Conde-Garrosa Ruth相关文献与解析，生知库

AcÃn-PÃ©rez Rebeca, Iborra Salvador, MartÃ-Mateos Yolanda, Cook Emma C L, Conde-Garrosa Ruth, Petcherski Anton, MuÃ±oz MÂª Del Mar, MartÃnez de Mena Raquel, Krishnan Karthickeyan Chella, JimÃ©nez ConcepciÃ³n, BolaÃ±os Juan Pedro, Laakso Markku, Lusis Aldon J, Shirihai Orian S, Sancho David, EnrÃquez JosÃ© Antonio

细胞生物学

巨噬细胞

发表日期：2020

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Optimal Generation of Tissue-Resident but Not Circulating Memory T Cells during Viral Infection Requires Crosspriming by DNGR-1(+) Dendritic Cells

病毒感染期间组织驻留记忆T细胞（而非循环记忆T细胞）的最佳生成需要DNGR-1(+)树突状细胞的交叉启动。

期刊:Immunity

影响因子:26.3

doi:10.1016/j.immuni.2016.08.019

Iborra, Salvador; Martínez-López, María; Khouili, Sofía C; Enamorado, Michel; Cueto, Francisco J; Conde-Garrosa, Ruth; Del Fresno, Carlos; Sancho, David

Leishmania Uses Mincle to Target an Inhibitory ITAM Signaling Pathway in Dendritic Cells that Dampens Adaptive Immunity to Infection

利什曼原虫利用Mincle靶向树突状细胞中抑制性的ITAM信号通路，从而减弱对感染的适应性免疫。

期刊:Immunity

影响因子:26.3

doi:10.1016/j.immuni.2016.09.012

Iborra, Salvador; Martínez-López, María; Cueto, Francisco J; Conde-Garrosa, Ruth; Del Fresno, Carlos; Izquierdo, Helena M; Abram, Clare L; Mori, Daiki; Campos-Martín, Yolanda; Reguera, Rosa María; Kemp, Benjamin; Yamasaki, Sho; Robinson, Matthew J; Soto, Manuel; Lowell, Clifford A; Sancho, David

SHIP-1 Couples to the Dectin-1 hemITAM and Selectively Modulates Reactive Oxygen Species Production in Dendritic Cells in Response to Candida albicans

SHIP-1 与 Dectin-1 半 ITAM 偶联，并选择性地调节树突状细胞中活性氧的产生以响应白色念珠菌感染。

期刊:Journal of Immunology

影响因子:3.4

doi:10.4049/jimmunol.1402874

Blanco-Menéndez, Noelia; Del Fresno, Carlos; Fernandes, Sandra; Calvo, Enrique; Conde-Garrosa, Ruth; Kerr, William G; Sancho, David

Fgr kinase is required for proinflammatory macrophage activation during diet-induced obesity.

Fgr激酶是饮食诱导肥胖过程中促炎巨噬细胞活化所必需的

Optimal Generation of Tissue-Resident but Not Circulating Memory T Cells during Viral Infection Requires Crosspriming by DNGR-1(+) Dendritic Cells

病毒感染期间组织驻留记忆T细胞（而非循环记忆T细胞）的最佳生成需要DNGR-1(+)树突状细胞的交叉启动。

Leishmania Uses Mincle to Target an Inhibitory ITAM Signaling Pathway in Dendritic Cells that Dampens Adaptive Immunity to Infection

利什曼原虫利用Mincle靶向树突状细胞中抑制性的ITAM信号通路，从而减弱对感染的适应性免疫。

SHIP-1 Couples to the Dectin-1 hemITAM and Selectively Modulates Reactive Oxygen Species Production in Dendritic Cells in Response to Candida albicans

SHIP-1 与 Dectin-1 半 ITAM 偶联，并选择性地调节树突状细胞中活性氧的产生以响应白色念珠菌感染。