日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Regulation of Ferroptosis in Cancer and Immune Cells

癌细胞和免疫细胞中铁死亡的调控

Jang, Naeyoon; Kim, Il-Kyu; Jung, Dawoon; Chung, Yeonseok; Kang, Yun Pyo

IL-27-induced PD-L1(high)Sca-1(+) innate lymphoid cells suppress contact hypersensitivity in an IL-10-dependent manner

IL-27诱导的PD-L1(high)Sca-1(+)固有淋巴细胞以IL-10依赖的方式抑制接触性超敏反应。

Min, Keun Young; Kim, Do-Kyun; Jo, Min Geun; Choi, Min Yeong; Lee, Dajeong; Park, Jeong Won; Park, Young-Jun; Chung, Yeonseok; Kim, Young Mi; Park, Yeong-Min; Kim, Hyuk Soon; Choi, Wahn Soo

IL-17 and IL-21: Their Immunobiology and Therapeutic Potentials

IL-17 和 IL-21:它们的免疫生物学和治疗潜力

Koh, Choong-Hyun; Kim, Byung-Seok; Kang, Chang-Yuil; Chung, Yeonseok; Seo, Hyungseok

Control of T-cell immunity by fatty acid metabolism

脂肪酸代谢对T细胞免疫的调控

Kim, Jaemin; Lee, Yoosun; Chung, Yeonseok

Strategies to Overcome Hurdles in Cancer Immunotherapy

克服癌症免疫疗法障碍的策略

Kim, Jihyun; Lee, Byung Joon; Moon, Sehoon; Lee, Hojeong; Lee, Juyong; Kim, Byung-Soo; Jung, Keehoon; Seo, Hyungseok; Chung, Yeonseok

TYRO3 blockade enhances anti-PD-1 therapy response by modulating expression of CCN1 in tumor microenvironment

TYRO3阻断通过调节肿瘤微环境中CCN1的表达来增强抗PD-1疗法的疗效。

Park, Miso; Kuen, Da-Sol; Park, Jaewoo; Choi, Munkyung; Kim, Yeonji; Roh, Eun Chae; Choi, Yong June; Kim, Yoon Gyoon; Chung, Yeonseok; Cho, Sung Yun; Kang, Keon Wook

Golden Age of Immunotherapy: Challenges and Opportunities

免疫疗法的黄金时代:挑战与机遇

Chung, Yeonseok; Lee, Seung-Woo; Kim, Wan-Uk

Critical regulation of follicular helper T cell differentiation and function by Gα(13) signaling

Gα(13)信号通路对滤泡辅助性T细胞分化和功能的关键调控

Kuen, Da-Sol; Park, Miso; Ryu, Heeju; Choi, Garam; Moon, Young-Hye; Kim, Jae-Ouk; Kang, Keon Wook; Kim, Sang Geon; Chung, Yeonseok

No Time to Die, Born to Be Killers: Survival Is Accompanied by Exhaustion after an Endless Battle. After intense combat, defending killer T cells surrender to chronic disease pathogens or cancer by capitulating into an exhausted, dysfunctional cell type, T(Ex). Is their surrender definitive?

《007:无暇赴死》和《天生杀手》:无尽的战斗之后,生存伴随着精疲力竭。在激烈的战斗之后,防御型杀伤性T细胞会向慢性疾病病原体或癌细胞投降,转变为一种疲惫不堪、功能失调的细胞类型——T(Ex)。它们的投降是永久性的吗?

Diederich, Sarah; Chung, Yeonseok

Immunologic Aspects of Dyslipidemia: a Critical Regulator of Adaptive Immunity and Immune Disorders

血脂异常的免疫学方面:适应性免疫和免疫紊乱的关键调节因子

Kim, Daehong; Chung, Hayeon; Lee, Jeong-Eun; Kim, Jiyeon; Hwang, Junseok; Chung, Yeonseok