Increased Excitability and Synaptic Plasticity of Drd1- and Drd2-Expressing Prelimbic Neurons Projecting to Nucleus Accumbens after Heroin Abstinence Are Reversed by Cue-Induced Relapse and Protein Kinase A Inhibition

戒除海洛因后，投射到伏隔核的表达 Drd1 和 Drd2 的前边缘神经元的兴奋性和突触可塑性增加，而线索诱发的复发和蛋白激酶 A 抑制可逆转这一现象