Chiakang相关文献与解析，生知库 | 链接生命科学的每一步

Matrix stiffness-induced α-tubulin acetylation is required for skin fibrosis formation through activation of Yes-associated protein

基质僵硬诱导的 α-微管蛋白乙酰化是通过激活 Yes 相关蛋白来形成皮肤纤维化的必要条件

期刊:MedComm

影响因子:

doi:10.1002/mco2.319

Dongsheng Wen, Ya Gao, Yangdan Liu, Chiakang Ho, Jiaming Sun, Lu Huang, Yuxin Liu, Qingfeng Li, Yifan Zhang

表观遗传

发表日期：2023

文献求助收藏

Targeting the stem cell niche: role of collagen XVII in skin aging and wound repair

靶向干细胞微环境：胶原蛋白XVII在皮肤衰老和伤口修复中的作用

期刊:Theranostics

影响因子:13.3

doi:10.7150/thno.78016

Liu, Yangdan; Ho, Chiakang; Wen, Dongsheng; Sun, Jiaming; Huang, Lu; Gao, Ya; Li, Qingfeng; Zhang, Yifan

HDAC5-mediated Smad7 silencing through MEF2A is critical for fibroblast activation and hypertrophic scar formation

HDAC5 通过 MEF2A 介导的 Smad7 沉默对于成纤维细胞活化和肥厚性瘢痕形成至关重要

期刊:International Journal of Biological Sciences

影响因子:8.2

doi:10.7150/ijbs.76140

Ya Gao, Yangdan Liu, Danning Zheng, Chiakang Ho, Dongsheng Wen, Jiaming Sun, Lu Huang, Yuxin Liu, Qingfeng Li, Yifan Zhang

Focusing on Mechanoregulation Axis in Fibrosis: Sensing, Transduction and Effecting

聚焦纤维化中的机械调节轴：感知、转导和效应

期刊:Frontiers in Molecular Biosciences

影响因子:4

doi:10.3389/fmolb.2022.804680

Wen, Dongsheng; Gao, Ya; Ho, Chiakang; Yu, Li; Zhang, Yuguang; Lyu, Guozhong; Hu, Dahai; Li, Qingfeng; Zhang, Yifan

信号转导

发表日期：2022

文献求助收藏

LRG1 Promotes Keratinocyte Migration and Wound Repair through Regulation of HIF-1α Stability

LRG1通过调节HIF-1α的稳定性促进角质形成细胞迁移和伤口修复

期刊:Journal of Investigative Dermatology

影响因子:5.7

doi:10.1016/j.jid.2019.06.143

Ya Gao, Zhibo Xie, Chiakang Ho, Jing Wang, Qingfeng Li, Yifan Zhang, Jia Zhou

Ubiquitin-Specific Protease 15 Maintains Transforming Growth Factor-β Pathway Activity by Deubiquitinating Transforming Growth Factor-β Receptor I during Wound Healing

泛素特异性蛋白酶 15 在伤口愈合过程中通过去泛素化转化生长因子-β 受体 I 维持转化生长因子-β 通路活性

期刊:American Journal of Pathology

影响因子:4.7

doi:10.1016/j.ajpath.2019.03.005

Yixuan Zhao, Zi Wang, Chiakang Ho, Guoyou Zhang, Qingfeng Li

表观遗传

发表日期：2019

文献求助收藏

Alisol A attenuates high-fat-diet-induced obesity and metabolic disorders via the AMPK/ACC/SREBP-1c pathway

Alisol A 通过 AMPK/ACC/SREBP-1c 通路减轻高脂饮食引起的肥胖和代谢紊乱。

期刊:Journal of Cellular and Molecular Medicine

影响因子:4.3

doi:10.1111/jcmm.14380.

Chiakang Ho,Ya Gao,Danning Zheng,Yanjun Liu,Shengzhou Shan,Bin Fang,Yixuan Zhao,Dingzhong Song,Yifan Zhang,Qingfeng Li

Piezo1 and tissue fibrosis: insights into its role and potential for modulation

Piezo1与组织纤维化：对其作用及潜在调控的深入研究

PKM2-mediated collagen XVII expression is critical for wound repair

PKM2介导的胶原蛋白XVII表达对伤口修复至关重要

Epigenetics as a versatile regulator of fibrosis

表观遗传学作为纤维化的多功能调节因子

Matrix stiffness-induced α-tubulin acetylation is required for skin fibrosis formation through activation of Yes-associated protein

基质僵硬诱导的 α-微管蛋白乙酰化是通过激活 Yes 相关蛋白来形成皮肤纤维化的必要条件

Targeting the stem cell niche: role of collagen XVII in skin aging and wound repair

靶向干细胞微环境：胶原蛋白XVII在皮肤衰老和伤口修复中的作用

HDAC5-mediated Smad7 silencing through MEF2A is critical for fibroblast activation and hypertrophic scar formation

HDAC5 通过 MEF2A 介导的 Smad7 沉默对于成纤维细胞活化和肥厚性瘢痕形成至关重要

Focusing on Mechanoregulation Axis in Fibrosis: Sensing, Transduction and Effecting

聚焦纤维化中的机械调节轴：感知、转导和效应

LRG1 Promotes Keratinocyte Migration and Wound Repair through Regulation of HIF-1α Stability

LRG1通过调节HIF-1α的稳定性促进角质形成细胞迁移和伤口修复

Ubiquitin-Specific Protease 15 Maintains Transforming Growth Factor-β Pathway Activity by Deubiquitinating Transforming Growth Factor-β Receptor I during Wound Healing

泛素特异性蛋白酶 15 在伤口愈合过程中通过去泛素化转化生长因子-β 受体 I 维持转化生长因子-β 通路活性

Alisol A attenuates high-fat-diet-induced obesity and metabolic disorders via the AMPK/ACC/SREBP-1c pathway

Alisol A 通过 AMPK/ACC/SREBP-1c 通路减轻高脂饮食引起的肥胖和代谢紊乱。