Corazzari Marco相关文献与解析，生知库 | 链接生命科学的每一步

The CXCR3/PLC/IP3-IP3R axis is responsible for the ignition of UPR in intestinal epithelial cells exposed to gliadin peptide, during the onset of celiac disease

在乳糜泻发病过程中，CXCR3/PLC/IP3-IP3R轴负责启动暴露于麦醇溶蛋白肽的肠上皮细胞中的未折叠蛋白反应（UPR）。

Monzani, Romina; Gagliardi, Mara; Saverio, Valentina; Clemente, Nausicaa; Monzani, Alice; Rabbone, Ivana; Nigrelli, Francesca; Pellizzaro, Samuele; Ferrario, Emanuele; Saettone, Silvia; Pagano, Nico; De Leo, Luigina; Lim, Dmitry; Sblattero, Daniele; Corazzari, Marco

上皮细胞

Gliadin-dependent UPR induction directly triggers the expression of TG2 and pro-inflammatory cytokines, dysregulates intestinal permeability, and reduces CFTR expression in intestinal epithelial cells of celiac disease patients

麦醇溶蛋白依赖的UPR诱导可直接触发TG2和促炎细胞因子的表达，扰乱肠道通透性，并降低乳糜泻患者肠上皮细胞中CFTR的表达。

期刊:Biology Direct

影响因子:4.9

doi:10.1186/s13062-025-00644-9

Tellurium-Doped Bioactive Glass Induces Ferroptosis in Osteosarcoma Cells Regardless of FSP1

碲掺杂生物活性玻璃诱导骨肉瘤细胞发生铁死亡，且该过程与FSP1无关。

期刊:Antioxidants

影响因子:6.6

doi:10.3390/antiox13111327

Pańczyszyn, Elżbieta; Lallukka, Mari; Gagliardi, Mara; Saverio, Valentina; Monzani, Romina; Miola, Marta; Verné, Enrica; Corazzari, Marco

Characterization of gut microbiota dynamics in an Alzheimer's disease mouse model through clade-specific marker-based analysis of shotgun metagenomic data

通过基于谱系特异性标记的宏基因组鸟枪法数据分析，表征阿尔茨海默病小鼠模型中肠道菌群的动态变化

期刊:Biology Direct

影响因子:4.9

doi:10.1186/s13062-024-00541-7

Favero, Francesco; Re, Angela; Dason, Mohammed Salim; Gravina, Teresa; Gagliardi, Mara; Mellai, Marta; Corazzari, Marco; Corà, Davide

Transglutaminase 2 and Ferroptosis: a new liaison?

转谷氨酰胺酶 2 和铁死亡：新的联系？

期刊:Cell Death Discovery

影响因子:7

doi:10.1038/s41420-023-01394-1

Gagliardi, Mara; Saverio, Valentina; Rossin, Federica; D'Eletto, Manuela; Corazzari, Marco

铁死亡

发表日期：2023

文献求助收藏

Wild-type and mutant p53 in cancer-related ferroptosis. A matter of stress management?

野生型和突变型p53在癌症相关铁死亡中的作用。这与压力管理有关吗？

期刊:Frontiers in Genetics

影响因子:2.8

doi:10.3389/fgene.2023.1148192

Corazzari, Marco; Collavin, Licio

The Gut-Ex-Vivo System (GEVS) Is a Dynamic and Versatile Tool for the Study of DNBS-Induced IBD in BALB/C and C57BL/6 Mice, Highlighting the Protective Role of Probiotics

肠道离体系统（GEVS）是一种动态且用途广泛的工具，可用于研究 BALB/C 和 C57BL/6 小鼠中 DNBS 诱导的 IBD，并突显益生菌的保护作用。

期刊:Biology-Basel

影响因子:3.5

doi:10.3390/biology11111574

Monzani, Romina; Gagliardi, Mara; Clemente, Nausicaa; Saverio, Valentina; Pańczyszyn, Elżbieta; Santoro, Claudio; Yissachar, Nissan; Visciglia, Annalisa; Pane, Marco; Amoruso, Angela; Corazzari, Marco

