Cole Susan E相关文献与解析，生知库 | 链接生命科学的每一步

Putative binding sites for mir-125 family miRNAs in the mouse Lfng 3'UTR affect transcript expression in the segmentation clock, but mir-125a-5p is dispensable for normal somitogenesis

小鼠 Lfng 3'UTR 中 mir-125 家族 miRNA 的假定结合位点影响节律时钟中的转录本表达，但 mir-125a-5p 对于正常的体节发生并非必需。

期刊:Developmental Dynamics

影响因子:1.5

doi:10.1002/dvdy.24552

Wahi, Kanu; Friesen, Sophia; Coppola, Vincenzo; Cole, Susan E

miRNA

发表日期：2017

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Fine-tuning of Notch signaling sets the boundary of the organ of Corti and establishes sensory cell fates

Notch信号的精细调控决定了科尔蒂氏器的边界，并决定了感觉细胞的命运。

期刊:Elife

影响因子:6.4

doi:10.7554/eLife.19921

Basch, Martin L; Brown, Rogers M 2nd; Jen, Hsin-I; Semerci, Fatih; Depreux, Frederic; Edlund, Renée K; Zhang, Hongyuan; Norton, Christine R; Gridley, Thomas; Cole, Susan E; Doetzlhofer, Angelika; Maletic-Savatic, Mirjana; Segil, Neil; Groves, Andrew K

Collagen Fibril Ultrastructure in Mice Lacking Discoidin Domain Receptor 1.

缺乏盘状结构域受体 1 的小鼠的胶原纤维超微结构

期刊:Microscopy and Microanalysis

影响因子:3

doi:10.1017/S1431927616000787

Tonniges Jeffrey R, Albert Benjamin, Calomeni Edward P, Roy Shuvro, Lee Joan, Mo Xiaokui, Cole Susan E, Agarwal Gunjan

信号转导

发表日期：2016

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Mir-125a-5p-mediated regulation of Lfng is essential for the avian segmentation clock

Mir-125a-5p介导的Lfng调控对鸟类节律时钟至关重要

期刊:Developmental Cell

影响因子:8.7

doi:10.1016/j.devcel.2013.01.024

Riley, Maurisa F; Bochter, Matthew S; Wahi, Kanu; Nuovo, Gerard J; Cole, Susan E

信号转导

发表日期：2013

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Manic fringe is not required for embryonic development, and fringe family members do not exhibit redundant functions in the axial skeleton, limb, or hindbrain

狂躁边缘蛋白并非胚胎发育所必需，边缘蛋白家族成员在轴骨骼、四肢或后脑中也不表现出冗余功能。

期刊:Developmental Dynamics

影响因子:1.5

doi:10.1002/dvdy.21982

Moran, Jennifer L; Shifley, Emily T; Levorse, John M; Mani, Shyamala; Ostmann, Kristin; Perez-Balaguer, Ariadna; Walker, Dawn M; Vogt, Thomas F; Cole, Susan E

NOTCH1 mutations in individuals with left ventricular outflow tract malformations reduce ligand-induced signaling

左心室流出道畸形患者的 NOTCH1 突变会降低配体诱导的信号传导。

期刊:Human Molecular Genetics

影响因子:3.2

doi:10.1093/hmg/ddn187

McBride, Kim L; Riley, Maurisa F; Zender, Gloria A; Fitzgerald-Butt, Sara M; Towbin, Jeffrey A; Belmont, John W; Cole, Susan E

A tale of two clocks: phosphorylation of NICD by CDKs links cell cycle and segmentation clock

两个时钟的故事：CDK 对 NICD 的磷酸化连接细胞周期和节段时钟

Putative binding sites for mir-125 family miRNAs in the mouse Lfng 3'UTR affect transcript expression in the segmentation clock, but mir-125a-5p is dispensable for normal somitogenesis

小鼠 Lfng 3'UTR 中 mir-125 家族 miRNA 的假定结合位点影响节律时钟中的转录本表达，但 mir-125a-5p 对于正常的体节发生并非必需。

Fine-tuning of Notch signaling sets the boundary of the organ of Corti and establishes sensory cell fates

Notch信号的精细调控决定了科尔蒂氏器的边界，并决定了感觉细胞的命运。

Collagen Fibril Ultrastructure in Mice Lacking Discoidin Domain Receptor 1.

缺乏盘状结构域受体 1 的小鼠的胶原纤维超微结构

Mir-125a-5p-mediated regulation of Lfng is essential for the avian segmentation clock

Mir-125a-5p介导的Lfng调控对鸟类节律时钟至关重要

Manic fringe is not required for embryonic development, and fringe family members do not exhibit redundant functions in the axial skeleton, limb, or hindbrain

狂躁边缘蛋白并非胚胎发育所必需，边缘蛋白家族成员在轴骨骼、四肢或后脑中也不表现出冗余功能。

NOTCH1 mutations in individuals with left ventricular outflow tract malformations reduce ligand-induced signaling

左心室流出道畸形患者的 NOTCH1 突变会降低配体诱导的信号传导。