日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Bax and Bak function as the outer membrane component of the mitochondrial permeability pore in regulating necrotic cell death in mice

Bax和Bak作为线粒体通透性孔的外膜成分,在小鼠中调控坏死性细胞死亡。

Karch, Jason; Kwong, Jennifer Q; Burr, Adam R; Sargent, Michelle A; Elrod, John W; Peixoto, Pablo M; Martinez-Caballero, Sonia; Osinska, Hanna; Cheng, Emily H-Y; Robbins, Jeffrey; Kinnally, Kathleen W; Molkentin, Jeffery D

A new pathway that regulates 53BP1 stability implicates cathepsin L and vitamin D in DNA repair.

一条调节 53BP1 稳定性的新途径表明,组织蛋白酶 L 和维生素 D 参与了 DNA 修复

Gonzalez-Suarez Ignacio, Redwood Abena B, Grotsky David A, Neumann Martin A, Cheng Emily H-Y, Stewart Colin L, Dusso Adriana, Gonzalo Susana

Taspase1 functions as a non-oncogene addiction protease that coordinates cancer cell proliferation and apoptosis

Taspase1 是一种非癌基因依赖性蛋白酶,它协调癌细胞的增殖和凋亡。

Chen, David Y; Liu, Han; Takeda, Shugaku; Tu, Ho-Chou; Sasagawa, Satoru; Van Tine, Brian A; Lu, Dongsi; Cheng, Emily H-Y; Hsieh, James J-D

Dual autonomous mitochondrial cell death pathways are activated by Nix/BNip3L and induce cardiomyopathy

Nix/BNip3L激活双重自主线粒体细胞死亡通路,诱发心肌病。

Chen, Yun; Lewis, William; Diwan, Abhinav; Cheng, Emily H-Y; Matkovich, Scot J; Dorn, Gerald W 2nd

The VDAC2-BAK rheostat controls thymocyte survival

VDAC2-BAK 调节器控制胸腺细胞存活

Ren, Decheng; Kim, Hyungjin; Tu, Ho-Chou; Westergard, Todd D; Fisher, Jill K; Rubens, Jeff A; Korsmeyer, Stanley J; Hsieh, James J-D; Cheng, Emily H-Y

MLL fusions: pathways to leukemia

MLL融合基因:导致白血病的途径

Liu, Han; Cheng, Emily H Y; Hsieh, James J D

BAX activation is initiated at a novel interaction site

BAX激活起始于一个新的相互作用位点。

Gavathiotis, Evripidis; Suzuki, Motoshi; Davis, Marguerite L; Pitter, Kenneth; Bird, Gregory H; Katz, Samuel G; Tu, Ho-Chou; Kim, Hyungjin; Cheng, Emily H-Y; Tjandra, Nico; Walensky, Loren D

Proapoptotic BAX and BAK control multiple initiator caspases

促凋亡蛋白BAX和BAK调控多种起始半胱天冬酶

Ruiz-Vela, Antonio; Opferman, Joseph T; Cheng, Emily H-Y; Korsmeyer, Stanley J