Claudio Cardinali相关文献与解析，生知库

Giorgio Santoni, Consuelo Amantini, Maria Beatrice Morelli, Daniele Tomassoni, Matteo Santoni, Oliviero Marinelli, Massimo Nabissi, Claudio Cardinali, Vittorio Paolucci, Mariangela Torniai, Silvia Rinaldi, Francesca Morgese, Giovanni Bernardini, Rossana Berardi

胸腺瘤

发表日期：2018

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The TRPV1 ion channel regulates thymocyte differentiation by modulating autophagy and proteasome activity

TRPV1 离子通道通过调节自噬和蛋白酶体活性来调节胸腺细胞分化

期刊:Oncotarget

影响因子:

doi:10.18632/oncotarget.21798

Consuelo Amantini, Valerio Farfariello, Claudio Cardinali, Maria Beatrice Morelli, Oliviero Marinelli, Massimo Nabissi, Matteo Santoni, Laura Bonfili, Valentina Cecarini, Anna Maria Eleuteri, Giorgio Santoni

Capsaicin triggers autophagic cell survival which drives epithelial mesenchymal transition and chemoresistance in bladder cancer cells in an Hedgehog-dependent manner

辣椒素触发自噬细胞存活，以 Hedgehog 依赖的方式驱动膀胱癌细胞的上皮间质转化和化学抗性

期刊:Oncotarget

影响因子:

doi:10.18632/oncotarget.10326

Consuelo Amantini, Maria Beatrice Morelli, Massimo Nabissi, Claudio Cardinali, Matteo Santoni, Angela Gismondi, Giorgio Santoni

Cannabinoids synergize with carfilzomib, reducing multiple myeloma cells viability and migration

大麻素与卡非佐米协同作用，降低多发性骨髓瘤细胞的活力和迁移

期刊:Oncotarget

影响因子:

doi:10.18632/oncotarget.12721

Massimo Nabissi, Maria Beatrice Morelli, Massimo Offidani, Consuelo Amantini, Silvia Gentili, Alessandra Soriani, Claudio Cardinali, Pietro Leoni, Giorgio Santoni

Sorafenib induces cathepsin B-mediated apoptosis of bladder cancer cells by regulating the Akt/PTEN pathway. The Akt inhibitor, perifosine, enhances the sorafenib-induced cytotoxicity against bladder cancer cells

索拉非尼通过调节 Akt/PTEN 通路诱导蛋白酶 B 介导的膀胱癌细胞凋亡。Akt 抑制剂哌利福星可增强索拉非尼诱导的对膀胱癌细胞的细胞毒性

期刊:

影响因子:1.3

doi:10.18632/oncoscience.147

Consuelo Amantini, Maria Beatrice Morelli, Matteo Santoni, Alessandra Soriani, Claudio Cardinali, Valerio Farfariello, Anna Maria Eleuteri, Laura Bonfili, Matteo Mozzicafreddo, Massimo Nabissi, Stefano Cascinu, Giorgio Santoni

Cross-talk between alpha1D-adrenoceptors and transient receptor potential vanilloid type 1 triggers prostate cancer cell proliferation

α1D 肾上腺素能受体与瞬时受体电位香草酸 1 型之间的相互作用引发前列腺癌细胞增殖

期刊:BMC Cancer

影响因子:3.4

doi:10.1186/1471-2407-14-921

Maria Beatrice Morelli, Consuelo Amantini, Massimo Nabissi, Sonia Liberati, Claudio Cardinali, Valerio Farfariello, Daniele Tomassoni, Wilma Quaglia, Alessandro Piergentili, Alessandro Bonifazi, Fabio Del Bello, Matteo Santoni, Gabriele Mammana, Lucilla Servi, Alessandra Filosa, Angela Gismondi, Gio

High CTLA-4 expression correlates with poor prognosis in thymoma patients

CTLA-4 高表达与胸腺瘤患者预后不良相关

The TRPV1 ion channel regulates thymocyte differentiation by modulating autophagy and proteasome activity

TRPV1 离子通道通过调节自噬和蛋白酶体活性来调节胸腺细胞分化

Capsaicin triggers autophagic cell survival which drives epithelial mesenchymal transition and chemoresistance in bladder cancer cells in an Hedgehog-dependent manner

辣椒素触发自噬细胞存活，以 Hedgehog 依赖的方式驱动膀胱癌细胞的上皮间质转化和化学抗性

Cannabinoids synergize with carfilzomib, reducing multiple myeloma cells viability and migration

大麻素与卡非佐米协同作用，降低多发性骨髓瘤细胞的活力和迁移

Sorafenib induces cathepsin B-mediated apoptosis of bladder cancer cells by regulating the Akt/PTEN pathway. The Akt inhibitor, perifosine, enhances the sorafenib-induced cytotoxicity against bladder cancer cells

索拉非尼通过调节 Akt/PTEN 通路诱导蛋白酶 B 介导的膀胱癌细胞凋亡。Akt 抑制剂哌利福星可增强索拉非尼诱导的对膀胱癌细胞的细胞毒性

Cross-talk between alpha1D-adrenoceptors and transient receptor potential vanilloid type 1 triggers prostate cancer cell proliferation

α1D 肾上腺素能受体与瞬时受体电位香草酸 1 型之间的相互作用引发前列腺癌细胞增殖