Copeland O'Neal相关文献与解析，生知库 | 链接生命科学的每一步

日期影响因子

Pressure Overload Is Associated With Low Levels of Troponin I and Myosin Binding Protein C Phosphorylation in the Hearts of Patients With Aortic Stenosis

压力负荷过重与主动脉瓣狭窄患者心脏中肌钙蛋白I和肌球蛋白结合蛋白C磷酸化水平降低有关

期刊:Frontiers in Physiology

影响因子:3.4

doi:10.3389/fphys.2020.00241

Copeland, O'neal; Messer, Andrew; Jabbour, Andrew; Poggesi, Corrado; Prasad, Sanjay; Marston, Steven

发表日期：2020

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Investigations into the Sarcomeric Protein and Ca(2+)-Regulation Abnormalities Underlying Hypertrophic Cardiomyopathy in Cats (Felix catus).

对猫（Felix catus）肥厚型心肌病肌节蛋白和 Ca(2+) 调节异常的研究

期刊:Frontiers in Physiology

影响因子:3.4

doi:10.3389/fphys.2017.00348

Messer Andrew E, Chan Jasmine, Daley Alex, Copeland O'Neal, Marston Steven B, Connolly David J

发表日期：2017

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A post-MI power struggle: adaptations in cardiac power occur at the sarcomere level alongside MyBP-C and RLC phosphorylation

心肌梗死后权力斗争：心肌力量的适应性变化发生在肌节水平，伴随MyBP-C和RLC磷酸化。

期刊:American Journal of Physiology-Heart and Circulatory Physiology

影响因子:4.1

doi:10.1152/ajpheart.00899.2015

Toepfer, Christopher N; Sikkel, Markus B; Caorsi, Valentina; Vydyanath, Anupama; Torre, Iratxe; Copeland, O'Neal; Lyon, Alexander R; Marston, Steven B; Luther, Pradeep K; Macleod, Kenneth T; West, Timothy G; Ferenczi, Michael A

发表日期：2016

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The dilated cardiomyopathy-causing mutation ACTC E361G in cardiac muscle myofibrils specifically abolishes modulation of Ca(2+) regulation by phosphorylation of troponin I

导致扩张型心肌病的ACTC E361G突变存在于心肌肌原纤维中，特异性地消除了肌钙蛋白I磷酸化对Ca(2+)调节的调控作用。

期刊:Biophysical Journal

影响因子:3.1

doi:10.1016/j.bpj.2014.10.024

Vikhorev, Petr G; Song, Weihua; Wilkinson, Ross; Copeland, O'Neal; Messer, Andrew E; Ferenczi, Michael A; Marston, Steven B

发表日期：2014

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