Selective disruption of DNMT1/ELK1 interactions induces DGKI re-expression and promotes temozolomide sensitivity of MGMT(methylated)/DGKI(methylated) glioblastoma.
选择性破坏 DNMT1/ELK1 相互作用可诱导 DGKI 重新表达，并促进 MGMT(甲基化)/DGKI(甲基化) 胶质母细胞瘤对替莫唑胺的敏感性
期刊:Clinical Epigenetics
影响因子:4.4
doi:10.1186/s13148-025-01943-8
Besson Jean-Maxime, Etcheverry Amandine, Nadaradjane Arulraj, Bougras-Cartron Gwenola, Aubry Marc, Frenel Jean-Sebastien, Chevanieu Alain, Lopez Marie, Mosser Jean, Cartron Pierre-François
