Daniel Prantner相关文献与解析，生知库 | 链接生命科学的每一步

Classically activated mouse macrophages produce methylglyoxal that induces a TLR4- and RAGE-independent proinflammatory response

经典激活的小鼠巨噬细胞产生甲基乙二醛，从而诱导 TLR4 和 RAGE 独立的促炎反应

期刊:Journal of Leukocyte Biology

影响因子:3.6

doi:10.1002/JLB.3A0520-745RR

Daniel Prantner, Shreeram Nallar, Katharina Richard, David Spiegel, Kim D Collins, Stefanie N Vogel

The role of RAGE in host pathology and crosstalk between RAGE and TLR4 in innate immune signal transduction pathways

RAGE 在宿主病理学中的作用以及 RAGE 与 TLR4 在先天免疫信号转导途径中的串扰

期刊:FASEB Journal

影响因子:4.4

doi:10.1096/fj.202002136R

Daniel Prantner, Shreeram Nallar, Stefanie N Vogel

AMP-activated Kinase (AMPK) Promotes Innate Immunity and Antiviral Defense through Modulation of Stimulator of Interferon Genes (STING) Signaling

AMP 活化激酶 (AMPK) 通过调节干扰素基因刺激因子 (STING) 信号传导来促进先天免疫和抗病毒防御

期刊:Journal of Biological Chemistry

影响因子:4

doi:10.1074/jbc.M116.763268

Daniel Prantner, Darren J Perkins, Stefanie N Vogel

The θ-defensin retrocyclin 101 inhibits TLR4- and TLR2-dependent signaling and protects mice against influenza infection

θ-防御素逆转录酶 101 抑制 TLR4 和 TLR2 依赖性信号传导并保护小鼠免受流感感染

期刊:Journal of Leukocyte Biology

影响因子:3.6

doi:10.1189/jlb.2A1215-567RR

Daniel Prantner, Kari Ann Shirey, Wendy Lai, Wuyuan Lu, Alexander M Cole, Stefanie N Vogel, Alfredo Garzino-Demo

The DNA sensor, cyclic GMP-AMP synthase, is essential for induction of IFN-β during Chlamydia trachomatis infection

DNA 传感器环磷酸鸟苷-腺苷酸合酶对于沙眼衣原体感染期间诱导 IFN-β 至关重要

期刊:Journal of Immunology

影响因子:3.6

doi:10.4049/jimmunol.1302718

Yugen Zhang, Laxmi Yeruva, Anthony Marinov, Daniel Prantner, Priscilla B Wyrick, Vladimir Lupashin, Uma M Nagarajan

信号转导

发表日期：2014

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CD4+ T cell expression of MyD88 is essential for normal resolution of Chlamydia muridarum genital tract infection

MyD88 的 CD4+ T 细胞表达对于沙眼衣原体生殖道感染的正常消退至关重要

期刊:Journal of Immunology

影响因子:3.6

doi:10.4049/jimmunol.1301547

Lauren C Frazer, Jeanne E Sullivan, Matthew A Zurenski, Margaret Mintus, Tammy E Tomasak, Daniel Prantner, Uma M Nagarajan, Toni Darville

5,6-Dimethylxanthenone-4-acetic acid (DMXAA) activates stimulator of interferon gene (STING)-dependent innate immune pathways and is regulated by mitochondrial membrane potential

5,6-二甲基黄酮-4-乙酸 (DMXAA) 可激活干扰素基因刺激因子 (STING) 依赖的先天免疫途径，并受线粒体膜电位调节

期刊:Journal of Biological Chemistry

影响因子:4

doi:10.1074/jbc.M112.382986

Daniel Prantner, Darren J Perkins, Wendy Lai, Mark S Williams, Shruti Sharma, Katherine A Fitzgerald, Stefanie N Vogel

Significant role of IL-1 signaling, but limited role of inflammasome activation, in oviduct pathology during Chlamydia muridarum genital infection

在沙眼衣原体生殖器感染期间，IL-1 信号在输卵管病理中起重要作用，但炎症小体激活作用有限

期刊:Journal of Immunology

影响因子:3.6

doi:10.4049/jimmunol.1103461

Uma M Nagarajan, James D Sikes, Laxmi Yeruva, Daniel Prantner

Interferon regulatory transcription factor 3 protects mice from uterine horn pathology during Chlamydia muridarum genital infection

干扰素调节转录因子 3 在小鼠生殖器感染衣原体期间保护小鼠免受子宫角病变

期刊:Infection and Immunity

影响因子:2.9

doi:10.1128/IAI.00140-11

Daniel Prantner, James D Sikes, Leah Hennings, Alena V Savenka, Alexei G Basnakian, Uma M Nagarajan

信号转导

发表日期：2011

文献求助收藏

M2a macrophages facilitate resolution of chemically-induced colitis in TLR4-SNP mice

M2a 巨噬细胞促进 TLR4-SNP 小鼠化学诱发结肠炎的消退

Classically activated mouse macrophages produce methylglyoxal that induces a TLR4- and RAGE-independent proinflammatory response

经典激活的小鼠巨噬细胞产生甲基乙二醛，从而诱导 TLR4 和 RAGE 独立的促炎反应

The role of RAGE in host pathology and crosstalk between RAGE and TLR4 in innate immune signal transduction pathways

RAGE 在宿主病理学中的作用以及 RAGE 与 TLR4 在先天免疫信号转导途径中的串扰

AMP-activated Kinase (AMPK) Promotes Innate Immunity and Antiviral Defense through Modulation of Stimulator of Interferon Genes (STING) Signaling

AMP 活化激酶 (AMPK) 通过调节干扰素基因刺激因子 (STING) 信号传导来促进先天免疫和抗病毒防御

The θ-defensin retrocyclin 101 inhibits TLR4- and TLR2-dependent signaling and protects mice against influenza infection

θ-防御素逆转录酶 101 抑制 TLR4 和 TLR2 依赖性信号传导并保护小鼠免受流感感染

The DNA sensor, cyclic GMP-AMP synthase, is essential for induction of IFN-β during Chlamydia trachomatis infection

DNA 传感器环磷酸鸟苷-腺苷酸合酶对于沙眼衣原体感染期间诱导 IFN-β 至关重要

CD4+ T cell expression of MyD88 is essential for normal resolution of Chlamydia muridarum genital tract infection

MyD88 的 CD4+ T 细胞表达对于沙眼衣原体生殖道感染的正常消退至关重要

5,6-Dimethylxanthenone-4-acetic acid (DMXAA) activates stimulator of interferon gene (STING)-dependent innate immune pathways and is regulated by mitochondrial membrane potential

5,6-二甲基黄酮-4-乙酸 (DMXAA) 可激活干扰素基因刺激因子 (STING) 依赖的先天免疫途径，并受线粒体膜电位调节

Significant role of IL-1 signaling, but limited role of inflammasome activation, in oviduct pathology during Chlamydia muridarum genital infection

在沙眼衣原体生殖器感染期间，IL-1 信号在输卵管病理中起重要作用，但炎症小体激活作用有限

Interferon regulatory transcription factor 3 protects mice from uterine horn pathology during Chlamydia muridarum genital infection

干扰素调节转录因子 3 在小鼠生殖器感染衣原体期间保护小鼠免受子宫角病变