日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

SAT-404 Effect of Nsmf Knockout upon Hypothalamic and Pituitary Gene Expression in the Nsmf KO Mouse

SAT-404 Nsmf基因敲除对Nsmf KO小鼠下丘脑和垂体基因表达的影响

Edwards, C K 3rd; Yunger, L M; Lorence, R M; Dantzer, R; Kelley, K W; Kashat, Mary; De Mattei, Leticia; Shelden, Daniel; Godlewska, Magdalena; Nowak, Andrzej Jerzy; Boguslawska, Anna; Kluczynski, Lukasz; Grochowska, Anna; Hubalewska-Dydejczyk, Alicja; Gilis-Januszewska, Aleksandra; Louden, Erica; Ko, Eun Kyung; Chorich, Lynn; Layman, Lawrence

Antidepressant activity of anti-cytokine treatment: a systematic review and meta-analysis of clinical trials of chronic inflammatory conditions

抗细胞因子治疗的抗抑郁活性:慢性炎症疾病临床试验的系统评价和荟萃分析

Kappelmann, N; Lewis, G; Dantzer, R; Jones, P B; Khandaker, G M

Serum kynurenic acid is reduced in affective psychosis

情感性精神病患者血清犬尿酸水平降低

Wurfel, B E; Drevets, W C; Bliss, S A; McMillin, J R; Suzuki, H; Ford, B N; Morris, H M; Teague, T K; Dantzer, R; Savitz, J B

Immunopsychiatry: important facts

免疫精神病学:重要事实

Khandaker, G M; Dantzer, R; Jones, P B

Correction to “Glutamate Receptor Ion Channels: Structure, Regulation, and Function”

对《谷氨酸受体离子通道:结构、调控和功能》的更正

Walker, A K; Kavelaars, A; Heijnen, C J; Dantzer, R

Lipopolysaccharide-induced depressive-like behavior is mediated by indoleamine 2,3-dioxygenase activation in mice

脂多糖诱导的小鼠抑郁样行为是由吲哚胺2,3-双加氧酶激活介导的。

O'Connor, J C; Lawson, M A; André, C; Moreau, M; Lestage, J; Castanon, N; Kelley, K W; Dantzer, R

Aging sensitizes mice to behavioral deficits induced by central HIV-1 gp120

衰老使小鼠对中枢 HIV-1 gp120 诱导的行为缺陷更加敏感

Abraham, J; Jang, S; Godbout, J P; Chen, J; Kelley, K W; Dantzer, R; Johnson, R W

Exaggerated sickness behavior and brain proinflammatory cytokine expression in aged mice in response to intracerebroventricular lipopolysaccharide

老年小鼠脑室内注射脂多糖后出现过度疾病行为和脑内促炎细胞因子表达增加

Huang, Y; Henry, C J; Dantzer, R; Johnson, R W; Godbout, J P

Tumor necrosis factor-alpha-induced sickness behavior is impaired by central administration of an inhibitor of c-jun N-terminal kinase.

通过中枢注射 c-jun N 端激酶抑制剂,可以削弱肿瘤坏死因子-α 诱发的疾病行为

Palin K, McCusker R H, Strle K, Moos F, Dantzer R, Kelley K W

A new mechanism of neurodegeneration: a proinflammatory cytokine inhibits receptor signaling by a survival peptide

神经退行性疾病的新机制:促炎细胞因子通过生存肽抑制受体信号传导

Venters, H D; Tang, Q; Liu, Q; VanHoy, R W; Dantzer, R; Kelley, K W