Chinopoulos Christos相关文献与解析，生知库

Underestimation of mitochondrial respiratory capacity in gray matter voxels of the human brain map due to limited OXPHOS and TCA cycle in astrocytes

由于星形胶质细胞中氧化磷酸化和三羧酸循环的局限性，导致人脑图谱灰质体素中线粒体呼吸能力被低估。

期刊:Frontiers in Cellular Neuroscience

影响因子:4

doi:10.3389/fncel.2025.1661231

Chinopoulos, Christos

The Warburg hypothesis and the emergence of the mitochondrial metabolic theory of cancer

瓦尔堡假说与线粒体代谢癌症理论的出现

期刊:Journal of Bioenergetics and Biomembranes

影响因子:3

doi:10.1007/s10863-025-10059-w

Seyfried, Thomas N; Lee, Derek C; Duraj, Tomas; Ta, Nathan L; Mukherjee, Purna; Kiebish, Michael; Arismendi-Morillo, Gabriel; Chinopoulos, Christos

Complex I activity in hypoxia: implications for oncometabolism

缺氧条件下复合物I活性：对肿瘤代谢的影响

期刊:Biochemical Society Transactions

影响因子:4.3

doi:10.1042/BST20230189

Chinopoulos, Christos

Amino Acid and Glucose Fermentation Maintain ATP Content in Mouse and Human Malignant Glioma Cells.

氨基酸和葡萄糖发酵维持小鼠和人类恶性胶质瘤细胞中的 ATP 含量

期刊:ASN Neuro

影响因子:3.7

doi:10.1080/17590914.2024.2422268

Lee Derek C, Ta Linh, Mukherjee Purna, Duraj Tomas, Domin Marek, Greenwood Bennett, Karmacharya Srada, Narain Niven R, Kiebish Michael, Chinopoulos Christos, Seyfried Thomas N

Reverse and Forward Electron Flow-Induced H(2)O(2) Formation Is Decreased in α-Ketoglutarate Dehydrogenase (α-KGDH) Subunit (E2 or E3) Heterozygote Knock Out Animals

在α-酮戊二酸脱氢酶（α-KGDH）亚基（E2或E3）杂合敲除动物中，反向和正向电子流诱导的H(2)O(2)生成减少

期刊:Antioxidants

影响因子:6.6

doi:10.3390/antiox11081487

Horváth, Gergő; Sváb, Gergely; Komlódi, Tímea; Ravasz, Dora; Kacsó, Gergely; Doczi, Judit; Chinopoulos, Christos; Ambrus, Attila; Tretter, László

发表日期：2022

文献求助收藏

Proline Oxidation Supports Mitochondrial ATP Production When Complex I Is Inhibited

当复合物I受到抑制时，脯氨酸氧化可支持线粒体ATP的产生。

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms23095111

Pallag, Gergely; Nazarian, Sara; Ravasz, Dora; Bui, David; Komlódi, Timea; Doerrier, Carolina; Gnaiger, Erich; Seyfried, Thomas N; Chinopoulos, Christos

线粒体

发表日期：2022

文献求助收藏

Metabolic management of microenvironment acidity in glioblastoma

胶质母细胞瘤微环境酸度的代谢调控

期刊:Frontiers in Oncology

影响因子:3.3

doi:10.3389/fonc.2022.968351

Seyfried, Thomas N; Arismendi-Morillo, Gabriel; Zuccoli, Giulio; Lee, Derek C; Duraj, Tomas; Elsakka, Ahmed M; Maroon, Joseph C; Mukherjee, Purna; Ta, Linh; Shelton, Laura; D'Agostino, Dominic; Kiebish, Michael; Chinopoulos, Christos

The Mystery of Extramitochondrial Proteins Lysine Succinylation

线粒体外蛋白赖氨酸琥珀酰化的奥秘

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms22116085

Chinopoulos, Christos

Can the Mitochondrial Metabolic Theory Explain Better the Origin and Management of Cancer than Can the Somatic Mutation Theory?

线粒体代谢理论能否比体细胞突变理论更好地解释癌症的起源和治疗？

期刊:Metabolites

影响因子:3.7

doi:10.3390/metabo11090572

Seyfried, Thomas N; Chinopoulos, Christos

Catabolic rewiring in cancer impacts dietary interventions

癌症中的分解代谢重编程会影响饮食干预

Underestimation of mitochondrial respiratory capacity in gray matter voxels of the human brain map due to limited OXPHOS and TCA cycle in astrocytes

由于星形胶质细胞中氧化磷酸化和三羧酸循环的局限性，导致人脑图谱灰质体素中线粒体呼吸能力被低估。

The Warburg hypothesis and the emergence of the mitochondrial metabolic theory of cancer

瓦尔堡假说与线粒体代谢癌症理论的出现

Complex I activity in hypoxia: implications for oncometabolism

缺氧条件下复合物I活性：对肿瘤代谢的影响

Amino Acid and Glucose Fermentation Maintain ATP Content in Mouse and Human Malignant Glioma Cells.

氨基酸和葡萄糖发酵维持小鼠和人类恶性胶质瘤细胞中的 ATP 含量

Reverse and Forward Electron Flow-Induced H(2)O(2) Formation Is Decreased in α-Ketoglutarate Dehydrogenase (α-KGDH) Subunit (E2 or E3) Heterozygote Knock Out Animals

在α-酮戊二酸脱氢酶（α-KGDH）亚基（E2或E3）杂合敲除动物中，反向和正向电子流诱导的H(2)O(2)生成减少

Proline Oxidation Supports Mitochondrial ATP Production When Complex I Is Inhibited

当复合物I受到抑制时，脯氨酸氧化可支持线粒体ATP的产生。

Metabolic management of microenvironment acidity in glioblastoma

胶质母细胞瘤微环境酸度的代谢调控

The Mystery of Extramitochondrial Proteins Lysine Succinylation

线粒体外蛋白赖氨酸琥珀酰化的奥秘

Can the Mitochondrial Metabolic Theory Explain Better the Origin and Management of Cancer than Can the Somatic Mutation Theory?

线粒体代谢理论能否比体细胞突变理论更好地解释癌症的起源和治疗？