日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Heparan Sulfates Regulate Vascular Reactivity in Syndecan 1 Knockout Mice

硫酸乙酰肝素调节 Syndecan 1 基因敲除小鼠的血管反应性

Potje, Simone R; Katiki, Aishwarya; Pires, Paulo W; Chignalia, Andreia Zago

Temporal expression dynamics of glypicans during hiPSC cardiac differentiation

hiPSC心脏分化过程中糖蛋白聚糖的动态表达

Mesquita, Fernanda C P; Kim, Stephanie J; Chignalia, Andreia Z; Hochman-Mendez, Camila

Glypican 1 mechanosensing mediates eNOS uncoupling during hydrostatic pulmonary edema

糖蛋白聚糖1的机械感受介导静水压性肺水肿期间的eNOS解偶联

Thota, Lakshmi Narasimha Rao; Lopez Rosales, Joaquin Enrique; Isbatan, Ayman; Bime, Christian; Carvalho-de-Souza, Joao Luis; Dull, Randal O; Black, Stephen M; Chignalia, Andreia Zago

Novel avenues to control blood pressure: targeting the renal lymphatic system

控制血压的新途径:靶向肾脏淋巴系统

Chignalia, Andreia Zago

The Pulmonary Endothelial Glycocalyx Modifications in Glypican 1 Knockout Mice Do Not Affect Lung Endothelial Function in Physiological Conditions

Glypican 1 基因敲除小鼠的肺内皮糖萼修饰不会影响生理条件下的肺内皮功能

Lakshmi N R Thota, Joaquin E Lopez Rosales, Ivan Placencia, Evgeny A Zemskov, Paola Tonino, Ashley N Michael, Stephen M Black, Andreia Z Chignalia

Modulation of cardiac voltage-activated K+ currents by glypican 1 heparan sulfate proteoglycan

肝素硫酸蛋白聚糖 1 对心脏电压激活 K+ 电流的调节

Diego Santos Souza, Andreia Zago Chignalia, Joao Luis Carvalho-de-Souza

Glypican 1 and syndecan 1 differently regulate noradrenergic hypertension development: Focus on IP3R and calcium

Glypican 1 和 syndecan 1 以不同的方式调节去甲肾上腺素能高血压的发展:重点关注 IP3R 和钙

Simone R Potje, Ayman Isbatan, Rita C Tostes, Lusiane M Bendhack, Randal O Dull, Joao L Carvalho-de-Souza, Andreia Z Chignalia

Norepinephrine Induces Lung Microvascular Endothelial Cell Death by NADPH Oxidase-Dependent Activation of Caspase-3

去甲肾上腺素通过 NADPH 氧化酶依赖性激活 Caspase-3 诱导肺微血管内皮细胞死亡

Chignalia, Andreia Z; Weinberg, Guy; Dull, Randal O

Reduced caveolae density in arteries of SHR contributes to endothelial dysfunction and ROS production

SHR 动脉中小泡密度降低导致内皮功能障碍和 ROS 产生

Simone R Potje, Marcella D Grando, Andreia Z Chignalia, Cristina Antoniali, Lusiane M Bendhack

Pressure-dependent NOS activation contributes to endothelial hyperpermeability in a model of acute heart failure

在急性心力衰竭模型中,压力依赖性NOS激活导致内皮细胞高通透性。

Chignalia, Andreia Z; Isbatan, Ayman; Patel, Milan; Ripper, Richard; Sharlin, Jordan; Shosfy, Joelle; Borlaug, Barry A; Dull, Randal O