Choksi Swati相关文献与解析，生知库 | 链接生命科学的每一步

TGFB signaling induces mitophagy via PLSCR3-mediated cardiolipin externalization in conjunction with a BNIP3L/NIX-, BNIP3-, and FUNDC1-dependent mechanism.

TGFB 信号通过 PLSCR3 介导的心磷脂外化诱导线粒体自噬，并依赖于 BNIP3L/NIX、BNIP3 和 FUNDC1 机制

期刊:Autophagy

影响因子:14.3

doi:10.1080/15548627.2025.2483441

Yan Jiong, Chen Xin, Choksi Swati, Liu Zheng-Gang

ATF4-mediated expression of NOXA is critical for Necroptosis driven by Glucose Deprivation

ATF4介导的NOXA表达对于葡萄糖剥夺驱动的坏死性凋亡至关重要

期刊:

影响因子:

doi:10.3389/fceld.2024.1507960

Sonkaew, Sasiprapa; Rajna, Ruwaida; Park, Yeon-Ji; Yan, Jiong; Liu, Zhaoshan; Jitkaew, Siriporn; Liu, Zheng-Gang; Choksi, Swati

Mitochondria engage the integrated stress response to promote tumor growth

线粒体启动整合应激反应以促进肿瘤生长

期刊:Oncotarget

影响因子:

doi:10.18632/oncotarget.28404

Liu, Zhaoshan; Choksi, Swati; Liu, Zheng-Gang; Boulton, Dillon P; Caino, M Cecilia

Transition from TNF-Induced Inflammation to Death Signaling

从TNF诱导的炎症到死亡信号的转变

期刊:Methods in Molecular Biology

影响因子:

doi:10.1007/978-1-0716-1130-2_5

Choksi, Swati; Choudhary, Gourav; Liu, Zheng-Gang

炎症/感染

信号转导

Immunology/Inflammation

炎症

发表日期：2021

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Necroptosis of tumor cells leads to tumor necrosis and promotes tumor metastasis

肿瘤细胞坏死性凋亡导致肿瘤坏死，并促进肿瘤转移。

期刊:Cell Research

影响因子:25.9

doi:10.1038/s41422-018-0058-y

Jiao, Delong; Cai, Zhenyu; Choksi, Swati; Ma, Dan; Choe, Moran; Kwon, Hyung-Joon; Baik, Jin Young; Rowan, Brian G; Liu, Chengyu; Liu, Zheng-Gang

Switching from TNF-induced inflammation to death signaling

从TNF诱导的炎症信号转向死亡信号

期刊:Molecular and Cellular Oncology

影响因子:1.9

doi:10.1080/23723556.2017.1392402

Xu, Qing; Choksi, Swati; Liu, Zhengang

Essential roles of receptor-interacting protein and TRAF2 in oxidative stress-induced cell death

受体相互作用蛋白和TRAF2在氧化应激诱导的细胞死亡中起着至关重要的作用

期刊:Molecular and Cellular Biology

影响因子:2.7

doi:10.1128/MCB.24.13.5914-5922.2004

Shen, Han-Ming; Lin, Yong; Choksi, Swati; Tran, Jamie; Jin, Tian; Chang, Lufen; Karin, Michael; Zhang, Jianke; Liu, Zheng-gang

Soluble tissue factor generated by necroptosis-triggered shedding is responsible for thrombosis.

坏死性凋亡触发的脱落产生的可溶性组织因子是血栓形成的原因。

TGFB signaling induces mitophagy via PLSCR3-mediated cardiolipin externalization in conjunction with a BNIP3L/NIX-, BNIP3-, and FUNDC1-dependent mechanism.

TGFB 信号通过 PLSCR3 介导的心磷脂外化诱导线粒体自噬，并依赖于 BNIP3L/NIX、BNIP3 和 FUNDC1 机制

ATF4-mediated expression of NOXA is critical for Necroptosis driven by Glucose Deprivation

ATF4介导的NOXA表达对于葡萄糖剥夺驱动的坏死性凋亡至关重要

Mitochondria engage the integrated stress response to promote tumor growth

线粒体启动整合应激反应以促进肿瘤生长

Transition from TNF-Induced Inflammation to Death Signaling

从TNF诱导的炎症到死亡信号的转变

Necroptosis of tumor cells leads to tumor necrosis and promotes tumor metastasis

肿瘤细胞坏死性凋亡导致肿瘤坏死，并促进肿瘤转移。

Switching from TNF-induced inflammation to death signaling

从TNF诱导的炎症信号转向死亡信号

Essential roles of receptor-interacting protein and TRAF2 in oxidative stress-induced cell death

受体相互作用蛋白和TRAF2在氧化应激诱导的细胞死亡中起着至关重要的作用