日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

In vivo imaging approach to detect and quantify early fibrogenic remodeling following low-dose bleomycin exposure

利用体内成像方法检测和量化低剂量博来霉素暴露后早期纤维化重塑

Mantovani, Elena; Ragnoli, Beatrice; Clemente, Nausicaa; Chiazza, Fausto; Malerba, Mario

ICOSL: more than a trigger of ICOS function

ICOSL:不仅仅是ICOS功能的触发器

Stoppa, Ian; Provera, Alessia; Sacchetti, Sara; Clemente, Nausicaa; Pantham, Deepika; Christaki, Foteini; Vecchio, Cristina; Sheferaw, Anteneh Nigussie; Rolla, Roberta; Dianzani, Chiara; Sutti, Salvatore; Dianzani, Umberto

Exploring Anti-Neoplastic Activity of Chitosan Nanobubbles Decorated with ICOS-Fc and Loaded with Paclitaxel in a Human and Murine Model of Melanoma

在人和小鼠黑色素瘤模型中探索ICOS-Fc修饰并负载紫杉醇的壳聚糖纳米泡的抗肿瘤活性

Pantham, Deepika; Argenziano, Monica; Christaki, Foteini; Clemente, Nausicaa; Colombo, Chiara; Benetti, Elisa; Pizzimenti, Stefania; Dianzani, Umberto; Stoppa, Ian; Cavalli, Roberta; Dianzani, Chiara

Monensin Inhibits Triple-Negative Breast Cancer in Mice by a Na(+)-Dependent Cytotoxic Action Unrelated to Cytostatic Effects

莫能菌素通过一种与细胞抑制作用无关的钠离子依赖性细胞毒性作用抑制小鼠三阴性乳腺癌。

Fiorilla, Simone; Tasso, Francesco; Clemente, Nausicaa; Trisciuoglio, Tommaso; Boldorini, Renzo; Carini, Rita

The CXCR3/PLC/IP3-IP3R axis is responsible for the ignition of UPR in intestinal epithelial cells exposed to gliadin peptide, during the onset of celiac disease

在乳糜泻发病过程中,CXCR3/PLC/IP3-IP3R轴负责启动暴露于麦醇溶蛋白肽的肠上皮细胞中的未折叠蛋白反应(UPR)。

Monzani, Romina; Gagliardi, Mara; Saverio, Valentina; Clemente, Nausicaa; Monzani, Alice; Rabbone, Ivana; Nigrelli, Francesca; Pellizzaro, Samuele; Ferrario, Emanuele; Saettone, Silvia; Pagano, Nico; De Leo, Luigina; Lim, Dmitry; Sblattero, Daniele; Corazzari, Marco

Gliadin-dependent UPR induction directly triggers the expression of TG2 and pro-inflammatory cytokines, dysregulates intestinal permeability, and reduces CFTR expression in intestinal epithelial cells of celiac disease patients

麦醇溶蛋白依赖的UPR诱导可直接触发TG2和促炎细胞因子的表达,扰乱肠道通透性,并降低乳糜泻患者肠上皮细胞中CFTR的表达。

Monzani, Romina; Gagliardi, Mara; Saverio, Valentina; Clemente, Nausicaa; Monzani, Alice; Rabbone, Ivana; Nigrelli, Francesca; Pellizzaro, Samuele; Ferrario, Emanuele; Saettone, Silvia; Pagano, Nico; De Leo, Luigina; Lim, Dmitry; Sblattero, Daniele; Corazzari, Marco

The Association of Axonal Damage Biomarkers and Osteopontin at Diagnosis Could Be Useful in Newly Diagnosed MS Patients

轴突损伤生物标志物与骨桥蛋白在诊断时的关联性可能对新诊断的多发性硬化症患者有所帮助

Virgilio, Eleonora; Puricelli, Chiara; Clemente, Nausicaa; Ciampana, Valentina; Imperatore, Ylenia; Perga, Simona; Stangalini, Sveva; Boggio, Elena; Appiani, Alice; Gigliotti, Casimiro Luca; Dianzani, Umberto; Comi, Cristoforo; Vecchio, Domizia

The Gut-Ex-Vivo System (GEVS) Is a Dynamic and Versatile Tool for the Study of DNBS-Induced IBD in BALB/C and C57BL/6 Mice, Highlighting the Protective Role of Probiotics

肠道离体系统(GEVS)是一种动态且用途广泛的工具,可用于研究 BALB/C 和 C57BL/6 小鼠中 DNBS 诱导的 IBD,并突显益生菌的保护作用。

Monzani, Romina; Gagliardi, Mara; Clemente, Nausicaa; Saverio, Valentina; Pańczyszyn, Elżbieta; Santoro, Claudio; Yissachar, Nissan; Visciglia, Annalisa; Pane, Marco; Amoruso, Angela; Corazzari, Marco

DUSP12 acts as a novel endogenous protective signal against hepatic ischemia-reperfusion damage by inhibiting ASK1 pathway

DUSP12通过抑制ASK1通路,发挥新型内源性保护信号作用,对抗肝脏缺血再灌注损伤。

Boldorini, Renzo; Clemente, Nausicaa; Alchera, Elisa; Carini, Rita

Gut-Ex-Vivo system as a model to study gluten response in celiac disease.

以离体肠道系统为模型研究乳糜泻中麸质反应

Gagliardi Mara, Clemente Nausicaa, Monzani Romina, Fusaro Luca, Ferrari Eleonora, Saverio Valentina, Grieco Giovanna, Pańczyszyn Elżbieta, Carton Flavia, Santoro Claudio, Del Mare-Roumani Sara, Amidror Sivan, Yissachar Nissan, Boccafoschi Francesca, Zucchelli Silvia, Corazzari Marco