日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Graded Notch Signaling Functions as a Rheostat of Lineage Plasticity and Therapy Resistance in Prostate Cancer

Notch信号通路在调节前列腺癌谱系可塑性和治疗耐药性方面发挥着关键作用

Jiang, Yuyin; Cheng, Siyuan; Zhang, Catherine Yijia; Jin, Xiao; Li, Longjun; Fraidenburg, Melanie; Wang, Choushi; Kim, Isaac Yi; Mu, Ping

ZNF397 Deficiency Triggers TET2-Driven Lineage Plasticity and AR-Targeted Therapy Resistance in Prostate Cancer

ZNF397 缺乏会引发前列腺癌中 TET2 驱动的谱系可塑性和 AR 靶向治疗耐药性

Yaru Xu, Yuqiu Yang, Zhaoning Wang, Martin Sjöström, Yuyin Jiang, Yitao Tang, Siyuan Cheng, Su Deng, Choushi Wang, Julisa Gonzalez, Nickolas A Johnson, Xiang Li, Xiaoling Li, Lauren A Metang, Atreyi Mukherji, Quanhui Xu, Carla R Tirado, Garrett Wainwright, Xinzhe Yu, Spencer Barnes, Mia Hofstad, Yu

UBE2J1 is the E2 ubiquitin-conjugating enzyme regulating androgen receptor degradation and antiandrogen resistance

UBE2J1 是 E2 泛素结合酶,可调节雄激素受体降解和抗雄激素抵抗

Carla Rodriguez Tirado, Choushi Wang, Xiaoling Li, Su Deng, Julisa Gonzalez, Nickolas A Johnson, Yaru Xu, Lauren A Metang, Medha Sundar Rajan, Yuqiu Yang, Yi Yin, Mia Hofstad, Ganesh V Raj, Song Zhang, Andrew Lemoff, Wei He, Jie Fan, Yunguan Wang, Tao Wang, Ping Mu

Loss of SYNCRIP unleashes APOBEC-driven mutagenesis, tumor heterogeneity, and AR-targeted therapy resistance in prostate cancer

SYNCRIP 的缺失会导致前列腺癌中 APOBEC 驱动的诱变、肿瘤异质性和 AR 靶向治疗耐药性

Xiaoling Li, Yunguan Wang, Su Deng, Guanghui Zhu, Choushi Wang, Nickolas A Johnson, Zeda Zhang, Carla Rodriguez Tirado, Yaru Xu, Lauren A Metang, Julisa Gonzalez, Atreyi Mukherji, Jianfeng Ye, Yuqiu Yang, Wei Peng, Yitao Tang, Mia Hofstad, Zhiqun Xie, Heewon Yoon, Liping Chen, Xihui Liu, Sujun Chen,

Author Correction: Ectopic JAK-STAT activation enables the transition to a stem-like and multilineage state conferring AR-targeted therapy resistance

作者更正:异位JAK-STAT激活可使细胞向干细胞样多谱系状态转变,从而赋予其对雄激素受体靶向治疗的耐药性。

Deng, Su; Wang, Choushi; Wang, Yunguan; Xu, Yaru; Li, Xiaoling; Johnson, Nickolas A; Mukherji, Atreyi; Lo, U-Ging; Xu, Lingfan; Gonzalez, Julisa; Metang, Lauren A; Ye, Jianfeng; Tirado, Carla Rodriguez; Rodarte, Kathia; Zhou, Yinglu; Xie, Zhiqun; Arana, Carlos; Annamalai, Valli; Liu, Xihui; Vander Griend, Donald J; Strand, Douglas; Hsieh, Jer-Tsong; Li, Bo; Raj, Ganesh; Wang, Tao; Mu, Ping

Ectopic JAK-STAT activation enables the transition to a stem-like and multilineage state conferring AR-targeted therapy resistance

异位JAK-STAT激活可使细胞转变为干细胞样和多谱系状态,从而产生AR靶向治疗耐药性

Su Deng #, Choushi Wang #, Yunguan Wang #, Yaru Xu, Xiaoling Li, Nickolas A Johnson, Atreyi Mukherji, U-Ging Lo, Lingfan Xu, Julisa Gonzalez, Lauren A Metang, Jianfeng Ye, Carla Rodriguez Tirado, Kathia Rodarte, Yinglu Zhou, Zhiqun Xie, Carlos Arana, Valli Annamalai, Xihui Liu, Donald J Vander Grien

Loss of CHD1 Promotes Heterogeneous Mechanisms of Resistance to AR-Targeted Therapy via Chromatin Dysregulation

CHD1 的缺失通过染色质失调促进对 AR 靶向治疗产生异质性耐药机制

Zeda Zhang, Chuanli Zhou, Xiaoling Li, Spencer D Barnes, Su Deng, Elizabeth Hoover, Chi-Chao Chen, Young Sun Lee, Yanxiao Zhang, Choushi Wang, Lauren A Metang, Chao Wu, Carla Rodriguez Tirado, Nickolas A Johnson, John Wongvipat, Kristina Navrazhina, Zhen Cao, Danielle Choi, Chun-Hao Huang, Eliot Lin