Fibroblasts-specific p16(INK4a) exacerbates inflammageing-mediated post-infarction ventricular remodelling through interacting with STAT3 to regulate NLRP3 transcription
成纤维细胞特异性p16(INK4a)通过与STAT3相互作用调节NLRP3转录,加剧炎症衰老介导的梗死后心室重塑。
期刊:Clinical and Translational Medicine
影响因子:6.8
doi:10.1002/ctm2.70344
Gu, Xin; Du, Yingqiang; Zhang, Jin'ge; Li, Jiyu; Chen, Haiyun; Lin, Yujie; Wang, Yue; Zhang, Chunli; Lin, Shiyu; Hao, Nannan; Peng, Chengyi; Ge, Jiacheng; Liu, Jin; Liang, Yan; Zhang, Yongjie; Wang, Xiaoyan; Wang, Fang; Jin, Jianliang
成纤维细胞
炎症
炎症/感染
JAK/STAT
STAT3
LRP3
衰老
NLRP3
细胞生物学