Endurance exercise with reduced muscle glycogen content influences substrate utilization and attenuates acute mTORC1- and autophagic signaling in human type I and type II muscle fibers.

肌肉糖原含量降低的耐力运动会影响底物利用，并减弱人类 I 型和 II 型肌纤维中的急性 mTORC1 和自噬信号