Interaction of the late endo-lysosomal lipid PI(3,5)P2 with the Vph1 isoform of yeast V-ATPase increases its activity and cellular stress tolerance.

晚期内溶酶体脂质 PI(3,5)P2 与酵母 V-ATPase 的 Vph1 同工型相互作用，可提高其活性和细胞应激耐受性