日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

The neuroprotective γ-hydroxybutyrate analog 3-hydroxycyclopent-1-enecarboxylic acid does not directly affect CaMKIIα autophosphorylation at T286 or binding to GluN2B

具有神经保护作用的γ-羟基丁酸类似物3-羟基环戊-1-烯羧酸不直接影响CaMKIIα在T286位点的自磷酸化或与GluN2B的结合。

Brown, Carolyn Nicole; Blaine, Rachel E; Barker, Chase Madison; Coultrap, Steven J; Bayer, Karl Ulrich

Aβ impairs the LTP-related movement of endogenous CaMKII but not of exogenous GFP-CaMKII

Aβ会损害内源性CaMKII的LTP相关运动,但不会影响外源性GFP-CaMKII的运动。

Brown, Carolyn Nicole; Chao, Fan-Yi; Quang, Daphne; Rumian, Nicole L; Kleinjan, Mason S; Coultrap, Steven J; Bayer, K Ulrich

The CaMKII D135N mutation blocks kinase activity and reduces GluN2B binding

CaMKII D135N 突变会阻断激酶活性并降低 GluN2B 的结合。

Larsen, Matthew E; Barker, C Madison; Vargas, Raul Satoshi; Coultrap, Steven J; Bayer, K Ulrich

CaMKII monomers are sufficient for GluN2B binding, co-condensation, and synaptic potentiation.

CaMKII 单体足以与 GluN2B 结合、共凝聚和突触增强

Brown Carolyn Nicole, Barker C Madison, Miller Carley N, Aoto Jason, Coultrap Steven J, Bayer K Ulrich

LTP expression mediated by autonomous activity of GluN2B-bound CaMKII.

LTP表达由GluN2B结合的CaMKII的自主活性介导

Rumian Nicole L, Barker C Madison, Larsen Matthew E, Tullis Jonathan E, Freund Ronald K, Taslimi Amir, Coultrap Steven J, Tucker Chandra L, Dell'Acqua Mark L, Bayer K Ulrich

LTP induction by structural rather than enzymatic functions of CaMKII

CaMKII 的结构功能而非酶功能诱导 LTP

Tullis, Jonathan E; Larsen, Matthew E; Rumian, Nicole L; Freund, Ronald K; Boxer, Emma E; Brown, Carolyn Nicole; Coultrap, Steven J; Schulman, Howard; Aoto, Jason; Dell'Acqua, Mark L; Bayer, K Ulrich

Decreased nitrosylation of CaMKII causes aging-associated impairments in memory and synaptic plasticity in mice

CaMKII亚硝基化水平降低会导致小鼠出现与衰老相关的记忆力和突触可塑性障碍。

Rumian, Nicole L; Freund, Ronald K; Dell'Acqua, Mark L; Coultrap, Steven J; Bayer, K Ulrich

CaMKII versus DAPK1 Binding to GluN2B in Ischemic Neuronal Cell Death after Resuscitation from Cardiac Arrest.

心脏骤停复苏后缺血性神经元细胞死亡中 CaMKII 与 DAPK1 与 GluN2B 的结合

Buonarati Olivia R, Cook Sarah G, Goodell Dayton J, Chalmers Nicholas E, Rumian Nicole L, Tullis Jonathan E, Restrepo Susana, Coultrap Steven J, Quillinan Nidia, Herson Paco S, Bayer K Ulrich

Analysis of the CaMKIIα and β splice-variant distribution among brain regions reveals isoform-specific differences in holoenzyme formation

对脑区中 CaMKIIα 和 β 剪接变体分布的分析揭示了全酶形成过程中同工型特异性的差异

Cook, Sarah G; Bourke, Ashley M; O'Leary, Heather; Zaegel, Vincent; Lasda, Erika; Mize-Berge, Janna; Quillinan, Nidia; Tucker, Chandra L; Coultrap, Steven J; Herson, Paco S; Bayer, K Ulrich

The CaMKII holoenzyme structure in activation-competent conformations

CaMKII全酶在激活活性构象下的结构

Myers, Janette B; Zaegel, Vincent; Coultrap, Steven J; Miller, Adam P; Bayer, K Ulrich; Reichow, Steve L