Chengming Fei相关文献与解析，生知库 | 链接生命科学的每一步

Cellular senescence induced by S100A9 in mesenchymal stromal cells through NLRP3 inflammasome activation

S100A9 通过 NLRP3 炎症小体激活诱导间充质基质细胞衰老

期刊:Aging-Us

影响因子:

doi:10.18632/aging.102409

Lei Shi, Youshan Zhao, Chengming Fei, Juan Guo, Yan Jia, Dong Wu, Lingyun Wu, Chunkang Chang

Dicer1 downregulation by multiple myeloma cells promotes the senescence and tumor-supporting capacity and decreases the differentiation potential of mesenchymal stem cells

多发性骨髓瘤细胞中 Dicer1 下调促进间充质干细胞衰老和肿瘤支持能力并降低其分化潜能

期刊:Cell Death & Disease

影响因子:8.1

doi:10.1038/s41419-018-0545-6

Juan Guo, Youshan Zhao, Chengming Fei, Sida Zhao, Qingqing Zheng, Jiying Su, Dong Wu, Xiao Li, Chunkang Chang

Iron overload promotes mitochondrial fragmentation in mesenchymal stromal cells from myelodysplastic syndrome patients through activation of the AMPK/MFF/Drp1 pathway

铁过载通过激活 AMPK/MFF/Drp1 通路促进骨髓增生异常综合征患者间充质基质细胞的线粒体碎裂

期刊:Cell Death & Disease

影响因子:8.1

doi:10.1038/s41419-018-0552-7

Qingqing Zheng, Youshan Zhao, Juan Guo, Sida Zhao, Chengming Fei, Chao Xiao, Dong Wu, Lingyun Wu, Xiao Li, Chunkang Chang

Downregulation of MMP1 in MDS-derived mesenchymal stromal cells reduces the capacity to restrict MDS cell proliferation

MDS 衍生的间充质基质细胞中 MMP1 的下调降低了限制 MDS 细胞增殖的能力

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/srep43849

Sida Zhao, Youshan Zhao, Juan Guo, Chengming Fei, Qingqing Zheng, Xiao Li, Chunkang Chang

Lenalidomide restores the osteogenic differentiation of bone marrow mesenchymal stem cells from multiple myeloma patients via deactivating Notch signaling pathway

来那度胺通过抑制 Notch 信号通路恢复多发性骨髓瘤患者骨髓间充质干细胞的成骨分化

期刊:Oncotarget

影响因子:

doi:10.18632/oncotarget.19265

Juan Guo, Chengming Fei, Youshan Zhao, Sida Zhao, Qingqing Zheng, Jiying Su, Dong Wu, Xiao Li, Chunkang Chang

High expression of APAF-1 elevates erythroid apoptosis in iron overload myelodysplastic syndrome

APAF-1 高表达可增加铁过载骨髓增生异常综合征中的红细胞凋亡

期刊:Tumor Biology

影响因子:

doi:10.1007/s13277-013-1294-x

Shucheng Gu, Youshan Zhao, Juan Guo, Feng Xu, Chengming Fei, Xi Zhang, Chao Xiao, Chunkang Chang, Xiao Li

ID1 overexpression promotes HCC progression by amplifying the AURKA/Myc signaling pathway

ID1 过表达通过扩增 AURKA/Myc 信号通路促进 HCC 进展

Cellular senescence induced by S100A9 in mesenchymal stromal cells through NLRP3 inflammasome activation

S100A9 通过 NLRP3 炎症小体激活诱导间充质基质细胞衰老

Dicer1 downregulation by multiple myeloma cells promotes the senescence and tumor-supporting capacity and decreases the differentiation potential of mesenchymal stem cells

多发性骨髓瘤细胞中 Dicer1 下调促进间充质干细胞衰老和肿瘤支持能力并降低其分化潜能

Iron overload promotes mitochondrial fragmentation in mesenchymal stromal cells from myelodysplastic syndrome patients through activation of the AMPK/MFF/Drp1 pathway

铁过载通过激活 AMPK/MFF/Drp1 通路促进骨髓增生异常综合征患者间充质基质细胞的线粒体碎裂

Downregulation of MMP1 in MDS-derived mesenchymal stromal cells reduces the capacity to restrict MDS cell proliferation

MDS 衍生的间充质基质细胞中 MMP1 的下调降低了限制 MDS 细胞增殖的能力

Lenalidomide restores the osteogenic differentiation of bone marrow mesenchymal stem cells from multiple myeloma patients via deactivating Notch signaling pathway

来那度胺通过抑制 Notch 信号通路恢复多发性骨髓瘤患者骨髓间充质干细胞的成骨分化

High expression of APAF-1 elevates erythroid apoptosis in iron overload myelodysplastic syndrome

APAF-1 高表达可增加铁过载骨髓增生异常综合征中的红细胞凋亡