发表日期：2022

文献求助收藏

A Metabologenomic approach reveals alterations in the gut microbiota of a mouse model of Alzheimer's disease

代谢基因组学方法揭示了阿尔茨海默病小鼠模型肠道菌群的改变

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0273036

Favero, Francesco; Barberis, Elettra; Gagliardi, Mara; Espinoza, Stefano; Contu, Liliana; Gustincich, Stefano; Boccafoschi, Francesca; Borsotti, Chiara; Lim, Dmitry; Rubino, Vito; Mignone, Flavio; Pasolli, Edoardo; Manfredi, Marcello; Zucchelli, Silvia; Corà, Davide; Corazzari, Marco

Gut-Ex-Vivo system as a model to study gluten response in celiac disease.

以离体肠道系统为模型研究乳糜泻中麸质反应

期刊:Cell Death Discovery

影响因子:7

doi:10.1038/s41420-021-00430-2

Gagliardi Mara, Clemente Nausicaa, Monzani Romina, Fusaro Luca, Ferrari Eleonora, Saverio Valentina, Grieco Giovanna, PaÅczyszyn ElÅ¼bieta, Carton Flavia, Santoro Claudio, Del Mare-Roumani Sara, Amidror Sivan, Yissachar Nissan, Boccafoschi Francesca, Zucchelli Silvia, Corazzari Marco

AKRs confer oligodendrocytes resistance to differentiation-stimulated ferroptosis

AKR赋予少突胶质细胞抵抗分化刺激性铁死亡的能力

The CXCR3/PLC/IP3-IP3R axis is responsible for the ignition of UPR in intestinal epithelial cells exposed to gliadin peptide, during the onset of celiac disease

在乳糜泻发病过程中，CXCR3/PLC/IP3-IP3R轴负责启动暴露于麦醇溶蛋白肽的肠上皮细胞中的未折叠蛋白反应（UPR）。

Gliadin-dependent UPR induction directly triggers the expression of TG2 and pro-inflammatory cytokines, dysregulates intestinal permeability, and reduces CFTR expression in intestinal epithelial cells of celiac disease patients

麦醇溶蛋白依赖的UPR诱导可直接触发TG2和促炎细胞因子的表达，扰乱肠道通透性，并降低乳糜泻患者肠上皮细胞中CFTR的表达。

Tellurium-Doped Bioactive Glass Induces Ferroptosis in Osteosarcoma Cells Regardless of FSP1

碲掺杂生物活性玻璃诱导骨肉瘤细胞发生铁死亡，且该过程与FSP1无关。

Characterization of gut microbiota dynamics in an Alzheimer's disease mouse model through clade-specific marker-based analysis of shotgun metagenomic data

通过基于谱系特异性标记的宏基因组鸟枪法数据分析，表征阿尔茨海默病小鼠模型中肠道菌群的动态变化

Transglutaminase 2 and Ferroptosis: a new liaison?

转谷氨酰胺酶 2 和铁死亡：新的联系？

Wild-type and mutant p53 in cancer-related ferroptosis. A matter of stress management?

野生型和突变型p53在癌症相关铁死亡中的作用。这与压力管理有关吗？

The Gut-Ex-Vivo System (GEVS) Is a Dynamic and Versatile Tool for the Study of DNBS-Induced IBD in BALB/C and C57BL/6 Mice, Highlighting the Protective Role of Probiotics

肠道离体系统（GEVS）是一种动态且用途广泛的工具，可用于研究 BALB/C 和 C57BL/6 小鼠中 DNBS 诱导的 IBD，并突显益生菌的保护作用。

A Metabologenomic approach reveals alterations in the gut microbiota of a mouse model of Alzheimer's disease

代谢基因组学方法揭示了阿尔茨海默病小鼠模型肠道菌群的改变

Gut-Ex-Vivo system as a model to study gluten response in celiac disease.

以离体肠道系统为模型研究乳糜泻中麸质反应