pathway

卡利斯他汀通过调节microRNA-34a-SIRT1通路来减少血管衰老和老化。

期刊:Aging Cell

影响因子:7.1

doi:10.1111/acel.12615

Guo, Youming; Li, Pengfei; Gao, Lin; Zhang, Jingmei; Yang, Zhirong; Bledsoe, Grant; Chang, Eugene; Chao, Lee; Chao, Julie

Plasma kallistatin in critically ill patients with severe sepsis and septic shock.

重症脓毒症和脓毒性休克危重患者的血浆激肽释放酶抑制剂水平

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0178387

Lin Wei-Chieh, Chen Chang-Wen, Chao Lee, Chao Julie, Lin Yee-Shin

其它

发表日期：2017

文献求助收藏

Role of Kallistatin Treatment in Aging and Cancer by Modulating miR-34a and miR-21 Expression

卡利斯他汀治疗通过调节 miR-34a 和 miR-21 的表达在衰老和癌症中发挥作用

期刊:Oxidative Medicine and Cellular Longevity

影响因子:

doi:10.1155/2017/5025610

Chao, Julie; Guo, Youming; Li, Pengfei; Chao, Lee

Opposing Effects of Oxygen Regulation on Kallistatin Expression: Kallistatin as a Novel Mediator of Oxygen-Induced HIF-1-eNOS-NO Pathway

氧调节对卡利司他汀表达的相反作用：卡利司他汀作为氧诱导的HIF-1-eNOS-NO通路的新型介质

期刊:Oxidative Medicine and Cellular Longevity

影响因子:

doi:10.1155/2017/5262958

Chao, Julie; Guo, Youming; Li, Pengfei; Chao, Lee

Kallistatin induces breast cancer cell apoptosis and autophagy by modulating Wnt signaling and microRNA synthesis

卡利斯他汀通过调节Wnt信号通路和microRNA合成诱导乳腺癌细胞凋亡和自噬。

期刊:Experimental Cell Research

影响因子:3.5

doi:10.1016/j.yexcr.2016.01.004

Li, Pengfei; Guo, Youming; Bledsoe, Grant; Yang, Zhirong; Chao, Lee; Chao, Julie

Kallistatin treatment attenuates lethality and organ injury in mouse models of established sepsis

卡利司他汀治疗可减轻已确诊脓毒症的小鼠模型的致死率和器官损伤

期刊:Critical Care

影响因子:8.8

doi:10.1186/s13054-015-0919-4

Pengfei Li, Youming Guo, Grant Bledsoe, Zhi-Rong Yang, Hongkuan Fan, Lee Chao, Julie Chao

Human tissue kallikrein in the treatment of acute ischemic stroke

人组织激肽释放酶在急性缺血性卒中治疗中的应用

Reduced Plasma Kallistatin Is Associated With the Severity of Coronary Artery Disease, and Kallistatin Treatment Attenuates Atherosclerotic Plaque Formation in Mice

血浆中卡利斯他汀水平降低与冠状动脉疾病的严重程度相关，而卡利斯他汀治疗可减轻小鼠动脉粥样硬化斑块的形成。

Kallistatin attenuates endothelial senescence by modulating Let-7g-mediated miR-34a-SIRT1-eNOS pathway

卡利斯他汀通过调节Let-7g介导的miR-34a-SIRT1-eNOS通路来减弱内皮细胞衰老。

Protective Role of Endogenous Kallistatin in Vascular Injury and Senescence by Inhibiting Oxidative Stress and Inflammation

内源性激肽释放酶抑制剂通过抑制氧化应激和炎症在血管损伤和衰老中发挥保护作用

Kallistatin reduces vascular senescence and aging by regulating microRNA-34a-SIRT1 pathway

卡利斯他汀通过调节microRNA-34a-SIRT1通路来减少血管衰老和老化。

Plasma kallistatin in critically ill patients with severe sepsis and septic shock.

重症脓毒症和脓毒性休克危重患者的血浆激肽释放酶抑制剂水平

Role of Kallistatin Treatment in Aging and Cancer by Modulating miR-34a and miR-21 Expression

卡利斯他汀治疗通过调节 miR-34a 和 miR-21 的表达在衰老和癌症中发挥作用

Opposing Effects of Oxygen Regulation on Kallistatin Expression: Kallistatin as a Novel Mediator of Oxygen-Induced HIF-1-eNOS-NO Pathway

氧调节对卡利司他汀表达的相反作用：卡利司他汀作为氧诱导的HIF-1-eNOS-NO通路的新型介质

Kallistatin induces breast cancer cell apoptosis and autophagy by modulating Wnt signaling and microRNA synthesis

卡利斯他汀通过调节Wnt信号通路和microRNA合成诱导乳腺癌细胞凋亡和自噬。

Kallistatin treatment attenuates lethality and organ injury in mouse models of established sepsis

卡利司他汀治疗可减轻已确诊脓毒症的小鼠模型的致死率和器官损伤