Chen Bijun相关文献与解析，生知库 | 链接生命科学的每一步

Sex-specific associations between levels of high-sensitivity C-reactive protein and severity of depression: retrospective cross-sectional analysis of inpatients in China

中国住院患者高敏C反应蛋白水平与抑郁症严重程度的性别特异性关联：回顾性横断面分析

期刊:BMC Psychiatry

影响因子:3.6

doi:10.1186/s12888-024-06123-1

Chen, Bijun; Dong, Jiangwen; Guo, Wanjun; Li, Tao

Macrophages in the infarcted heart acquire a fibrogenic phenotype, expressing matricellular proteins, but do not undergo fibroblast conversion

梗死心脏中的巨噬细胞获得纤维化表型，表达细胞外基质蛋白，但不会转化为成纤维细胞。

期刊:Journal of Molecular and Cellular Cardiology

影响因子:

doi:10.1016/j.yjmcc.2024.07.010

Li, Ruoshui; Hanna, Anis; Huang, Shuaibo; Hernandez, Silvia C; Tuleta, Izabela; Kubota, Akihiko; Humeres, Claudio; Chen, Bijun; Liu, Yang; Zheng, Deyou; Frangogiannis, Nikolaos G

Renal NKCC2 Is Dual Regulated by the Synergy of 20-HETE and High-Fat Diet in CYP4F2 Transgenic Mice

在 CYP4F2 转基因小鼠中，肾脏 NKCC2 受 20-HETE 和高脂饮食的协同作用双重调节

期刊:Kidney & Blood Pressure Research

影响因子:2.3

doi:10.1159/000517382

Jingjing Wu, Guangrui Lai, Fangjie Chen, Bijun Zhang, Yanyan Zhao

信号转导

发表日期：2021

文献求助收藏

The Functional Heterogeneity of Resident Cardiac Macrophages in Myocardial Injury(CCR2(+) Cells Promote Inflammation, Whereas CCR2(-) Cells Protect)

心肌损伤中驻留心脏巨噬细胞的功能异质性（CCR2(+)细胞促进炎症，而CCR2(-)细胞发挥保护作用）

期刊:Circulation Research

影响因子:16.2

doi:10.1161/CIRCRESAHA.118.314357

Chen, Bijun; Brickshawana, Adipong; Frangogiannis, Nikolaos G

Immunology/Inflammation

Protective Effects of Activated Myofibroblasts in the Pressure-Overloaded Myocardium Are Mediated Through Smad-Dependent Activation of a Matrix-Preserving Program

活化肌成纤维细胞在压力超负荷心肌中的保护作用是通过Smad依赖性基质保护程序的激活介导的。

期刊:Circulation Research

影响因子:16.2

doi:10.1161/CIRCRESAHA.118.314438

Russo, Ilaria; Cavalera, Michele; Huang, Shuaibo; Su, Ya; Hanna, Anis; Chen, Bijun; Shinde, Arti V; Conway, Simon J; Graff, Jonathan; Frangogiannis, Nikolaos G

Macrophage Smad3 Protects the Infarcted Heart, Stimulating Phagocytosis and Regulating Inflammation

巨噬细胞Smad3通过刺激吞噬作用和调节炎症来保护梗死的心脏

期刊:Circulation Research

影响因子:16.2

doi:10.1161/CIRCRESAHA.119.315069

Chen, Bijun; Huang, Shuaibo; Su, Ya; Wu, Yi-Jin; Hanna, Anis; Brickshawana, Adipong; Graff, Jonathan; Frangogiannis, Nikolaos G

Immunology/Inflammation

Distinct roles of myofibroblast-specific Smad2 and Smad3 signaling in repair and remodeling of the infarcted heart.

肌成纤维细胞特异性 Smad2 和 Smad3 信号在梗死心脏的修复和重塑中发挥着不同的作用

期刊:Journal of Molecular and Cellular Cardiology

影响因子:4.7

doi:10.1016/j.yjmcc.2019.05.006

Huang Shuaibo, Chen Bijun, Su Ya, Alex Linda, Humeres Claudio, Shinde Arti V, Conway Simon J, Frangogiannis Nikolaos G

细胞生物学

发表日期：2019

文献求助收藏

Opposing Actions of Fibroblast and Cardiomyocyte Smad3 Signaling in the Infarcted Myocardium

梗死心肌中成纤维细胞和心肌细胞Smad3信号的相反作用

期刊:Circulation

影响因子:38.6

doi:10.1161/CIRCULATIONAHA.117.029622

Kong, Ping; Shinde, Arti V; Su, Ya; Russo, Ilaria; Chen, Bijun; Saxena, Amit; Conway, Simon J; Graff, Jonathan M; Frangogiannis, Nikolaos G

Macrophages in the Remodeling Failing Heart

重塑衰竭心脏中的巨噬细胞

期刊:Circulation Research

影响因子:16.2

doi:10.1161/CIRCRESAHA.116.309624

Chen, Bijun; Frangogiannis, Nikolaos G

Stressful Life Events and Neuroticism among Chinese Women with Recurrent Major Depressive Disorder

压力性生活事件与中国复发性重度抑郁症女性的神经质

Sex-specific associations between levels of high-sensitivity C-reactive protein and severity of depression: retrospective cross-sectional analysis of inpatients in China

中国住院患者高敏C反应蛋白水平与抑郁症严重程度的性别特异性关联：回顾性横断面分析

Macrophages in the infarcted heart acquire a fibrogenic phenotype, expressing matricellular proteins, but do not undergo fibroblast conversion

梗死心脏中的巨噬细胞获得纤维化表型，表达细胞外基质蛋白，但不会转化为成纤维细胞。

Renal NKCC2 Is Dual Regulated by the Synergy of 20-HETE and High-Fat Diet in CYP4F2 Transgenic Mice

在 CYP4F2 转基因小鼠中，肾脏 NKCC2 受 20-HETE 和高脂饮食的协同作用双重调节

The Functional Heterogeneity of Resident Cardiac Macrophages in Myocardial Injury(CCR2(+) Cells Promote Inflammation, Whereas CCR2(-) Cells Protect)

心肌损伤中驻留心脏巨噬细胞的功能异质性（CCR2(+)细胞促进炎症，而CCR2(-)细胞发挥保护作用）

Protective Effects of Activated Myofibroblasts in the Pressure-Overloaded Myocardium Are Mediated Through Smad-Dependent Activation of a Matrix-Preserving Program

活化肌成纤维细胞在压力超负荷心肌中的保护作用是通过Smad依赖性基质保护程序的激活介导的。

Macrophage Smad3 Protects the Infarcted Heart, Stimulating Phagocytosis and Regulating Inflammation

巨噬细胞Smad3通过刺激吞噬作用和调节炎症来保护梗死的心脏

Distinct roles of myofibroblast-specific Smad2 and Smad3 signaling in repair and remodeling of the infarcted heart.

肌成纤维细胞特异性 Smad2 和 Smad3 信号在梗死心脏的修复和重塑中发挥着不同的作用

Opposing Actions of Fibroblast and Cardiomyocyte Smad3 Signaling in the Infarcted Myocardium

梗死心肌中成纤维细胞和心肌细胞Smad3信号的相反作用

Macrophages in the Remodeling Failing Heart

重塑衰竭心脏中的巨噬细胞