Coleman Michael P相关文献与解析，生知库 | 链接生命科学的每一步

日期影响因子

SARM1 activation induces reversible mitochondrial dysfunction and can be prevented in human neurons by antisense oligonucleotides.

SARM1 激活可导致可逆性线粒体功能障碍，而反义寡核苷酸可以预防人类神经元中的这种功能障碍

期刊:Neurobiology of Disease

影响因子:5.6

doi:10.1016/j.nbd.2025.106986

Loreto Andrea, Cramb Kaitlyn M L, McDermott Lucy A, Antoniou Christina, Cirilli Ilenia, Caiazza Maria Claudia, Merlini Elisa, Arthur-Farraj Peter, du Preez W Daniel, Mock Elliot D, Zhao Hien T, Bennett David L, Orsomando Giuseppe, Coleman Michael P, Wade-Martins Richard

发表日期：2025

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Author Correction: NMNAT2 is a druggable target to drive neuronal NAD production

作者更正：NMNAT2 是一个可作为药物靶点以促进神经元 NAD 生成的靶点

期刊:Nature Communications

影响因子:15.7

doi:10.1038/s41467-024-52439-7

Tribble, James R; Jöe, Melissa; Varricchio, Carmine; Otmani, Amin; Canovai, Alessio; Habchi, Baninia; Daskalakis, Evangelia; Chaleckis, Romanas; Loreto, Andrea; Gilley, Jonathan; Wheelock, Craig E; Jóhannesson, Gauti; Wong, Raymond C B; Coleman, Michael P; Brancale, Andrea; Williams, Pete A

发表日期：2024

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Programmed axon death: a promising target for treating retinal and optic nerve disorders

程序性轴突死亡：治疗视网膜和视神经疾病的一个有前景的靶点

doi:10.1038/s41433-024-03025-0

Loreto, Andrea; Merlini, Elisa; Coleman, Michael P

发表日期：2024

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Axon Biology in ALS: Mechanisms of Axon Degeneration and Prospects for Therapy

肌萎缩侧索硬化症中的轴突生物学：轴突退化的机制和治疗前景

期刊:Neurotherapeutics

影响因子:6.9

doi:10.1007/s13311-022-01297-6

Coleman, Michael P

发表日期：2022

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Selective androgen receptor modulators activate the canonical prostate cancer androgen receptor program and repress cancer growth

选择性雄激素受体调节剂可激活典型的前列腺癌雄激素受体程序并抑制癌症生长

期刊:Journal of Clinical Investigation

影响因子:13.3

doi:10.1172/JCI146777

Michael D Nyquist, Lisa S Ang, Alexandra Corella, Ilsa M Coleman, Michael P Meers, Anthony J Christiani, Cordell Pierce, Derek H Janssens, Hannah E Meade, Arnab Bose, Lauren Brady, Timothy Howard, Navonil De Sarkar, Sander B Frank, Ruth F Dumpit, James T Dalton, Eva Corey, Stephen R Plymate, Michael

发表日期：2021

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Lessons from Injury: How Nerve Injury Studies Reveal Basic Biological Mechanisms and Therapeutic Opportunities for Peripheral Nerve Diseases

从损伤中汲取教训：神经损伤研究如何揭示周围神经疾病的基本生物学机制和治疗机会

期刊:Neurotherapeutics

影响因子:6.9

doi:10.1007/s13311-021-01125-3

Arthur-Farraj, Peter; Coleman, Michael P

发表日期：2021

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Correction to: Lessons from Injury: How Nerve Injury Studies Reveal Basic Biological Mechanisms and Therapeutic Opportunities for Peripheral Nerve Diseases

更正：从损伤中汲取教训：神经损伤研究如何揭示周围神经疾病的基本生物学机制和治疗机会

期刊:Neurotherapeutics

影响因子:6.9

doi:10.1007/s13311-021-01162-y

Arthur-Farraj, Peter; Coleman, Michael P

发表日期：2021

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Loss of highwire Protects Against the Deleterious Effects of Traumatic Brain Injury in Drosophila Melanogaster.

高线缺失可保护黑腹果蝇免受创伤性脑损伤的有害影响

期刊:Frontiers in Neurology

影响因子:2.8

doi:10.3389/fneur.2020.00401

Hill Ciaran S, Sreedharan Jemeen, Loreto Andrea, Menon David K, Coleman Michael P

发表日期：2020

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Interaction between a MAPT variant causing frontotemporal dementia and mutant APP affects axonal transport

导致额颞叶痴呆的MAPT变异体与突变型APP之间的相互作用会影响轴突运输。

期刊:Neurobiology of Aging

影响因子:3.5

doi:10.1016/j.neurobiolaging.2018.03.033

Adalbert, Robert; Milde, Stefan; Durrant, Claire; Ando, Kunie; Stygelbout, Virginie; Yilmaz, Zehra; Gould, Stacey; Brion, Jean-Pierre; Coleman, Michael P

发表日期：2018

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P7C3-A20 neuroprotection is independent of Wallerian degeneration in primary neuronal culture

P7C3-A20 的神经保护作用与原代神经元培养中的瓦勒氏变性无关

期刊:Neuroreport

影响因子:1.7

doi:10.1097/WNR.0000000000001146

Hill, Ciaran S; Menon, David K; Coleman, Michael P

发表日期：2018

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