Chellaiah相关文献与解析，生知库 | 链接生命科学的每一步

L-Plastin deficiency produces increased trabecular bone due to attenuation of sealing ring formation and osteoclast dysfunction

L-Plastin缺乏会导致骨小梁增多，这是由于骨小梁封闭环形成减弱和破骨细胞功能障碍所致。

Chellaiah, Meenakshi A; Moorer, Megan C; Majumdar, Sunipa; Aljohani, Hanan; Morley, Sharon C; Yingling, Vanessa; Stains, Joseph P

细胞生物学

发表日期：2020

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Identification of sequence-specific interactions of the CD44-intracellular domain with RUNX2 in the transcription of matrix metalloprotease-9 in human prostate cancer cells

鉴定 CD44 胞内结构域与 RUNX2 在人类前列腺癌细胞基质金属蛋白酶 9 转录中的序列特异性相互作用

期刊:Cancer Drug Resistance

影响因子:4.6

doi:10.20517/cdr.2020.21

Linda T Senbanjo, Hanan AlJohani, Mohammed AlQranei, Sunipa Majumdar, Tao Ma, Meenakshi A Chellaiah

C-phycocyanin attenuates RANKL-induced osteoclastogenesis and bone resorption in vitro through inhibiting ROS levels, NFATc1 and NF-κB activation

C-藻蓝蛋白通过抑制 ROS 水平、NFATc1 和 NF-κB 活化，减弱 RANKL 诱导的破骨细胞生成和骨吸收

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-020-59363-y

Mohammed S AlQranei, Hanan Aljohani, Sunipa Majumdar, Linda T Senbanjo, Meenakshi A Chellaiah

Characterization of CD44 intracellular domain interaction with RUNX2 in PC3 human prostate cancer cells

PC3 人前列腺癌细胞中 CD44 胞内结构域与 RUNX2 相互作用的表征

期刊:Cell Communication and Signaling

影响因子:8.2

doi:10.1186/s12964-019-0395-6

Linda T Senbanjo, Hanan AlJohani, Sunipa Majumdar, Meenakshi A Chellaiah

Androgen receptor expression reduces stemness characteristics of prostate cancer cells (PC3) by repression of CD44 and SOX2

雄激素受体表达通过抑制 CD44 和 SOX2 降低前列腺癌细胞 (PC3) 的干细胞特征

期刊:Journal of Cellular Biochemistry

影响因子:3

doi:10.1002/jcb.27573

Deepa Srinivasan, Linda Senbanjo, Sunipa Majumdar, Renty B Franklin, Meenakshi A Chellaiah

Methylsulfonylmethane increases osteogenesis and regulates the mineralization of the matrix by transglutaminase 2 in SHED cells

甲基磺酰甲烷通过转谷氨酰胺酶2促进SHED细胞的成骨作用并调节基质矿化。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0225598

Aljohani, Hanan; Senbanjo, Linda T; Chellaiah, Meenakshi A

细胞生物学

发表日期：2019

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Peptidomimetic inhibitor of L-plastin reduces osteoclastic bone resorption in aging female mice

L-plastin肽模拟抑制剂可降低衰老雌性小鼠的破骨细胞骨吸收

L-Plastin Phosphorylation: Possible Regulation by a TNFR1 Signaling Cascade in Osteoclasts

L-Plastin磷酸化：破骨细胞中TNFR1信号级联可能对其进行调控

Methylsulfonylmethane Increases the Alveolar Bone Density of Mandibles in Aging Female Mice

甲基磺酰甲烷可增加老年雌性小鼠下颌骨的牙槽骨密度

Lipopolysaccharide- TLR-4 Axis regulates Osteoclastogenesis independent of RANKL/RANK signaling

脂多糖-TLR-4 轴独立于 RANKL/RANK 信号传导调节破骨细胞生成

L-Plastin deficiency produces increased trabecular bone due to attenuation of sealing ring formation and osteoclast dysfunction

L-Plastin缺乏会导致骨小梁增多，这是由于骨小梁封闭环形成减弱和破骨细胞功能障碍所致。

Identification of sequence-specific interactions of the CD44-intracellular domain with RUNX2 in the transcription of matrix metalloprotease-9 in human prostate cancer cells

鉴定 CD44 胞内结构域与 RUNX2 在人类前列腺癌细胞基质金属蛋白酶 9 转录中的序列特异性相互作用

C-phycocyanin attenuates RANKL-induced osteoclastogenesis and bone resorption in vitro through inhibiting ROS levels, NFATc1 and NF-κB activation

C-藻蓝蛋白通过抑制 ROS 水平、NFATc1 和 NF-κB 活化，减弱 RANKL 诱导的破骨细胞生成和骨吸收

Characterization of CD44 intracellular domain interaction with RUNX2 in PC3 human prostate cancer cells

PC3 人前列腺癌细胞中 CD44 胞内结构域与 RUNX2 相互作用的表征

Androgen receptor expression reduces stemness characteristics of prostate cancer cells (PC3) by repression of CD44 and SOX2

雄激素受体表达通过抑制 CD44 和 SOX2 降低前列腺癌细胞 (PC3) 的干细胞特征

Methylsulfonylmethane increases osteogenesis and regulates the mineralization of the matrix by transglutaminase 2 in SHED cells

甲基磺酰甲烷通过转谷氨酰胺酶2促进SHED细胞的成骨作用并调节基质矿化。