日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Innate Immunity in Type 1 Diabetes

1型糖尿病的先天免疫

Bertrand, Léo; Chervonsky, Alexander V; Lehuen, Agnès

Microbial influences on severity and sex bias of systemic autoimmunity

微生物对系统性自身免疫疾病严重程度和性别差异的影响

Lee, Jean; Reiman, Derek; Singh, Samara; Chang, Anthony; Morel, Laurence; Chervonsky, Alexander V

Mouse Homologue of Human HLA-DO Does Not Preempt Autoimmunity but Controls Murine Gammaherpesvirus MHV68

人类HLA-DO的小鼠同源物不抑制自身免疫,但可控制小鼠γ疱疹病毒MHV68

Lee, Jean; Cullum, Emily; Stoltz, Kyle; Bachmann, Niklas; Strong, Zoe; Millick, Danielle D; Denzin, Lisa K; Chang, Anthony; Tarakanova, Vera; Chervonsky, Alexander V; Golovkina, Tatyana

The MHC-II peptidome of pancreatic islets identifies key features of autoimmune peptides.

胰岛的 MHC-II 肽组可识别自身免疫肽的关键特征

Wan Xiaoxiao, Vomund Anthony N, Peterson Orion J, Chervonsky Alexander V, Lichti Cheryl F, Unanue Emil R

Integrated Pancreatic Blood Flow: Bidirectional Microcirculation Between Endocrine and Exocrine Pancreas

胰腺血流整合:内分泌胰腺和外分泌胰腺之间的双向微循环

Dybala, Michael P; Kuznetsov, Andrey; Motobu, Maki; Hendren-Santiago, Bryce K; Philipson, Louis H; Chervonsky, Alexander V; Hara, Manami

Cutting Edge: Commensal Microbiota Has Disparate Effects on Manifestations of Polyglandular Autoimmune Inflammation

前沿研究:共生微生物群对多腺体自身免疫性炎症的表现具有不同的影响

Hansen, Camilla H F; Yurkovetskiy, Leonid A; Chervonsky, Alexander V

Mammalian Lipopolysaccharide Receptors Incorporated into the Retroviral Envelope Augment Virus Transmission

整合到逆转录病毒包膜中的哺乳动物脂多糖受体增强病毒传播

Wilks, Jessica; Lien, Egil; Jacobson, Amy N; Fischbach, Michael A; Qureshi, Nilofer; Chervonsky, Alexander V; Golovkina, Tatyana V

Microbiota and autoimmunity: exploring new avenues

微生物群与自身免疫:探索新途径

Yurkovetskiy, Leonid A; Pickard, Joseph M; Chervonsky, Alexander V

Intestinal fucose as a mediator of host-microbe symbiosis

肠道岩藻糖作为宿主-微生物共生的介质

Pickard, Joseph M; Chervonsky, Alexander V

Loss of the death receptor CD95 (Fas) expression by dendritic cells protects from a chronic viral infection

树突状细胞死亡受体CD95(Fas)表达的缺失可预防慢性病毒感染。

Varanasi, Vineeth; Khan, Aly Azeem; Chervonsky, Alexander V